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Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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<strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong> – Dr. Leonardo Izecksohn – page 151<br />

b- When there is a relative deficiency of outflow or resorption of these excessive fluids from that<br />

space, its overload causes both:<br />

b 1 - Edema in those tissues wich have free expansion possibilities.<br />

b 2 - Pressure rise in those closed inelastic spaces fluids filled. The raised fluids pressure stretch all<br />

the tissues, the nerves mainly at their laminas cribosas, the arteries reducing the arterial blood with<br />

oxygen, and the veins reducing their volumes. The migraines and variants are the feelings of all<br />

this.<br />

The water retention in the body of the migraine patients already was measured:<br />

“Bioelectric impedance assay demonstrated a slight total body water increase in alcohol-induced migraine<br />

patients.” (Martelletti P, and others).<br />

Many etiologies cause water retention through the arterial capillaries in the body and cause the fluids<br />

hypertension and migraines. The main ones are:<br />

-Caffeine.<br />

-Excessive water drank.<br />

-Vasopressin.<br />

-Wine.<br />

-Beer.<br />

-Estrogens.<br />

-Corticosteroids.<br />

-Toxins.<br />

The water retention in the entire body of migraineurs is stimulated by the increased production of the<br />

pituitary hormone Vasopressin: “During an attack, vasopressin was consistently raised (median (range)<br />

3.5 (1.2-9.6) pg/ml… The highest vasopressin concentration occurred in the only patient who vomited.<br />

The results suggest vasopressin rises during an attack of spontaneous migraine, and this may, in part, be<br />

related to emesis. In the majority, vasopressin levels only rose sufficiently to have some renal antidiuretic<br />

effect, although in some these levels could have been sufficient to cause alteration in peripheral<br />

blood flow. Release of vasopressin may be responsible for the facial pallor and antidiuresis observed in<br />

migraine.” (Hampton K K, and others).<br />

This vasopressin excessive secretion can be caused by the Pituitary gland been squeezed by the<br />

Cerebrospinal Fluid <strong>Hypertension</strong>, similarly as an squeezed sponge, and releasing its accumulated<br />

vasopressin hormone into the blood circulation.<br />

The Ayurveda medicine knows this for more than 2,000 years ago, and denominate it as “Pitta”: “<strong>Migraines</strong><br />

frequently happen when systemic Pitta moves into the cardiovascular system, circulates, and affects<br />

the blood vessels around the brain. The blood vessels dilate due to the hot, sharp quality of Pitta.<br />

This, in turn, creates pressure on the nerves, resulting in migraines. Pitta disorders are characterized by<br />

the red complexion and eyes, light sensitivity, burning sensation, anger, irritability, and nose bleeds.<br />

Liver and blood toxicity are often associated with these symptoms.” (Ayurveda).<br />

When the patient already has water retention, anyone of many triggers can rise the <strong>Fluids</strong>’ pressures<br />

and cause the migraines: “Coughing, sneezing, climbing stairs, or bending over increases headache<br />

pain by increasing intracranial pressure.” (Robertson Jr WC and Mack P). Without any previous water<br />

retention, these same activities cause nothing.<br />

Inside an inelastic closed space with constant volume, whether one expands, other shrinks:<br />

During a migraine, as there is increased secretion of the Cerebrospinal Fluid that raises its pressure,<br />

its volume also increases. As the skull and Dura mater space do not expand, the brain and the blood<br />

vessels are squeezed. Squeezing the vessels, the cerebral blood flow is reduced, the blood volume inside<br />

the brain reduces, and the brain oxygen consumption (extraction) from this restricted blood is increased.<br />

Bednarczyk already measured this:<br />

“In nine patients (seven female and two male), global cerebral blood flow (ml/min/100 g [SD]) was<br />

measured as 52.70 (6.9) during migraine and 59.65 (10.6) in the migraine-free state; p=0.028.

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