Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 196
5) Rapid improvement and shortened duration with corticosteroid therapy and;
6) Transient, reversible magnetic resonance image contrast enhancement of the affected cranial nerve
(86% of patients).” (McMillan H J, and others).
This magnetic resonance image visualization of the affected cranial nerve in ophthalmoplegic migraines
is an aseptic neuritis of this nerve. It is typically caused, so by the caffeine and so by the
Cerebrospinal fluid´s hypertension causing the nerve ischemia.
- Migraines, ocular paresis, beer, caffeine and excessive water: We had a disk jockey with 33-yearold,
mulatto (black, Indian and white ancestors), 1.71 meters (5 feet and 7 inches) height, 61 Kilograms
(134 pounds) of weight, almost all healthy, who suddenly presented with diplopia one month
ago. He also complained about bi-temporal and occipital headaches, mainly at awakening.
He used to drink coffee 250 milliliter (8 fluid ounces), cola soft drink 600 milliliter (more than a pint)
and water 4,800 milliliter (more than a gallon) each day, for the last 10 years, more or less. He also
used to drink some pints of beer on weekends, and for the wakening headaches, he self-medicated with
caffeinated over-the-counter analgesics.
On the ophthalmological examination, we found almost all normal, as pupils, visual acuity, biomicroscopy
and intraocular pressures. He presented a small paresis of a portion of the third cranial nerve
on the right side, which included the Superior Rectus and Levator Palpebrae Superioris. His Optic
Nerve’s´ disks show 0.6/3/2/0.25 right eye and 0.5/3/1/0.25 left eye (Cup diameter/ cup depth/ lamina
cribosa’s pores visibility/ borders edema), which is a damage suspicious of glaucoma and small
cerebrospinal fluid hypertension. After 1 month, stopping all caffeine, beer, the excessive water drank
daily, and medicating only with vitamins B1, B6 and B12, one tablet each day, he became better from
the headaches and the ocular paresis.
On the Children’s Hospital Los Angeles, “patients 11 years and younger diagnosed with idiopathic intracranial
hypertension... Of the 10 patients, four were girls and six were boys. Only one patient was
obese. The most common presenting symptoms were stiff neck (four patients) and diplopia (four patients).
Six of eight patients with strabismus had abducens nerve palsy (four bilateral), one patient had a
sensory exotropia, and one had a comitant esotropia. Visual field abnormalities were present in 11 of 13
eyes (85%), and severe visual loss resulting in no light perception vision occurred in one eye of one patient.
Resolution of papilledema occurred rapidly in all patients, with a mean of 4.7 +/- 2.6 months.
Resolution of sixth nerve palsy also occurred rapidly in four of six patients in a mean of 1.6 +/- 1.2
months. One patient required strabismus surgery for persistent esotropia.”(Cinciripini G S, and others).
On the Department of Ophthalmology, University of Arkansas Medical Center, USA, “Three children
with pseudotumor cerebri presented with vertical diplopia and clinical signs of fourth cranial nerve
palsy including a hypertropia of the affected eye, which increased with adduction and ipsilateral head
tilt. The fourth cranial nerve palsy resolved after reduction of the intracranial pressure in all three children.”
(Speer C, and others).
Adie's pupil – Tonic pupil. Adie’s tonic pupil is defined as a dilated pupil, constricting poorly to light
and exhibiting light-near dissociation. It is consequent to a parasympathetic fibers lesion. It can be
caused by the Cerebrospinal fluid hypertension or by the vasospasm and consequent ischemia caused
by the caffeine, in a person with some propensity to it. We had no cases with it.
XIII- g - Cerebrospinal Fluid Hypertension squeezing the 5 th cranial nerve and the Gasserian
ganglion: Trigeminal Neuralgias: The squeeze of the Trigeminal Semilunar ganglion (Gasserian ganglion)
by the Cerebrospinal Fluid Hypertension possibly is the main etiology of the Trigeminal Neuralgias.
The Trigeminal Semilunar ganglion is on the midway between the Cerebrospinal Fluid that compresses
its roots, and the Ophthalmic, Maxillary and Mandibular nerves on the outside and free from
any fluid´s pressure. The Gasserian ganglion is just like a cork holding the pressure in the bottle, which
is our head. Consequently, the main treatment to cure the Trigeminal Neuralgia is the treatment of the
Cerebrospinal Fluid's Hypertension. To cure it, it is not necessary to mutilate the patient, destroying the
ganglion or the nerve: it is only necessary to shorten the caffeine, beer, wine and excessive water from
his daily diet. So high suffering with so easy cure.