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Handbook of Vitamin C Research

Handbook of Vitamin C Research

Handbook of Vitamin C Research

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258Jun Yang, Jiaren Liu and John Parry[71] Park, S-W. & Lee, S-M. (2008). Antioxidant and prooxidant properties <strong>of</strong> ascorbic acidon hepatic dysfunction induced by cold ischemia/reperfusion. Eur J Pharmacol, 580,401-406.[72] Mix, J. A. (1999). Do megadoses <strong>of</strong> vitamin C compromise folic acid‘s role in themetabolism <strong>of</strong> plasma homocysteine. Nutr Res, 19, 161-165.[73] Herbert, V., Jacob, E. & Wong, K-TJ. (1976). Destruction <strong>of</strong> vitamin B 12 by vitamin C.Am J Clin Nutr, 30, 297-299.[74] Herbert, V., Jacob. E., Wong, K-TJ., Scott, J. & Pfeffer, R. D. (1978). Low serumvitamin B 12 levels in patients receiving ascorbic acid in megadoses: studies concerningthe effect <strong>of</strong> ascorbate on radioisotope vitamin B 12 assay. Am J Clin Nutr, 31, 253-258.[75] Gr<strong>of</strong>f, JL. & Gropper, SS. Advanced Nutrition and Human Metabolism. (3rd ed.).Wadsworth/Thompson Learning: Belmont, CA. pp 295-302. 1999.[76] Frank, J., Flaccus, A., Schwarz, C., Lambert, C. & Biesalski, H. K. (2006). Ascorbicacid suppresses cell death in rat DS-sarcoma cancer cells induced by 5-aminolevulinicacid-based photodynamic therapy. Free Radical Biol Med, 40, 827-836.[77] Rees, D. C., Kelsey, H. & Richards, J. D. M. (1993). Acute haemolysis induced by highdose ascorbic acid in glucose-6-phosphate dehydrogenase deficiency. BMJ 306, 841-842.[78] Udomratn, T., Steinberg, M. H., Campbell, G. D. & Oelshlegel, F. J. (1977). Effects <strong>of</strong>ascorbic acid on glucose-6-phosphatase dehydrogenase-deficient erythrocytes: studiesin animal model. Blood, 49, 471-475.[79] Michael, A. J. (1978). Kidney stone hospitalization in relation to vitamin Cconsumption in Austrailia 1966-1978. Commun Health Studies, 2, 9-13.[80] Schmidt, K-H., Hagmaier, V., Hornig, D. H., Vuilleumier, J-P. & Rutishauser, G.(1981). Urinary oxalate excretion after large intakes <strong>of</strong> ascorbic acid in man. Am J ClinNutr, 34, 305-311.[81] Adams, A. & de Kimpe, N. (2009). Formation <strong>of</strong> pyrazines from ascorbic acid andamino acids under dry roasting conditions. Food Chem doi: 10.1016/j.foodchem.[82] Limacher, A., Kerler, J., Conde-Petit, B. & Blank, I. (2007). Formation <strong>of</strong> furan andmethylfuran from ascorbic acid in model food systems. Food Additives andContaminants, 24 (suppl 1), 143.[83] Park, J. B. & Levine, M. (2000). Intracellular Accumulation <strong>of</strong> Ascorbic Acid IsInhibited by Flavonoids via Blocking <strong>of</strong> Dehydroascorbic Acid and Ascorbic AcidUptakes in HL-60, U937 and Jurkat Cells. Journal <strong>of</strong> Nutrition. 130, 1297-1302.[84] Song, J., Kwon, O., Chen, S., Daruwala, R., Eck, P., Park, J. B. & Levine, M. (2002).Flavonoid Inhibition <strong>of</strong> Sodium-dependent <strong>Vitamin</strong> C Transporter 1 (SVCT1) andGlucose Transporter Is<strong>of</strong>orm 2 (GLUT2), Intestinal Transporters for <strong>Vitamin</strong> C andGlucose. J. Biol. Chem., 277, 18, 15252-15260.[85] Biondi, C., Pavan, B., Dalpiaz, A., Medici, S. & Lunghi, L. (2007). Expression andcharacterization <strong>of</strong> vitamin C transporter in the human trophoblast cell line HTR-8/SVneo: effect <strong>of</strong> steroids, flavonoids and NSAIDs. MHR, 13, 77-83.

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