Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 100
X) - B – Hospitalization improves the Normal (Peak) Tension Glaucoma patients: The Etiologies
or Risk Factors 4, 5, 6, 7, 8, 9, 10, 15, 19, 20, only happen because the patient drinks or does something
in his environment, and these are the main etiologies to his migraines and Normal (Peak) Tension
Glaucoma. When hospitalized, these patients are in an environment without their pleasant drinks. Immediately
their intraocular pressures have no more peaks, their glaucoma stop their evolutions and their
migraines reduce. The normal tension glaucoma patients, while hospitalized, become better without
any medication, and their intraocular pressures measured in this environment are not their daily truth.
“Hager (I958) comments on the tendency for the ocular tension of glaucoma patients to fall when
they are admitted to hospital even when no treatment is being given.” (Leighton D A).
“Glaucoma patients showed a significant decrease in intraocular pressure during hospitalization. Although
this decrease was more pronounced among the treated patients, it was also present in nontreated
patients. Consequently, other factors than improved compliance during hospitalization must play a role
in this phenomenon.” (Haufschild T, Orgul S and Flammer J).
X) - C – Drinks timing influence on the glaucoma: Between the Normal (Peak) Tension Glaucoma
patients caused by the excessive ingestion of water or other liquids, we observed the bigger glaucomatous
disk’s cups among those who daily drank them after dinner, before the sleeping hour. The
patient that drinks excessively at morning and afternoon, but not after dinner, presented smaller Optic
Nerve’s damage. The water drank only at morning has lesser glaucomatous effect than the water drank
after dinner. We observed this liquids timing, but we could not measure it.
This timing influence is caused by the second peak of intraocular raised pressures. The ophthalmologists
know well the first raise of intraocular pressure which occur 15 minutes after drinking excessive
water (1,000 milliliters). After these drinks during the day, on the following night it occurs a second
peak of raised intraocular pressure, and this one is ignored by the physicians. This second sleeping
pressure is higher than the first peak, and it causes much more glaucomatous optic neuropathy. This
second sleeping peak of intraocular pressure is the main etiology to the “Normal” or “Low” pressure
glaucoma and to the next wakening migraines and other symptoms. It is caused by the sum of:
a- The absorption of the digestive fluids, drinks and meals when sleeping, because there was no time to
their absorption before sleep.
b- The spread to the entire body on horizontal position, of the water retained in the inferior part of the
body during the upright position.
c- The hydrostatic increase of the cranial venous pressure on the horizontal position.
d- The closed upper eyelid compressing the aqueous veins on the eye.
e- The excessive water drank few hours before sleeping, without time enough to be excreted.
f- The caffeine and beer toxic effects increasing the ocular blood capillaries exudation.
The intraocular pressure increase at night together with the smaller arterial pressure, turns smaller the
blood perfusion pressure in the eye and causes the glaucomatous ischemic lesion when the patient is
sleeping: “24 healthy individuals and 29 primary open-angle glaucoma patients underwent intraocular
pressure and blood pressure measurements every 2 h, starting at 08:00 h until 06:00 h of the next
morning. In primary open-angle glaucoma patients, the mean diastolic blood pressure... was
significantly lower at 04:00 h... and the mean diastolic perfusion pressure was significantly lower from
24:00 h to 06:00 h.” (Costa V P, and others).
We conclude that the sum of excessive liquid drinks and the intraocular pressure rise when the
patient sleeps causes more Glaucoma than the excessive liquid drinks alone.