Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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Table 5.16 – Logistic regression for ECC* (excluding white spot lesions) – Infant feeding practices and soother use Variable Regression Coefficient (b) Standard Error b Standard Deviation <strong>of</strong> Variable in Adjusted Odds Ratio ± 95% Confidence Interval P value Age solids introduced (months) Infant Feeding (Bottle) (reference: mixed) Infant Feeding (Breast) (reference: mixed) Soother use (reference: no) Sample 0.027 0.080 2.82 0.93 0.60, 1.44 .74 -0.20 0.49 0.82 0.31, 2.16 .68 -0.80 1.27 0.45 0.038, 5.39 .53 -0.40 0.47 0.67 0.27, 1.67 .39 Sippy cup use (reference: no) *ECC reference = yes R 2 = 6.9% -1.02 0.79 0.36 0.076, 1.71 .20 Table 5.17 presents results from the logistic regression model that examined the relationship between personal and family finances and ECC. Specifically, it included both the annual income <strong>of</strong> the mother and overall household employment status <strong>of</strong> the mother during pregnancy. Results indicate that mothers with an annual income < $18,000 per year were not significantly more likely to have an infant with caries (p=.058). However, those who were from a household where someone was not employed full-time were over three times more likely to have a child with ECC (p=.031). Similar results also emerged from the backwards logistic regression model that included these same variables plus receiving government assistance. The final iteration <strong>of</strong> this backwards regression 5-18
only included annual income and overall household employment with both being significantly associated with ECC (p=.049 and p=.017, respectively) (data not shown). Table 5.17 – Logistic regression for ECC* (excluding white spot lesions) – Finances and full-time employment Variable Regression Coefficient (b) Standard Error b Adjusted Odds Ratio ± 95% Confidence Interval P value Low annual income (reference: > $18,000) No one with full-time employment in household (reference: no) *ECC reference = yes R 2 = 6.7% -1.90 1.00 0.15 0.021, 1.07 .058 1.31 0.60 3.70 1.13, 12.09 .031 Another model examined the relationship between ECC and the dental status <strong>of</strong> the infants and their family’s dental history and awareness (not shown). Initially, variables in this model were whether the infant had enamel hypoplasia, their age at the time <strong>of</strong> the dental examination (dichotomized), whether the infant had experienced a dental problem, whether the infant’s teeth were being brushed and cleaned, mother’s attitude <strong>of</strong> whether ECC is preventable and if the infant’s siblings had been diagnosed with ECC. However, since enamel hypoplasia is the strongest single predictor <strong>of</strong> ECC, enamel hypoplasia was removed from the model (Table 5.18). As reported earlier, infants with enamel hypoplasia were eight times more likely to suffer from ECC. Results from Table 5.18 indicate that infants who were ≥ 14 months old were 5.8 (1/0.17) times more likely to have ECC (p=.047). 5-19
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Influence
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Abstract Influence
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Acknowledgements This longitudinal
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Table of Contents
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Status and Non-<st
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Table 1.1-1 - Recent studies report
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Table 1.1-2 - Previous used terms f
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The Link Between Enamel Hypoplasia
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If enamel hypoplasia results from n
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malocclusions. 145 Additional human
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Daily intakes of v
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13. Milnes AR, Rubin CW, Karpa M, T
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40. Definition of
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71. Watt RG. From victim blaming to
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97. Fearne JM, Bryan EM, Elliman AM
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124. Burt BA, Pai S. Does low birth
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151. Tinanoff N, D
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Section 2 - Risk Factors for Enamel
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correspond to the timing of
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een published. 3,4,17,19 The intent
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Table 1.2-2 - Risk factors for DDE
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Opacity 51 Implicated Risk Developm
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Implicated Risk Factor Factors asso
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o Metabolic Abnormalities and Healt
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12 months of life.
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Other Factors Some uncommon risk fa
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devastating problem of</str
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13. An epidemiological index <stron
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54. Seow WK, Perham S. Enamel hypop
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82. Berdal A, Papagerakis P, Hotton
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Vitamin D may be d
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have been used to prof</str
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health’. In addition, articles we
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36 month trial had ended. 45 The nu
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Recently, a cross-sectional study <
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andomized, placebo-controlled study
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Further, amelogenin and enamelin, p
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A study of Austral
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Both dietary interventions and supp
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incidence of carie
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important nutrient. However, many <
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caries, and more hardening
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controls. 106 Interestingly, childr
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A few years later, additional resea
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Delayed Tooth Eruption Vita
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Description of Pop
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Influence
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D has an effect on the dentition, p
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14. Utiger RD. The need for more vi
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42. Spiller WF, Jr. A clinical eval
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67. Itota T, Tashiro Y, Torii Y, Ni
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95. Bruszt P. Kariesuntersuchungen
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121. Seow WK, Needleman HL, Holm IA
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Chapter 2 - Methods for a Prospecti
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Each participant also completed an
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Table 2.2 - Recognized case definit
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Population The target population fo
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allowed the project to suffer some
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women of limited m
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involving the surface of</s
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and training cup. In addition, each
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The questionnaire was administered
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statistics including frequencies an
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elationship between the two. Poisso
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References 1. Combs GF. Vit
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27. Seow WK, Amaratunge A, Bennett
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Table 3.2 - Self-declared heritage
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Question Number Valid % Do you take
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Maternal Awareness
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time of recruitmen
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Table 3.6- Prenatal</strong
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A total of 77 care
Page 146 and 147: indicated that they were receiving
Page 148 and 149: and 11 hypoplasia). DDEs were also
Page 150 and 151: Caries rates for children in this p
Page 152 and 153: Table 3.12 - Frequency of</
Page 154 and 155: Chapter 4 - Results: Associations <
Page 156 and 157: II. Relationship Between 25(OH)D Le
Page 158 and 159: Pregnancy & Health Prof</st
Page 160 and 161: The frequency of v
Page 162 and 163: did not (42.9 ± 21.2 nmol/L vs. 49
Page 164 and 165: Early Childhood Caries (ECC) Pr<str
Page 166 and 167: likely to have vitamin D levels at
Page 168 and 169: did not statistically differ from v
Page 170 and 171: Figure 4.4 - Predicted number <stro
Page 172 and 173: Table 4.7 - Relationship between ds
Page 174 and 175: frequently, and less likely to have
Page 176 and 177: health as average or poor, and more
Page 178 and 179: tail (p=.034) suggesting that early
Page 180 and 181: The next series of
Page 182 and 183: Results from the second model appea
Page 184 and 185: Table 5.6 - Multiple regression sta
Page 186 and 187: vitamin D. However, it did suggest
Page 188 and 189: significant at the bivariate level
Page 190 and 191: Table 5.12(a) - Logistic regression
Page 192 and 193: III. Multivariate Regression Analys
Page 194 and 195: Table 5.14 - Logistic regression fo
Page 198 and 199: Table 5.18 - Logistic regression fo
Page 200 and 201: Table 5.19(b) - Backwards logistic
Page 202 and 203: Table 5.20(a) - Logistic regression
Page 204 and 205: Table 5.21 - Poisson regression for
Page 206 and 207: If enamel hypoplasia is present and
Page 208 and 209: Since maternal calcium levels were
Page 210 and 211: vitamin D deficiency, and rickets)
Page 212 and 213: to obtain it. Health literacy may b
Page 214 and 215: vitamin D from the diet as dietary
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Page 218 and 219: hormone, calcium absorption and bon
Page 220 and 221: important nutrient. For many, this
Page 222 and 223: this potential relationship with ac
Page 224 and 225: genetic conditions, childhood medic
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Page 228 and 229: y cariogenic oral flora. 49 Since e
Page 230 and 231: (49.8 ± 26.3 nmol/L vs. 45.1 ± 20
Page 232 and 233: pregnancy, which may have influence
Page 234 and 235: observed and it allowed multiple ou
Page 236 and 237: scientific evidence linking prenata
Page 238 and 239: cognizant that children who undergo
Page 240 and 241: preventive program would need to be
Page 242 and 243: makers in the formative stages. As
Page 244 and 245: Surprisingly, the final regression
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13. First Nations IaMHCCPS. <strong
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38. Hollis BW. Circulating 25-hydro
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65. Li Y, Navia JM, Bian JY. Caries
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91. Brownell MD, Guevremont A, Au W
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Chapter 7 - Conclusions This thesis
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This study provided an interesting
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esiding in disadvantaged communitie
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If you are taking vitamins during t
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Do you Eat? Never Rarely (less than
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What problems can early childhood c
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Prenatal Nutrition
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Did you ever add sugar to the bottl
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Enamel Hypoplasia Enamel Hypoplasia
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Influence of Maternal Prenatal Vitamin D Status on Infant Oral Health

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