SLEEP 2011 Abstract Supplement

More documents

Recommendations

Info

B. Clinical Sleep Science III. Sleep Disorders – Insomnia Scale (PSAS) before bedtime. HRV and EEG spectrum analyses were conducted for the EKG and EEG data collected during the 5 minutes prior to sleep onset and the 5 minutes after. Results: Both cognitive and somatic subscales ratings were higher in insomnia group. Also, insomnia group showed higher alpha power both prior and after sleep onset, and higher beta power after fallen asleep (C3: F=4.78, p=.037). In terms of the correlations between PSAS score and EEG power, insomnia group showed significant negative correlation between somatic arousal and delta power (C3: r=-.56, p=.029; C4:r=- .53, p=.042), positive correlation between somatic arousal and alpha power (C3: r=.57, p=.027; C4:r=.56, p=.029), negative correlation between cognitive arousal and beta power (C3: r=-.55, p=.032; C4:r=-.53, p=.042) before sleep onset. After sleep onset, insomnia group showed positive correlation between somatic arousal and theta power (C3: r=.62, p=.015; C4:r=.54, p=.038). As regarding HRV, only correlation between somatic arousal and normalized high frequency (HF(nu)) in insomnia group after sleep onset (r=.52, p=.049) was significant. No significant correlations were obtained in normal sleepers. Conclusion: The results indicate that subjective somatic arousal is associated with cortical arousal as measured by EEG and ANS arousal by HRV in insomnia group, but not in good sleepers. Subjective feelings of cognitive arousal may be less associated with either ANS or cortical arousal. 0550 PATIENTS’ ATTRIBUTIONS ABOUT THE CAUSES OF INSOMNIA Harvey AG 1 , Morin CM 2 , Bélanger L 2 , Lombrozo T 1 , Rifkin JB 1 , Talbot LS 1 , Eidelman P 1 , Beaulieu-Bonneau S 2 , Fortier-Brochu E 2 , Soehner A 1 1 University of California, Berkeley, Berkeley, CA, USA, 2 École de Psychologie, Université Laval, Laval, QC, Canada Introduction: This study investigated the possibility that the ‘folk theories’ or attributions a patient holds about their disorder may impact their treatment preference. This is important because treatment preference may influence engagement and compliance with treatment which, in turn, will impact outcome. Insomnia is the focus as it is prevalent and associated with significant impairment of quality of life. Moreover, there are various treatment options. Methods: Participants were 69 patients with chronic insomnia. They completed the ‘Causal Attributions of My Insomnia Questionnaire’ (CAM-I), a questionnaire evaluating potential causal explanations/attributions about insomnia on Likert-type scales (1 = very likely; 7 = very unlikely). Several other assessment instruments were administered as part of a larger treatment study (e.g., Duke Interview for Sleep Disorders, SCID-I). Results: Of 12 factors rated, emotions and thinking patterns were most commonly endorsed as ‘contributing to insomnia’ (60% and 58% of patients, respectively, endorsed these factors as ‘very likely’). Patients were reasonably confident that targeting these factors in treatment would alleviate their insomnia (emotions M = 5.96; SD = 1.38; thinking patterns M = 6.00; SD = 1.25). Percentage of patients who rated that a psychological treatment could help varied from 30% for genetic factors to 59% for emotions. Patients who rated that a biological treatment could help varied from 19% for environmental factors to 40% for biochemical factors. Those who endorsed thinking patterns (t=6.57, p < .001), environment (t=4.50, p < .001), scheduling (t=4.29, p < .001) and emotions (t=5.76, p < .001) as likely contributors were more confident that a psychological than biological treatment would help. Surprisingly, no contributing factor assessed (hormones, arousal, genetics) were endorsed as more likely to be alleviated by a biological treatment. Conclusion: Patient’s attributions influenced treatment choice. Patients were more confident in psychological treatments over biological treatments. Support (If Any): 5R01MH079188-02 0551 SLEEP VARIABLES RELATED TO SLEEP QUALITY RATING Woosley J 1 , Lichstein KL 1 , Thomas J 1 , Taylor DJ 2 , Riedel BW 3 , Bush AJ 4 1 Psychology, University of Alabama, Tuscaloosa, AL, USA, 2 University of North Texas, Denton, TX, USA, 3 University of Memphis, Memphis, TN, USA, 4 University of Tennessee, Memphis, Memphis, TN, USA Introduction: Sleep researchers frequently use the term sleep quality, which lacks an established definition. The purpose of the present study is to determine which sleep variables are most closely associated with perceived sleep quality in people not having insomnia (PNI) and in people with insomnia (PWI). Methods: Using random-digit dialing, we recruited 772 participants ranging in age from 20 to 80+, with approximately 50 men and 50 women in each decade. Participants completed 2 weeks of sleep diaries which included a daily sleep quality rating (SQR) on a scale from 1 (poor sleep) to 5 (excellent sleep). The present sample included PNI (n = 400) and PWI (n = 137). PNI satisfied ICSD insomnia diagnostic criteria. Excluded were individuals who were partial qualifiers for PNI and PWI. Results: Stepwise multiple regression found that for PNI, sleep efficiency (SE) best predicted SQR, explaining 7.3% of variance in SQR. Number of awakenings (NWAK) explained an additional 1.8% of variance. Sleep onset latency (SOL), wake time after sleep onset (WASO), and total sleep time (TST) were excluded. For PWI, WASO was the best predictor of SQR, explaining 20.8% of the variance in SQR. Together, NWAK and TST explained an additional 9.9% of variance. SE and SOL were excluded. Conclusion: NWAK was the only sleep variable that predicted a significant amount of variance for both PNI and PWI. SE was the best predictor of SQR for PNI, whereas WASO was the best predictor of SQR for PWI. 91% of variability in SQR among PNI remained unexplained after accounting for self-reported sleep patterns, and 70% of SQR was unexplained among PWI. Most of perceived sleep quality may be a function of sleep stages and factors unrelated to sleep pattern and architecture sometimes grouped under the umbrella of nonrestorative sleep. Support (If Any): Research supported by National Institute on Aging grants AG12136 and AG14738. 0552 THE EFFECTS OF SLEEP DISTURBANCE ON PSYCHIATRIC AND COGNITIVE FUNCTIONING FOLLOWING TRAUMATIC BRAIN INJURY Orff HJ 1,2,3 , Jak A 1,2 , Twamley E 1,2 1 Dept. of Psychiatry, University of California, San Diego, San Diego, CA, USA, 2 Psychology Service, VA San Diego HCS, San Diego, CA, USA, 3 Research Service, VA San Diego HCS, San Diego, CA, USA Introduction: Traumatic brain injury (TBI) is a leading cause of death and permanent disability in the US each year and is considered the hallmark injury of the wars in Iraq and Afghanistan. Insomnia following TBI is one of the most common and yet least studied of the post-TBI sequelae and may contribute to residual mood and cognitive difficulties in these patients. We evaluated these factors in a sample of Veterans with mild to moderate TBI (mTBI). Methods: Retrospective chart reviews of Veterans’ clinical sleep, psychiatric, and neuropsychological test data were conducted. Bivariate correlations were performed to assess the relationship between sleep and measures of cognitive and emotional functioning. Results: 316 Veterans (mean age=32 yrs; mean education=13.5 yrs) were evaluated. 85% of patients reported sleep disturbance as measured by scores on Item #16 on the Beck Depression Inventory (BDI), with 71% of the total sample reporting getting less sleep. In a subset of patients (n=94) who were administered the Pittsburgh Sleep Quality Inventory (PSQI), 98% reported clinically significant sleep disturbance (PSQI SLEEP, Volume 34, Abstract Supplement, 2011 A188
B. Clinical Sleep Science III. Sleep Disorders – Insomnia >5). Self-reported sleep latencies averaged 51 minutes, total sleep times averaged 5.4 hours, and sleep efficiencies averaged 77%. Total PSQI scores were positively correlated with BDI total score (.547, p
Page 1:
JOURNAL OF SLEEP AND SLEEP DISORDER
Page 4 and 5:
EDITORIAL In June of 1986, roughly
Page 6 and 7:
A. Basic Science I. Pharmacology an
Page 8 and 9:
Page 10 and 11:
Page 12 and 13:
A. Basic Science II. Cell and Molec
Page 14 and 15:
Page 16 and 17:
Page 18 and 19:
Page 20 and 21:
Page 22 and 23:
Page 24 and 25:
Page 26 and 27:
A. Basic Science III. Ontogeny/Agin
Page 28 and 29:
Page 30 and 31:
Page 32 and 33:
A. Basic Science IV. Neurobiology S
Page 34 and 35:
A. Basic Science IV. Neurobiology o
Page 36 and 37:
A. Basic Science IV. Neurobiology
Page 38 and 39:
A. Basic Science IV. Neurobiology h
Page 40 and 41:
A. Basic Science IV. Neurobiology C
Page 42 and 43:
A. Basic Science IV. Neurobiology t
Page 44 and 45:
A. Basic Science V. Physiology 0110
Page 46 and 47:
A. Basic Science V. Physiology comp
Page 48 and 49:
A. Basic Science V. Physiology Resu
Page 50 and 51:
A. Basic Science V. Physiology Intr
Page 52 and 53:
A. Basic Science V. Physiology 0135
Page 54 and 55:
A. Basic Science V. Physiology Conc
Page 56 and 57:
A. Basic Science V. Physiology infl
Page 58 and 59:
A. Basic Science VI. Chronobiology
Page 60 and 61:
Page 62 and 63:
Page 64 and 65:
Page 66 and 67:
Page 68 and 69:
Page 70 and 71:
A. Basic Science VIII. Behavior 018
Page 72 and 73:
A. Basic Science VIII. Behavior 019
Page 74 and 75:
A. Basic Science VIII. Behavior a l
Page 76 and 77:
A. Basic Science VIII. Behavior Res
Page 78 and 79:
A. Basic Science IX. Learning, Memo
Page 80 and 81:
Page 82 and 83:
Page 84 and 85:
Page 86 and 87:
Page 88 and 89:
Page 90 and 91:
Page 92 and 93:
Page 94 and 95:
A. Basic Science X. Dreaming 0261 P
Page 96 and 97:
A. Basic Science XI. Sleep Deprivat
Page 98 and 99:
Page 100 and 101:
Page 102 and 103:
Page 104 and 105:
Page 106 and 107:
Page 108 and 109:
Page 110 and 111:
Page 112 and 113:
A. Basic Science XII. Instrumentati
Page 114 and 115:
Page 116 and 117:
Page 118 and 119:
B. Clinical Sleep Science I. Sleep
Page 120 and 121:
Page 122 and 123:
Page 124 and 125:
Page 126 and 127:
Page 128 and 129:
Page 130 and 131:
Page 132 and 133:
Page 134 and 135:
Page 136 and 137:
Page 138 and 139:
Page 140 and 141: B. Clinical Sleep Science I. Sleep
Page 164 and 165: B. Clinical Sleep Science II. Sleep
Page 172 and 173: B. Clinical Sleep Science III. Slee
Page 196 and 197: B. Clinical Sleep Science IV. Sleep
Page 202 and 203: B. Clinical Sleep Science V. Sleep
Page 210 and 211: B. Clinical Sleep Science VI. Sleep
Page 218 and 219: B. Clinical Sleep Science VII. Neur
Page 224 and 225: B. Clinical Sleep Science VIII. Med
Page 240 and 241:
B. Clinical Sleep Science VIII. Med
Page 242 and 243:
B. Clinical Sleep Science VIII. Med
Page 244 and 245:
B. Clinical Sleep Science IX. Psych
Page 246 and 247:
Page 248 and 249:
Page 250 and 251:
Page 252 and 253:
Page 254 and 255:
Page 256 and 257:
Page 258 and 259:
Page 260 and 261:
Page 262 and 263:
B. Clinical Sleep Science X. Normal
Page 264 and 265:
Page 266 and 267:
Page 268 and 269:
Page 270 and 271:
B. Clinical Sleep Science XI. Pedia
Page 272 and 273:
Page 274 and 275:
Page 276 and 277:
Page 278 and 279:
Page 280 and 281:
Page 282 and 283:
Page 284 and 285:
Page 286 and 287:
Page 288 and 289:
Page 290 and 291:
Page 292 and 293:
Page 294 and 295:
Page 296 and 297:
Page 298 and 299:
Page 300 and 301:
Page 302 and 303:
Page 304 and 305:
B. Clinical Sleep Science XII. Slee
Page 306 and 307:
Page 308 and 309:
Page 310 and 311:
B. Clinical Sleep Science XIII. Sle
Page 312 and 313:
Page 314 and 315:
Page 316 and 317:
Page 318 and 319:
Page 320 and 321:
Page 322 and 323:
Page 324 and 325:
Page 326 and 327:
B. Clinical Sleep Science XIV. Inst
Page 328 and 329:
Page 330 and 331:
Page 332 and 333:
Page 334 and 335:
Page 336 and 337:
B. Clinical Sleep Science XV. Healt
Page 338 and 339:
Page 340 and 341:
Page 342 and 343:
Page 344 and 345:
Page 346 and 347:
j u m p t o : A - B - C - D - E - F
Page 348 and 349:
j u m p t o : A - B - C - D - E - F
Page 350 and 351:
j u m p t o : A - B - C - D - E - F
Page 352 and 353:
j u m p t o : A - B - C - D - E - F
Page 354 and 355:
j u m p t o : A - B - C - D - E - F
Page 356 and 357:
j u m p t o : A - B - C - D - E - F
Page 358 and 359:
j u m p t o : A - B - C - D - E - F
Page 360 and 361:
j u m p t o : A - B - C - D - E - F
Page 362 and 363:
j u m p t o : A - B - C - D - E - F
Page 364 and 365:
j u m p t o : A - B - C - D - E - F
Page 366 and 367:
j u m p t o : A - B - C - D - E - F
Page 368 and 369:
j u m p t o : A - B - C - D - E - F
Page 370 and 371:
j u m p t o : 2 - A - B - C - D - E
Page 372 and 373:
j u m p t o : 2 - A - B - C - D - E
Page 374 and 375:
j u m p t o : 2 - A - B - C - D - E
Page 376 and 377:
j u m p t o : 2 - A - B - C - D - E
Page 378 and 379:
j u m p t o : 2 - A - B - C - D - E
Page 380 and 381:
j u m p t o : 2 - A - B - C - D - E
show all

SLEEP 2011 Abstract Supplement

You also want an ePaper? Increase the reach of your titles

Delete template?

Save as template?