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Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

Clinical Textbook of Addictive Disorders 3rd ed - R. Frances, S. Miller, A. Mack (Guilford, 2005) WW

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25. Adolescent Substance Abuse 5671993; Crumley, 1990; Kaminer, 1996). Possible mechanisms for this relationshipsinclude acute and chronic effects <strong>of</strong> psychoactive substances. Adolescentsuicide victims are frequently using alcohol or other drugs at the time <strong>of</strong> suicide(Brent, Perper, & Allman, 1987). The acute substance use may produce transientbut intense dysphoric states, disinhibition, impair<strong>ed</strong> judgment, and increas<strong>ed</strong>level <strong>of</strong> impulsivity or may exacerbate preexisting psychopathology,including depression or anxiety disorders.A number <strong>of</strong> studies <strong>of</strong> clinical populations show high rates <strong>of</strong> anxiety disordersamong youth with SUDs (Clark et al., 1995; Clark & Sayette, 1993). Inclinical populations <strong>of</strong> adolescents with SUDs, the prevalence <strong>of</strong> anxiety disorderrang<strong>ed</strong> from 7% to over 40% (Clark et al., 1995; DeMilio, 1989; Stowell,1991). The order <strong>of</strong> appearance <strong>of</strong> comorbid anxiety and SUD appears to bevariable, depending on the specific anxiety disorder. Social phobia usually prec<strong>ed</strong>esabuse, whereas panic and generaliz<strong>ed</strong> anxiety disorder more <strong>of</strong>ten followthe onset <strong>of</strong> a SUD (Kushner, Sher, & Beitman, 1990). Adolescents with SUDs<strong>of</strong>ten have a history <strong>of</strong> posttraumatic stress disorder (PTSD) following acute orchronic physical and sexual abuse (Clark et al., 1995; Van Hasselt, Null, Kempton,& Bukstein, 1993). Bulimia nervosa is also commonly associat<strong>ed</strong> with adolescentshaving substance use disorders (Bulik, 2002). SUDs are very commonamong individuals diagnos<strong>ed</strong> with schizophrenia (Kutcher, Kachur, & Marton,1992; Regier et al., 1990). Personality disorders (Cluster B in particular) amongadolescents with SUDs are highly prevalent (Grilo et al., 1995). Finally, as suggest<strong>ed</strong>by studies showing language deficits in youth affect<strong>ed</strong> by or at high riskfor SUDs, learning disabilities or disorders may also show an increas<strong>ed</strong> incidence<strong>of</strong> comorbidity (Moss, Kirisci, Gordon, & Tarter, 1994). Patients withcomorbid psychiatric disorders continue to be a challenge for clinicians andresearchers in the assessment and treatment domains.PREVENTIONEfforts to curtail substance abuse concentrate on activities design<strong>ed</strong> for supplyand-demandr<strong>ed</strong>uction. It has been report<strong>ed</strong> that the use <strong>of</strong> alcohol by youthsdeclines when either the price <strong>of</strong> alcoholic beverages or the legal drinking ageincreases (Coate & Grossman, 1987). Similarly, a r<strong>ed</strong>uction in car accidentsamong youth result<strong>ed</strong> from the increase <strong>of</strong> the minimum drinking age to 21(O’Malley & Wagenaar, 1991).The goal <strong>of</strong> primary prevention among children and adolescents is to deferor preclude initiation <strong>of</strong> gateway substances such as cigarettes, alcohol, andmarijuana. The traditional <strong>ed</strong>ucation program is a prevention strategy us<strong>ed</strong> toincrease knowl<strong>ed</strong>ge <strong>of</strong> the consequences <strong>of</strong> drug use. Investigators (Schinke,Botvin, & Orlani, 1991) found the assumption that increas<strong>ed</strong> knowl<strong>ed</strong>g<strong>ed</strong>ecreases drug use to be invalid. Affective <strong>ed</strong>ucation, which increases self-

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