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Handbook of Vitamin C Research

Handbook of Vitamin C Research

Handbook of Vitamin C Research

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120Ana I. Haza, Almudena García and Paloma Morales[40] Kerr, J.F., Wyllie, A.H. & Currie, A.R. (1972). Apoptosis: a basic biologicalphenomenon with wide-ranging implications in tissue kinetics. British Journal <strong>of</strong>Cancer, 26, 239-257.[41] Holme, J.A., Gorria, M., Arlt, V.M., Ovrebø, S., Solhaug, A., Tekpli, X., Landvik,N.E., Huc, L., Fardel, O. & Lagadic-Gossmann, D. (2007). Different mechanismsinvolved in apoptosis following exposure to benzo[a]pyrene in F258 and Hepa1c1c7cells. Chemico-biological Interactions, 167, 41-55.[42] Hashimoto, T., Sano, T., Ito, W., Kanazawa, K., Danno, G.I. & Ashida, H. (2004). 3-Amino-1,4-dimethyl-5H-pyrido[4,3-b]indole induces apoptosis and necrosis withactivation <strong>of</strong> different caspases in rat splenocytes. Bioscience, Biotechnology, andBiochemistry, 68, 964-967.[43] Shiotani, B. & Ashida, H. (2004). 3-Amino-1,4-dimethyl-5H-pyrido[4,3-b]indole (Trp-P-1) triggers apoptosis by DNA double-strand breaks caused by inhibition <strong>of</strong>topoisomerase I. Carcinogenesis, 25, 1149-1155.[44] García, A., Morales, P., Arranz, N., Delgado, E., Rafter, J. & Haza, A.I. 2008.Induction <strong>of</strong> apoptosis and reactive oxygen species production by N-Nitrosopiperidineand N-Nitrosodibutylamine in human leukemia cells. Journal <strong>of</strong> Applied Toxicology,28, 455-465.[45] García, A., Morales, P., Arranz, N., Delgado, E., Rafter, J. & Haza, A.I. (2009). N-Nitrosopiperidine and N-Nitrosodibutylamine induce apoptosis in HepG2 cells viacaspase dependent pathway. Annals <strong>of</strong> the New York Academy <strong>of</strong> Science, (submittedfor publication).[46] Arranz, N., Morales, P., García, A., Rafter, J. & Haza, A.I. (2009). Induction <strong>of</strong>apoptosis by N-nitrosopyrrolidine and N-nitrosodimethylamine in human hepatoma andleukemia cells. Journal <strong>of</strong> Biochemical and Molecular Toxicology, (submitted forpublication).[47] D‘Agostini, F., Izzotti, A., Balansky, R.M., Bennicelli, C. & De Flora, S. (2005).Modulation <strong>of</strong> apoptosis by cancer chemopreventive agents. Mutation <strong>Research</strong>, 591,173-186.[48] Kim, J.G., Koh, S.H., Lee, Y.J., Lee, K.Y., Kim, Y., Kim, S., Lee, M.K. & Kim, S.H.(2005). Differential effects <strong>of</strong> dialllyl disulfide on neuronal cells depend on itsconcentration. Toxicology, 211, 86-96.[49] Koh, S.H., Kwon, H., Park, K.H., Ko, J.K., Kim, J.H., Hwang, M.S., Yum, Y.N., Kim,O.H., Kim, J., Kim, H.T., Do, B.R., Kim, K.S., Kim, H., Roh, H., Yu, H.J., Jung, K.H.& Kim, S.H. (2005). Protective effect <strong>of</strong> diallyl disulfide on oxidative stress-injuredneuronally differenciated PC12 cells. Molecular Brain <strong>Research</strong>, 133, 176-186.[50] Revel, A., Raanani, H., Younglai, E., Xu, J., Han, R., Savouret, J.F. & Casper, R.F.(2001). Resveratrol, a natural aryl hydrocarbon receptor antagonist, protects spermfrom DNA damage and apoptosis caused by benzo(a)pyrene. Reproductive Toxicology,15, 479-486.[51] Revel, A., Raanani, H., Younglai, E., Xu, J., Han, R., Savouret, J.F. & Casper, R.F.(2003). Resveratrol, a natural aryl hydrocarbon receptor antagonist, protects lung fromDNA damage and apoptosis caused by benzo(a)pyrene. Journal <strong>of</strong> Applied Toxicology,23, 255-261.

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