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Handbook of Vitamin C Research

Handbook of Vitamin C Research

Handbook of Vitamin C Research

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Pro-Oxidant vs. Antioxidant Effects <strong>of</strong> <strong>Vitamin</strong> C 175[32] Dawkins, R.(1982/1999). The Extended Phenotype: The long Reach <strong>of</strong> the Gene,Oxford, New York: Oxford University Press; 1999[33] Deng X.S. & Tuo J.S. & Poulsen H.E. & L<strong>of</strong>t S. (1998). Prevention <strong>of</strong> oxidative DNAdamage in rats by Brussels sprouts. Free Radic Res., 28, 323–33.[34] Deutsch J.C. (1998). Ascorbic acid oxidation by hydrogen peroxide. Anal biochem,255, 1-7.[35] Diaz, Z.& Colombo, M.& Mann, K.K. et al. (2005). Trolox selectively enhancesarsenic-mediated oxidative stress and apoptosis in APL and other malignant cell lines.Blood, 105, 1237–1245.[36] Dietary Reference Intakes for <strong>Vitamin</strong> C, <strong>Vitamin</strong> E, Selenium, and Carotenoids(2000). A report <strong>of</strong> the panel on dietary antioxidants and related comounds,Subcomitees on upper reference levels <strong>of</strong> nutrients and interpretation and uses <strong>of</strong>dietary reference intakes, and the standing comitee on the scientific evaluation <strong>of</strong>dietary reference intakes, Food and Nutrition Board, Institute <strong>of</strong> Medicine, NationalAcademy Press, Washington, D.C. From internet: http:books.nap.edu/openbook.php?record_id=9810[37] Doroshow, J.H. (2006). Redox modulation <strong>of</strong> chemotherapy-induced tumor cell killingand normal tissue toxicity. J Natl Cancer Inst, 98, 223–225.[38] Dreosti I.E. (1991). Free radical pathology and the genome. In: Dreosti, IE (Eds.), Traceelements, micronutrients, and free radicals (149-171).New Jersey, The Humana Press.[39] Drouin, R.& Rodriguez, H. & Gao, S. W. & Gebreyes, Z. & O‘Connor, T. R. &Holmquist, G. P. & Akman, S. A. (1996). Cupric ion/ascorbate/hydrogen peroxideinducedDNA damage: DNA-bound copper ion primarily induces base modifications.Free Rad. Biol. Med. 21, 261–273 39.[40] Enstrom J (2008). The epidemiology <strong>of</strong> vitamin C. Elsevier.[41] Enstrom, J. & Kanim, L. & Klein, M.A. (1992). <strong>Vitamin</strong> C intake and mortality smongo samplc <strong>of</strong> thc Unit?d States populetion. Epidcmiology 3, 194-202.[42] Fiala, E. S. & Sodum, R. S. & Bhattacharya, M. & and Li, H. (1996). Epigallocatechingallate, a polyphenolic tea antioxidant, inhibits peroxynitrite-mediated formation <strong>of</strong> 8-oxodeoxyguanosine and 3-nitrotyrosine. Experientia 52, 922–926.[43] Fischer-Nielsen, A. & Poulsen, H. E., & L<strong>of</strong>t, S. (1992). 8-HydroxydeoxyguanosineHydroxydeoxyguanosine in vitro: effects <strong>of</strong> glutathione, ascorbate, and 5-aminosalicylic acid. Free Rad. Biol. Med. 13, 121–126 40.[44] Food and Agriculture Organization/ World Health Organization (2004):Fruit andVegetables for Health: Report <strong>of</strong> a Joint FAO/WHO Workshop, 1-3 September 2004,Kobe, Japan.[45] Food and Drug Administration. November 1-3. (1993). FDA Conference onAntioxidant <strong>Vitamin</strong>s and Cancer and Cardiovascular Disease. Washington. DC: Office<strong>of</strong> Special Nutritionals, 1993( transcript).[46] Fraga, C. G. & Motchnik, P. A. & Shigenaga, M. K. & Helbock, H. J. & Jacob, R. A. &Ames, B. N. (1991). Ascorbic acid protects against endogenous oxidative DNA damagein human sperm. Proc. Natl. Acad Sci. U. S. A. 88, 11003–11006.

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