Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 178 NAION: The ischemia causes the Optic Nerve’s infarct and more edema. The Optic nerve edema increases the pressure over the Optic Nerve in the scleral canal. This vicious cycle ensures the complete death of the Optic nerve's fibers in few minutes. The compressive arterial obstruction can be temporary, maybe few minutes, but the infarction is definitive. This only can happen when coincide all the above conditions on the same time, and possibly aggravated by some arteriosclerosis. The cerebrospinal fluid pressure doesn't need to be higher than the arterial pressure: It only needs to be high enough to cause the initial Optic nerve's edema, and the vicious cycle inside the scleral canal strangles the Optic nerve and completes the NAION. The NAION is associated with other health disturbs which are worsened by the caffeine, wine and beer: “The presence of other vascular conditions is frequent (with NAION), hypertension, 46.9%; diabetes, 23.9%; myocardial infarction, 11%”. (Younge B R). The best prevention of the NAION is to avoid drinking its etiologies: caffeine, wine, beer, and excessive water. “The casual definition of NAION is that of a sudden, painless, unilateral, irreversible ischemic event of the intraocular optic nerve without associated systemic disease, which has no effective treatment.” (Mathews, M K). The Optic Nerve’s infarction usually is painless, because usually it occurs when the patient sleeps, and sleeping the patient feels nothing. The mild Optic Nerve’s borders edema from the Cerebrospinal Fluid Hypertension previously to the NAION, is replaced by an enormous edema secondary to the Optic Nerve’s infarction. For the diagnose of the NAION etiology, it remains: - The mild borders edema and reduced cupping visible by direct ophthalmoscopy on the other eye’s Optic Nerve of the same patient, signal of his Cerebrospinal Fluid Hypertension. - Asking to the patient about his headaches, migraines, signs and symptoms from the Cerebrospinal Fluid Hypertension syndrome, felt days, weeks and years before the NAION. - Asking to the patient about his drinks during the last 2 days and nights before the NAION. The Optic Nerve's borders edema, from the Cerebrospinal Fluid Hypertension Syndrome, of around 1 Diopter height, before the occurrence of the NAION was denominated as “Incipient Nonarteritic Anterior Ischemic Optic Neuropathy”: “Incipient NAION is a distinct clinical entity, with asymptomatic optic disk edema and no visual loss attributable to NAION.” (Hayreh SS and Zimmerman MB). Other authors denominated it as “Optic Nerve’s Crowded Disk”. The NAION is a sickness similar to the hangover: both are caused by the excessive drinks few hours before, causing the acute Cerebrospinal Fluid Hypertension on the following night. Both are self-inflicted damage and mild forms of happy suicide. The Leber Hereditary Optic Neuropathy is probably caused by this Cerebrospinal Fluid Hypertension squeezing the Optic nerve, in a patient with the genetic susceptibility for this lesion. It is an hereditary variant of the NAION.
Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 179 Scheme XIII-1: Cerebrospinal fluid pressure higher than the intraocular pressure. This can cause: Squeezing the Lamina Cribosa towards inside the eye, and it bows and aches as Migraines. Squeezing the Optic Nerve’s Dura mater at the orbit, which swells and aches as migraines. Chronic edema of the Optic Nerve’s fibers inside the eye at the borders of the Optic nerve disk. Spreading these edemas through and under the retina, and through the choroids, to the posterior pole, causing many retinal and macular degeneration. Acute squeezing the Central Retinal Vein inside the Optic Nerve, causing its thrombosis. Chronic squeezing and raising the blood pressure inside the Central Retinal Vein and its branches inside the eye, causing its branches engorgement, hemorrhages, thrombosis, chronic edemas and degeneration at the Retina. Acute squeezing the Central Retinal Artery and the arterial ciliary branches that nourish the Optic Nerve; this can cause the Optic Nerve’s infarct (NAION). XIII - d – 3 - Cerebrospinal Fluid Hypertension squeezing the 2 nd cranial nerve - Acute Squeezing the Central Retinal Vein in the Optic Nerve. The acute Cerebrospinal Fluid Hypertension can cause acute squeezing of the Central Retinal Vein in the Optic Nerve, which causes the acute retention of blood and raised blood pressure in the Central Retinal Vein in the eye. This causes the acute venous engorgement and can result in the Central Retinal Vein Thrombosis, or thrombosis of one of its branches. The thrombosis causes hemorrhages, exudation, and damage in the eye. Central Retinal Vein Thrombosis caused by beer: We had a black 47-year-old patient, weighting 62 Kilograms (136 pounds), 1.72 meters (5 feet and 8 inches) tall, and arterial tension 120/80 mmHg. He once drank around 9.000 milliliter of beer at a two hours party, and after three days presented on our office with headaches, extreme engorgement of both eyes’ Central Retinal Veins, more than twenty intra-retinal hemorrhages in each eye and small edema of the borders of both Optic Nerve’s disks. The medicine classifies this as Central Retinal Vein Thrombosis in both eyes. There was no other neural damage. Since the first day, we prescribed him oral liquids restrain, no beer drinks, and medicated him with oral Acetazolamide 250 mg once a day. After two weeks, a cranial Computer Tomography with contrast shows no damage, which confirms the diagnosis of acute Cerebrospinal Fluid Hypertension Syndrome caused by the excessive beer, which injured his eyes and vanished. He presented slow recovering of all hemorrhagic damage in few months, but kept moderate Central Retinal Veins engorgement. He is still presenting normal visual acuity because he had not any macular damage. The Cerebrospinal fluid pressure rise can be caused by many etiologies. Probably the increased drinks of caffeine on winter caused the increased incidence of Central Retinal Vein thrombosis at Taiwan: “Our study (from the Taiwan National Health Insurance Research Database) demonstrates significant seasonal variations in the retinal vein occlusion incidence, with the peak occurrence in the winter month of January.” (Ho J D, and others).
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- 1 patient (7.7%) presented photop
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Advanced (Cup/Disk ratio=1) Fluids
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Migraines, Variants, Signs and Symp
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cornea aches and forces its owner t
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A(1)/A(2A) receptors.” (Nobrega J
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Urinary albumin excretion Albumin-c
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1. BadBoy 2. Burn 3. Café 4. Chá
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53. Tylenol DC Fluids Hypertension
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Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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