Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 158 “Information on the frequency of bowel movements was collected from 1971 to 1974 in 6790 men aged 51 to 75 years without Parkinson’s Disease in the Honolulu Heart Program. Follow-up for incident Parkinson’s Disease occurred over a 24-year period. Ninety-six men developed Parkinson’s disease an average of 12 years into follow-up…Incidence declined consistently from 18.9/10,000 person-years in men with 2/day. After adjustment … men with
Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 159 - Blurred vision. - Flashes of light, scintillations. - Hemianopia or temporary blindness. - Micropsia, macropsia, metamorphopsia. - Migrating scotoma (visual field defects). - Synesthesia: a mixing of senses during the aura. - Visual hallucinations. - Wavy or zigzag lines, usually colored lines (fortification spectra) (Teichopsia). Whether the ischemia occurs at another central nervous system place, the aura can present the corresponding symptom. Whether the ischemia is intense, it can cause a Transient Ischemic Attack, or there can be a definitive ischemic damage, as a brain stroke or a myocardial infarct. Usually, in less than 20 minutes after the visual aura, the patient has the rebound arterial dilatation, consequent fluids exudation, which results in rising the pressure inside that closed space: the eyes, the cerebrospinal fluid or inner ears spaces. Consequently, the stretched structure or nerve aches: the patient feels Migraine or a variant. Whether the pressure does not rise enough, there will be no migraine, and the patient with visual aura without Migraines is denominated with “Acephalgic Migraine’’. As the main etiology of an aura is the ischemia, other ischemic damages are more common on the patients with auras: myocardial infarction, brain, pontine and cerebellar infarcts (stroke). “Migraineurs, particularly with aura, have a higher cardiovascular risk profile than individuals without migraine.” (Scher A I, and others). “The risk of posterior circulation strokes, especially cerebellar, is increased in migraineurs with aura. Female migraineurs, with or without aura, have an increased risk of deep white matter brain lesions.” (Sahai-Srivastava S, and Cowan R). Caffeine is the most common etiology to the Auras and some cardiovascular diseases. - Other prodromes or premonitory signs and symptoms of migraines are: Non visual Auras caused by ischemia at another central nervous system places (discomfort, exhaustion, mood disorders, etc.). Unconscious feelings of the signs or symptoms of the surmounting pathophysiology that is rising the fluid pressures, and the patient unconsciously try to defend himself from the imminent migraine. migraines variants or diffuse pain, consequent to the Cerebrospinal Fluid, intraocular, or inner ears hypertension, and their neural reflexes. Sleeping is a self-defense measure, stopping the aches and neural reflexes. Vomits, increased urination, and diarrhea are the parasympathetic neural body defenses trying to get free from the body's excessive liquids and pressures. Diaschisis is a loss of some cerebral function and electrical activity in areas remote from the damage but neuronally connected to it. So, a brain ischemia can cause neurological dysfunction far from it. When the visual aura is intense and persistent, the brain edema can be visible on the Magnetic Resonance Image: “A 41-year-old woman with known basilar migraine for 5 years consulted about a persistent visual aura (visual snow phenomenon) plus bilateral paresthesias in the extremities for 4 days... An brain MRI detected ... an abnormal signal in the left occipital lobe in apparent water diffusion coefficient... A new brain MRI 8 days after did not show any previous lesion in the same sequences... These MRI signals during the aura, suggesting cytotoxic edema.” (Belvis R, and others). XII q) Only one etiology alone seldom is enough: When the patient has only one pure etiology to migraines, this usually can not cause the aches, and it is only a Risk Factor. “Vasoactive intestinal peptide mediates a marked dilatation of cranial arteries, but does not trigger migraine attacks in migraineurs. Vasoactive intestinal peptide induced a mild immediate headache. These data provide further evidence against a purely vascular origin of migraine.” (Rahmann A, and others).
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- 1 patient (7.7%) presented photop
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Advanced (Cup/Disk ratio=1) Fluids
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Migraines, Variants, Signs and Symp
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al from the caffeine. Fluids Hypert
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A(1)/A(2A) receptors.” (Nobrega J
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Urinary albumin excretion Albumin-c
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1. BadBoy 2. Burn 3. Café 4. Chá
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53. Tylenol DC Fluids Hypertension
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Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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