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Hibbs, J. B., Jr., R. R. Taintor, et al. (1988). "Nitric oxide: a cytotoxic activated macrophage
effector molecule." Biochemical & Biophysical Research Communications 157(1):
87-94.
Hibbs, J. B., Jr., Z. Vavrin, et al. (1987). "L-arginine is required for expression of the
activated macrophage effector mechanism causing selective metabolic inhibition in
target cells." Journal of Immunology 138(2):
l-5-0-6:'
Hickey,M.J.,K.A@.''Induciblenitricoxidesynthase-deficientmice
have enhanced leukocyte-endothelium interactions in endotoxemia." FASEB Journal
ll(r2):955-64.
Hierholzer, C., B. Harbrecht, et al. (1998). "Essential role of induced nitric oxide in the
initiation of the inflammatory response after hemorrhagic shock." Journal of
Experimental Medicine 187(6): 917 -28'
Higgins, B. G., J. R. B"'tton, et al. (1992). "Comparison of bronchial reactivity and peak
expiratory flow variability measurements for epidemiologic studies." American
Review of Respiratory Disease 145(3): 588-93'
uiggin,ffital.(1989).''Thedistributionofpeakexpiratory!l-y
variability in a population sample." American Review of Respiratory Disease 140(5):
1368-72.
Higgins, B. G., J. R. Britton, et al. (1993). "Factors affecting peak expiratory flow variability
and bronchial reactivity in a random population sample"'Thorax 48(9): 899-905'
Hiltermann, T. J., J. Stolk, et at. (tggg). "Asihma severity and susceptibility to air pollution"'
European Respiratory Journal f 1(3): 686-93'
Hirata,K.,R'ru,offi995}-Intriuitionofendothelialnitricoxidesynthaseactivityby
protein kinase C." Hypertension 25(2):180-5'
"Exhaled nitric oxide is not elevated in the inflammatory
Ho, L. p., J. A. Innes, "t iltg98)t
airways diseases of cystic fibrosis and bronchiectasis." European Respiratory Journal
12(6): 1290-4.
Ho, L. P., F. T. Wood, et al. (2000). "Atopy influences exhaled nitric oxide levels in adult
asthmatics." b! 118(5): L327-31.
Ho, L. P., F. T. Wood, et al. (2000). "The culrent
exhales nitric oxide is affected by airway
single exhalation method of measuring
calibre." European Respiratory Journal
15(6): 1009-13.
Hobbs, M. R., V. Udhayakumar, et al. (2002). "A new NOS2 promoter polymorphism
associated with increased nitric oxide production and protection from severe malaria
in Tanzanian and Kenyan children." Lancet 3ffi(9344):1468-75.
Hoekstra, M. O., M. H. Grol, et al. (1998). Tosinophil and mast cell parameters in children
with stable moderate asthma." Pediatric Allergy & tmmunology 9(3):143-9'
Hogg, J. C. (Lggl). "The pathology of asthma." APMIS 105(10): 735-45.
uoliian, vt., t. Holmkvist, et at. (zoo2). "Increased nitric oxide elimination from the ainvays
after smoking cessation." Clinical Science 103(l): 15-9'
Hogman, M., T. UJlmtvist, "t ul. tZOOa. g*t"nded NO analysis appligd- to patients with
COpD, allergic asthma and allergic rhinitis." Respiratory Medipine 96(1): 24-30'
Hogman, M., S. Stiomberg, et al. (19-97). "Nitric oxide from the human respiratory tract
efficiently quantifiei by standardized single breath measurements." Acta Physiologica
Scandinavica. APr: 159(a): 345-6.
HotU"rg, C. f.3. l;Wti-gnt, et ai. (1993). "Child day care, smoking by caregivers' and lower
respiratory tract il'iness in the rirsi 3 years of life. Group Health Medical Associates."
Pediatrics 91(5): 885-92.
Holcroft, C. A., E. A.'Eisen, et al. (2003). "Measurement characteristics of peak expiratory
flow." Chest 124(2): 50 1- 10.
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