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Handbook of Vitamin C Research

Handbook of Vitamin C Research

Handbook of Vitamin C Research

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124Ana I. Haza, Almudena García and Paloma Morales[96] Qi, W., Russel, J.R., Dun-Xian, T., Joaquin, J.G., Lucien, C.M., Malgorzata, K. &Juan, R.C. (2000). Chromiium(III)-induced 8-hydroxydeoxyguanosine in DNA and itsreduction by antioxidants: comparative effects <strong>of</strong> melatonin, ascorbate, and vitamin E.Environmental Health Perspectives, 108, 399–402.[97] Cheng, T.J., Kao, H.P., Chan, C.C. & Chang, W.P. (2003). Effects <strong>of</strong> ozone on DNAsingle-strand breaks and 8-oxoguanine formation in A549 cells. Environmental<strong>Research</strong>, 93, 279–284.[98] Rovichová, S., Slamenová, D., Chalupa, I. & Sebová, L. (2004). DNA lesions andcytogenetic changes induced by N-nitrosomorpholine in HepG2, V79 and VH10 cells:the protective effects <strong>of</strong> vitamins A, C and E. Mutation <strong>Research</strong>, 560, 91–99.[99] Uhal, BD. (1997). Cell cycle kinetics in the alveolar epithelium. The American Journal<strong>of</strong> Physiology (Lung Cellular and Molecular Physiology), 272, 1031-1045.[100] Green, D.R. & Reed, J.C. (1998). Mitochondria and apoptosis. Science, 281, 1309-1312.[101] Rich, T., Allen, R.L. & Wyllie, A.H. (2000). Defying death after DNA damage.Nature,407, 777-783.[102] Landvik, N.E., Gorria, M., Arlt, V.M., Asare, N., Solhaug, A., Lagadic- Gossmann, D.& Holme, J.A. (2007). Effects <strong>of</strong> nitrated-polycyclic aromatic hydrocarbons and dieselexhaust particle extracts on cell signaling related to apoptosis: Possible implications fortheir mutagenic and carcinogenic effects. Toxicology, 231, 159-174.[103] Gosslau, A. & Chen, K.Y. (2004). Nutraceuticals, apoptosis, and disease prevention.Nutrition, 20, 95-102.[104] Blasiak, J., Trzeciak, A., Dziki, A., Ulanska, J. & Pander, B. (2000). Synergestic effect<strong>of</strong> vitamin C on DNA damage induced by cadmium. General Physiology andBiophysics, 19, 373-379.[105] Bhat, S.H., Azmi, A.S., Hanif, S. & Hadi, S.M. (2006). Ascorbic acid mobilizesendogenous copper in human peripheral lymphocytes leading to oxidative DNAbreakage: a putative mechanism for anticancer properties. The International Journal <strong>of</strong>Biochemistry & Cell Biology, 38, 2074-2081.[106] Bram, S., Froussard, P. & Guichard, M. (1980). Ascorbic acid preferential toxicity formalignant melanoma cells. Nature, 284, 629- 631.[107] Park, S., Han, S.S., Park, C.H., Hahm, E.R., Lee, S.J., Park, H.K., Lee, S.H., Kim,W.S., Jung, C.W., Park, K., Riordan, H.D., Kimler, B.F., Kim, K. & Lee, J.H. (2004).L-Ascorbic acid induces apoptosis in acute myeloid leukemia cells via hydrogenperoxide-mediated mechanisms. The International Journal <strong>of</strong> Biochemistry & CellBiology, 36, 2180-2195.[108] Karasavvas, N., Cárcamo, J.M., Stratis, G. & Golde, D.W. (2005). <strong>Vitamin</strong> C protectsHL60 and U266 cells from arsenic toxicity. Blood, 105, 4004-4012.[109] Wenzel, U., Nickel, A., Kuntz, S. & Daniel, H. (2004). Ascorbic acid suppresses druginducedapoptosis in human colon cancer cells by scavenging mitochondrial superoxideanions. Carcinogenesis, 25, 703-712.

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