Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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Delayed Tooth Eruption <strong>Vitamin</strong> D may also have an influence on the eruption <strong>of</strong> the dentition. Specifically, low levels <strong>of</strong> vitamin D may result in delayed eruption. For instance, a cross-sectional study involving preschool Aboriginal children from a Northern Manitoba First Nations community reported that there was evidence <strong>of</strong> delayed eruption <strong>of</strong> primary teeth if mothers during pregnancy or infants were not receiving high-dose vitamin D supplementation. 97 Meanwhile, case report evidence suggests that long-standing, privational, vitamin D deficiency can result in many unerupted permanent teeth, including the maxillary canines, and molars. 70 Other Clinical Manifestations In addition to the previously discussed dental conditions and their associations with vitamin D, there are several other unique manifestations that require mentioning. These findings are summarized in Table 1.3-1. 1.3-30
Table 1.3-1 – Other dental conditions related to vitamin D status or rickets Description <strong>of</strong> Population Studied Clinical Findings Seven year old who experienced Abnormal pulp horns in permanent hypervitaminosis D during infancy central incisors at 7 years <strong>of</strong> age 71 resulting from over-fortification <strong>of</strong> milk Pulp horns partially filled in 71 19 year old with long-standing, privational, vitamin D deficiency Children with acute rickets Children with rickets Children with vitamin D dependent rickets (VDDR) Children with VDDR receiving treatment with vitamin D 3 and phosphate Children with vitamin D resistant rickets (VDRR) (X-linked hypophosphatemic rickets) Abnormal pulp chambers and root canals in permanent teeth 70 Presence <strong>of</strong> interglobular spaces in dentin 79 Minimal or no crowding in the permanent dentition 82 Higher incidence <strong>of</strong> malocclusion compared to controls receiving vitamin D prophylaxis (viosterol) during early childhood 78 Dental malocclusion, large pulp chambers and short roots 38 Interglobular spaces in the dentin, large pulp chambers and prominent pulp horns, loss <strong>of</strong> lamina dura, and early exfoliation <strong>of</strong> primary teeth 83,84 Disturbed normal dentinogenesis (dentin formation) in primary teeth 117 resulting in the presence <strong>of</strong> abundant interglobular dentin, lack <strong>of</strong> predentinal layer, thin dentin and large pulp chambers 117 Deficient mineralization <strong>of</strong> dentin, exhibited as globular dentin <strong>of</strong> varying degrees 118 Improved tooth mineralization 118 Globular dentin 118,119 Large pulp chambers and frequent dental absecesses 119,120 Increased prevalence <strong>of</strong> taurodontism and ectopic eruption <strong>of</strong> permanent canines 121 Children with VDRR receiving 1α-hydroxyvitamin D 3 Fewer endodontically treated teeth 98 Fewer dental abscesses 98 Aids in root canal and pulpal development 117 Normal enamel mineralization if receiving 1α-hydroxyvitamin 1.3-31
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Influence
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Abstract Influence
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Acknowledgements This longitudinal
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Table of Contents
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Status and Non-<st
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Table 1.1-1 - Recent studies report
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Table 1.1-2 - Previous used terms f
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The Link Between Enamel Hypoplasia
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If enamel hypoplasia results from n
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malocclusions. 145 Additional human
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Daily intakes of v
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13. Milnes AR, Rubin CW, Karpa M, T
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40. Definition of
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71. Watt RG. From victim blaming to
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97. Fearne JM, Bryan EM, Elliman AM
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124. Burt BA, Pai S. Does low birth
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151. Tinanoff N, D
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Section 2 - Risk Factors for Enamel
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correspond to the timing of
Page 39 and 40: een published. 3,4,17,19 The intent
Page 41 and 42: Table 1.2-2 - Risk factors for DDE
Page 43 and 44: Opacity 51 Implicated Risk Developm
Page 45 and 46: Implicated Risk Factor Factors asso
Page 47 and 48: o Metabolic Abnormalities and Healt
Page 49 and 50: 12 months of life.
Page 51 and 52: Other Factors Some uncommon risk fa
Page 53 and 54: devastating problem of</str
Page 55: 13. An epidemiological index <stron
Page 58 and 59: 54. Seow WK, Perham S. Enamel hypop
Page 60 and 61: 82. Berdal A, Papagerakis P, Hotton
Page 62 and 63: Vitamin D may be d
Page 64 and 65: have been used to prof</str
Page 66 and 67: health’. In addition, articles we
Page 68 and 69: 36 month trial had ended. 45 The nu
Page 70 and 71: Recently, a cross-sectional study <
Page 72 and 73: andomized, placebo-controlled study
Page 74 and 75: Further, amelogenin and enamelin, p
Page 76 and 77: A study of Austral
Page 78 and 79: Both dietary interventions and supp
Page 80 and 81: incidence of carie
Page 82 and 83: important nutrient. However, many <
Page 84 and 85: caries, and more hardening
Page 86 and 87: controls. 106 Interestingly, childr
Page 88 and 89: A few years later, additional resea
Page 92 and 93: Description of Pop
Page 94 and 95: Influence
Page 96 and 97: D has an effect on the dentition, p
Page 98 and 99: 14. Utiger RD. The need for more vi
Page 100 and 101: 42. Spiller WF, Jr. A clinical eval
Page 102 and 103: 67. Itota T, Tashiro Y, Torii Y, Ni
Page 104 and 105: 95. Bruszt P. Kariesuntersuchungen
Page 106 and 107: 121. Seow WK, Needleman HL, Holm IA
Page 108 and 109: Chapter 2 - Methods for a Prospecti
Page 110 and 111: Each participant also completed an
Page 112 and 113: Table 2.2 - Recognized case definit
Page 114 and 115: Population The target population fo
Page 116 and 117: allowed the project to suffer some
Page 118 and 119: women of limited m
Page 120 and 121: involving the surface of</s
Page 122 and 123: and training cup. In addition, each
Page 124 and 125: The questionnaire was administered
Page 126 and 127: statistics including frequencies an
Page 128 and 129: elationship between the two. Poisso
Page 130 and 131: References 1. Combs GF. Vit
Page 132 and 133: 27. Seow WK, Amaratunge A, Bennett
Page 134 and 135: Table 3.2 - Self-declared heritage
Page 136 and 137: Question Number Valid % Do you take
Page 138 and 139: Maternal Awareness
Page 140 and 141:
time of recruitmen
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Table 3.6- Prenatal</strong
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A total of 77 care
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indicated that they were receiving
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and 11 hypoplasia). DDEs were also
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Caries rates for children in this p
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Table 3.12 - Frequency of</
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Chapter 4 - Results: Associations <
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II. Relationship Between 25(OH)D Le
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Pregnancy & Health Prof</st
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The frequency of v
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did not (42.9 ± 21.2 nmol/L vs. 49
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Early Childhood Caries (ECC) Pr<str
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likely to have vitamin D levels at
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did not statistically differ from v
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Figure 4.4 - Predicted number <stro
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Table 4.7 - Relationship between ds
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frequently, and less likely to have
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health as average or poor, and more
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tail (p=.034) suggesting that early
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The next series of
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Results from the second model appea
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Table 5.6 - Multiple regression sta
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vitamin D. However, it did suggest
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significant at the bivariate level
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Table 5.12(a) - Logistic regression
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III. Multivariate Regression Analys
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Table 5.14 - Logistic regression fo
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Page 198 and 199:
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Table 5.19(b) - Backwards logistic
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Table 5.20(a) - Logistic regression
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Table 5.21 - Poisson regression for
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If enamel hypoplasia is present and
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Since maternal calcium levels were
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vitamin D deficiency, and rickets)
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to obtain it. Health literacy may b
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vitamin D from the diet as dietary
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home environment can be determinant
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hormone, calcium absorption and bon
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important nutrient. For many, this
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this potential relationship with ac
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genetic conditions, childhood medic
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vitamin D status including milk int
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y cariogenic oral flora. 49 Since e
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(49.8 ± 26.3 nmol/L vs. 45.1 ± 20
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pregnancy, which may have influence
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observed and it allowed multiple ou
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scientific evidence linking prenata
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cognizant that children who undergo
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preventive program would need to be
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makers in the formative stages. As
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Surprisingly, the final regression
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13. First Nations IaMHCCPS. <strong
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38. Hollis BW. Circulating 25-hydro
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65. Li Y, Navia JM, Bian JY. Caries
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91. Brownell MD, Guevremont A, Au W
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Chapter 7 - Conclusions This thesis
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This study provided an interesting
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esiding in disadvantaged communitie
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If you are taking vitamins during t
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Do you Eat? Never Rarely (less than
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What problems can early childhood c
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Prenatal Nutrition
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Did you ever add sugar to the bottl
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Enamel Hypoplasia Enamel Hypoplasia
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Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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