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MAGICAL MEDICINE: HOW TO MAKE AN ILLNESS ... - Invest in ME

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56<br />

“the authors were allowed to present op<strong>in</strong>ions as facts and to ignore the many studies that underm<strong>in</strong>ed their<br />

hypothesis. Their lack of objectivity resulted <strong>in</strong> the publication of a poorly‐researched article which misrepresented the<br />

research and perpetuated myths. What happened to evidence‐based medic<strong>in</strong>e?” (EM Goudsmit PhD)<br />

“Barsky and Borus managed to omit several hundred of peer‐reviewed articles document<strong>in</strong>g physiologic bases for<br />

illnesses such as the chronic fatigue syndrome. Even the review of the psychological literature left out articles<br />

<strong>in</strong>consistent with Barsky and Borus’ speculations and sometime <strong>in</strong>accurately portrayed the research they <strong>in</strong>cluded”<br />

(TE Hedrick PhD)<br />

“I’ve never been able to determ<strong>in</strong>e how secondary ga<strong>in</strong>s that <strong>in</strong>clude f<strong>in</strong>ancial hardship, social isolation and<br />

reduced quality of life can perpetuate illness behaviour” (J McSherry MB ChB)<br />

“The authors claim that (CFS) has ‘enough <strong>in</strong> common’ with other syndromes for them to be lumped<br />

together. S<strong>in</strong>ce when was ‘enough’ a suitable quantification to pass peer review?” (K Clemenger BS)<br />

“A shock<strong>in</strong>g article appeared <strong>in</strong> a widely read medical journal that seemed to turn back history. The authors argued<br />

that all somatic illnesses, those without a clear explanation of the cause, are fake. Such diseases, these psychiatrists<br />

argued, are little more than the expression of unhappy people who are desperate for attention. The authors further<br />

stated that doctors who appear to be ‘sympathetic’ to such patients only encourage these bogus maladies to persist”<br />

(Faces of CFS: Case Histories of Chronic Fatigue Syndrome. David S Bell. Lyndonville Publications, New<br />

York, 2000).<br />

Given the well‐known and resolute commitment of the PACE Trial Pr<strong>in</strong>cipal <strong>Invest</strong>igators (Professors<br />

Sharpe, White and Chalder) – and their powerful paymasters the medical and permanent health <strong>in</strong>surance<br />

<strong>in</strong>dustry ‐‐ to recategoris<strong>in</strong>g “CFS/<strong>ME</strong>” as a mental disorder, and given the Wessely School’s success <strong>in</strong><br />

ensur<strong>in</strong>g that the NICE Guidel<strong>in</strong>e rejected the ICD‐10 neurological classification that has held good for the<br />

last 40 years, and given the PACE Trial team’s forecast that the outcome of the Trial will <strong>in</strong>form any future<br />

revision of the NICE Guidel<strong>in</strong>e on “CFS/<strong>ME</strong>”, particularly given the content of the PACE Trial Manuals, it<br />

would be foolhardy to hope that the CISSD report will be able to counter such a determ<strong>in</strong>ed strategy by<br />

such powerful and <strong>in</strong>fluential advocates.<br />

The CISSD project <strong>in</strong>cludes psychiatrists who do not believe patients and who seem to denigrate them,<br />

and who also ignore volum<strong>in</strong>ous research evidence, so the long‐runn<strong>in</strong>g battle of the Wessely School’s<br />

unproven beliefs versus biomedical science seems set to cont<strong>in</strong>ue.<br />

For additional <strong>in</strong>formation about the CISSD Project and on the progress of the DSM and ICD revision<br />

processes, see http://meagenda.wordpress.com/dsm‐v‐directory/ (to whom grateful acknowledgment is<br />

made).<br />

Attempts to reclassify irritable bowel syndrome (IBS) as a mental disorder<br />

Peter White’s call for the separation of PVFS from “CFS/<strong>ME</strong>” and for a consequent revision of the ICD<br />

taxonomy seems a clear <strong>in</strong>dication of his <strong>in</strong>tention to reclassify “CFS/<strong>ME</strong>” as a somatoform disorder, along<br />

with irritable bowel syndrome (IBS), which the Wessely School believe is also a somatoform disorder<br />

(Lancet 1999:354:936‐939) <strong>in</strong> defiance of the evidence that it is not, of which the follow<strong>in</strong>g are recent<br />

illustrations:<br />

• at the 68 th Annual Scientific Meet<strong>in</strong>g of the American College of Gastroenterology held <strong>in</strong> 2003 at<br />

Baltimore, important f<strong>in</strong>d<strong>in</strong>gs were presented by lead <strong>in</strong>vestigators from the University of Vermont<br />

(Peter Moses, Associated Professor of Medic<strong>in</strong>e and Director of Cl<strong>in</strong>ical Research <strong>in</strong> the Digestive<br />

Diseases, and Gary Mawe, Professor of Anatomy and Neurobiology): “Seroton<strong>in</strong> is a critical<br />

signall<strong>in</strong>g molecule necessary for normal gut function. Our f<strong>in</strong>d<strong>in</strong>g that key elements of seroton<strong>in</strong> signall<strong>in</strong>g

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