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Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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<strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong> – Dr. Leonardo Izecksohn – page 169<br />

3.The third effect of the Valsalva maneuver is the rise of the Cerebrospinal fluid pressure, consequent<br />

to the rise of the intra-cranial venous pressure. This can be felt as a headache. Whether this<br />

Cerebrospinal fluid hypertension reaches higher values than the arterial pressure inside the brain, it can<br />

cause ischemic damage anywhere in the brain.<br />

4.The fourth effect of the Valsalva maneuver is a reduction of the blood supply to the brain, with<br />

few oxygen and accumulation of carbonic gas: “Profound reductions in middle cerebral artery blood<br />

velocity (mean) were observed … during the maintained Valsalva maneuver (-21 +/- 3% together with<br />

an elevation in central venous pressure to 40 +/- 7 mmHg). Responses to performance of the Valsalva<br />

maneuver with and without exercise were similar…”(Pott F, and others).<br />

5.The fifth effect of the Valsalva maneuver is a stimulus to the right-to-left blood shunt in the<br />

heart which has patent foramen ovale, or in the lung which has artery-venous malformation. This<br />

causes the venous blood with excessive carbonic gas and few oxygen to be distributed to the brain and<br />

the whole body together with the arterial blood. It causes vasodilation in the brain’s arteries, auras and<br />

migraines, besides other pathologies.<br />

Other etiologies, similar to Valsalva maneuver, also raise the cranial venous pressures and can cause<br />

the glaucoma:<br />

A- Queckenstedt test.<br />

B- High resistance wind instrument playing.<br />

C- Sirsasana (Shirshásana) (headstand) yoga posture.<br />

D- Tight neckties.<br />

A- Queckenstedt test: The manual pressure applied to both jugular veins to elevate the cranial<br />

venous pressure, known as the “Queckenstedt test”, also cause the firsts three above effects, rising<br />

simultaneously the Cerebrospinal, Intraocular and Inner Ears <strong>Fluids</strong> pressures, and worsening the migraine's<br />

aches: “The Queckenstedt test (30 seconds on 39 patients with acute migraine attacks) aggravated<br />

headache intensity in both sitting and supine positions. The presence of throbbing pain and higher<br />

pain intensities was associated with the Queckenstedt test effect in the supine position.” (Chou C H,<br />

and others).<br />

B- High resistance wind instrument playing causing <strong>Normal</strong> (Peak) tension Glaucoma:<br />

“High and low resistance wind musicians experience a transient rise in their intraocular pressure<br />

while playing their instruments as a result least in part of uveal engorgement. The magnitude of intraocular<br />

pressure increase is greater in high resistance wind (trumpet and oboe) players. High resistance<br />

wind musicians had a small but significantly greater incidence of visual field loss than other musicians,<br />

which was related to life hours of playing. The cumulative effects of long-term intermittent intraocular<br />

pressure elevation during high resistance wind instrument playing may result in glaucomatous<br />

damage, which could be misdiagnosed as normal-tension glaucoma.” (Schuman JS, et al.)<br />

This is really a <strong>Normal</strong> (Peak) tension glaucoma!<br />

C- Sirsasana (Shirshásana) (headstand) yoga posture causing <strong>Normal</strong> (Peak) Tension Glaucoma:<br />

“There was a uniform 2-fold increase in the intraocular pressure during Sirsasana, which was maintained<br />

during the posture in all age groups irrespective of the ocular biometry and ultrasound pachymetry.”<br />

(Baskaran M, and others).<br />

D- Tight neckties causing <strong>Normal</strong> (Peak) Tension Glaucoma:<br />

“A tight necktie may cause an increase in Intraocular Pressure in susceptible individuals and should<br />

be included among the confounders of accurate intraocular pressure measurement and considered as a<br />

risk factor for increased intraocular pressure.” (Teng C, and others).<br />

This is a similar pathophysiology of the Queckenstedt test explained above.

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