11.07.2015 Views

Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

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5. Alcohol 89individual with a history <strong>of</strong> alcoholism are early clues suggestive <strong>of</strong> alcoholinduc<strong>ed</strong>persisting dementia.Alcohol-Induc<strong>ed</strong> Anxiety, Affective, or Psychotic DisorderIf symptoms <strong>of</strong> an anxiety disorder, affective disorder (depressive, manic, ormix<strong>ed</strong>), or psychotic disorder (hallucinations or delusions) develop during orwithin 1 month <strong>of</strong> intoxication or withdrawal, an alcohol-induc<strong>ed</strong> anxiety,affective, or psychotic disorder may be diagnos<strong>ed</strong>. If the patient has insight thathallucinations are alcohol-induc<strong>ed</strong>, an alcohol-induc<strong>ed</strong> psychotic disorder isnot diagnos<strong>ed</strong>. The anxiety and affective symptoms must exce<strong>ed</strong> the usual presentation<strong>of</strong> such symptoms as they commonly occur during intoxication orwithdrawal (DSM-IV).These disorders must be distinguish<strong>ed</strong> from comorbid psychiatric disorders(see section on comorbidity). A careful history eliciting the onset and thecourse <strong>of</strong> symptoms during abstinence or reexposure to alcohol will help distinguishalcohol-induc<strong>ed</strong> syndromes from psychiatric comorbidity.Alcohol-Induc<strong>ed</strong> Sleep DisorderAlcohol consum<strong>ed</strong> at b<strong>ed</strong>time may decrease the time requir<strong>ed</strong> to fall asleep buttypically disrupts the second half <strong>of</strong> the sleep cycle, resulting in subsequent daytimefatigue and sleepiness. Even a moderate dose <strong>of</strong> alcohol consum<strong>ed</strong> within6 hours prior to b<strong>ed</strong>time can increase wakefulness during the second half <strong>of</strong>sleep (Vitiello, 1997). Alcohol use prior to b<strong>ed</strong>time will also aggravate obstructivesleep apnea, and heavy drinkers or those with alcoholism are at increas<strong>ed</strong>risk for sleep apnea. Patients with severe obstructive sleep apnea are at a fivefoldincreas<strong>ed</strong> risk for fatigue-relat<strong>ed</strong> traffic crashes if they consume two or mor<strong>ed</strong>rinks per day compar<strong>ed</strong> to obstructive sleep apnea patients who consume littleor no alcohol (Bassetti & Aldrich, 1996).In alcoholics, heavy drinking eventually leads to increas<strong>ed</strong> time requir<strong>ed</strong>to fall asleep, frequent awakenings, and a decrease in subjective quality <strong>of</strong> sleep.Slow-wave sleep is interrupt<strong>ed</strong>, and during periods <strong>of</strong> withdrawal there is pronounc<strong>ed</strong>insomnia and increas<strong>ed</strong> rapid eye movement (REM) sleep. Followingwithdrawal from alcohol, sleep patterns may be abnormal, even following years<strong>of</strong> abstinence.Alcohol-Induc<strong>ed</strong> Sexual DysfunctionSexual dysfunction refers to impairment in sexual desire, arousal, or orgasm, orpresence <strong>of</strong> pain associat<strong>ed</strong> with intercourse as a result <strong>of</strong> alcohol use. Alcoholinduc<strong>ed</strong>sexual dysfunction differs from a primary sexual disorder in thatimprovement would be expect<strong>ed</strong> with abstinence from alcohol.

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