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Appendix D Food Codes for NHANES - OEHHA

Appendix D Food Codes for NHANES - OEHHA

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Scientific Review PanelSRP Draft Version 2 February,June 2012<br />

Del Bubba et al. is a similar study by the same research group that includes<br />

additional participants from rural areas. The use of nonsmoking rural women<br />

should reduce confounding contributions from the inhalation pathway. Airborne<br />

concentrations of PAHs tend to be higher in urban areas due to mobile sources.<br />

Based on the estimated intake of the same measured PAHs in dietary studies<br />

and the PAHs found in breast milk from nonsmoking mothers (Del Bubba et al.,<br />

2005; Zanieri et al., 2007), <strong>OEHHA</strong> was able to estimate transfer coefficients<br />

(Tco) by Equation J-8, a version of Equation J-1:<br />

Tcohmoi = Cmoi /(Doi) Eq. J-<br />

8<br />

where:<br />

Cmoi = geometric average ith PAH concentration in mother’s milk (µg per<br />

kg milk as wet weight)<br />

Doi = geometric average dose of the ith PAH per day from dietary sources<br />

(µg/day)<br />

Cmoi is the geometric average of the ith PAH in whole milk from nonsmoking,<br />

rural dwelling women. <strong>OEHHA</strong> obtained estimates of GM and GSD by pooling<br />

and converting arithmetic estimates to geometric estimates of the mean and<br />

standard deviation from two studies of nonsmoking rural-dwelling women (Del<br />

Bubba et al., 2005; Zanieri et al., 2007). Doi is the geometric average of the ith<br />

PAH taken in through dietary sources. Oral PAH Tcos <strong>for</strong> both carcinogenic and<br />

noncancer PAHs are shown in Table J.3-6.<br />

The Italian dietary study by Lodovici et al. (1995) supplied data in which <strong>OEHHA</strong><br />

could calculate estimates of dietary intake of nine PAHs among a population<br />

living mostly in urban settings. <strong>OEHHA</strong> obtained GM and GSD estimates by<br />

converting arithmetic estimates of dietary intake reported in Lodovici et al (1995)<br />

and estimates of intake variability from Buiatti et al (1989).<br />

These investigators estimated that the entire study population consumes about<br />

1.9 µg of carcinogenic PAHs per day from dietary sources. Approximately 46% of<br />

the total carcinogenic PAH intake comes from cereal products, non-barbecued<br />

meat, oils and fats. Even though meat barbecued on wood charcoal has the<br />

highest PAH levels, the contribution of these barbecued foods is only about 13%<br />

of the carcinogenic PAH intake.<br />

A limitation of the Italian dietary intake study is that the population examined was<br />

58% men, and the study did not report any body weight adjustments. Thus, the<br />

sample population may not represent the female population sampled by Zanieri<br />

et al (2007). Other studies that have compared dietary PAH intake levels<br />

between men and women indicate that men consume slightly higher levels of<br />

J-30

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