Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 157
Caffeine poisoning the nerve. Caffeine is a pro-inflammatory drug, because it antagonizes the
cellular A2A adenosine receptors, which reduce the inflammations. (Ohta A, and Sitkovsky M).
The Cerebrospinal Fluid Hypertension stretching and causing ischemia on the nerve.
Both etiologies together.
These aseptic neuritis improve stopping beer, caffeine, excessive water drinks and chocolate. Any
medication is complementary or useless.
XII k) Caffeine causes intraocular aqueous secretion and outflow disturbs:
We suspect that the chronic action of caffeine on the Trabecular meshwork of the Anterior chamber
inside the eye, on the Schlemm's canal and on the Aqueous veins, which drains the Aqueous humor and
controls the intraocular pressure, causes their definitive damage on the long run. Their chronic damage
and respective chronic Aqueous humor insufficient outflow, raises the intraocular pressure and after
years it causes the chronic open angle glaucoma.
The fetuses inside their mothers are more sensible to the caffeine intoxication, because they are forming
their bodies, and also because they can not detoxify the caffeine. The caffeine that the mother
drinks causes in her child the congenital propensity of glaucoma and high myopia, besides many other
lesions. This is not genetic: it is caused by the mother intoxicating her fetus with caffeine.
“Caffeine could elevate intraocular pressure by either increasing aqueous formation or inhibiting
drainage. As a phosphodiesterase inhibitor, caffeine may increase intracellular cyclic AMP, stimulating
the production of aqueous by the ciliary body…Caffeine could also inhibit aqueous drainage by decreasing
tone in smooth muscle cells of the angle via adenosine receptor blockade, with closure of the
fenestrae that drain aqueous into Schlemm's canal.” (Chandrasekaran S, and others).
XII l) Caffeine increases the dopamine receptors in the brain, which causes migraines:
“Prodromal symptomatology (mood changes, yawning, drowsiness, food craving), accompanying
symptoms (nausea, vomiting, hypotension) and postdromal symptoms (mood changes, drowsiness,
tiredness) may be related to dopaminergic activation. The dopaminergic system could also play a role
in the headache phase, either by taking part in nociception mechanisms, or by regulating cerebral blood
flow…Altered dopaminergic control of prolactin secretion exists in migrainous women…A high density
of lymphocytic (dopamine) D5 receptors has been found in migraine sufferers, thus suggesting their
upregulation. These findings support the view that hypersensitivity of peripheral and central dopaminergic
receptors is a specific migraine trait.” (Fanciullacci M, and others).
“It has been previously demonstrated how rats can develop behavioral dopamine supersensitivity
after long-term administration of caffeine… An increase of 126% in striatal dopamine D2(High) receptors
was found in caffeine-sensitized rats. This marked elevation in D2(High) receptors may account
for the caffeine-induced behavioral dopamine supersensitivity and may help elucidate the interactions
between caffeine and dopamine neurotransmission.” (Simola N, and others).
XII m)Chronic digestive intoxication causes migraines and neurological disorders:
We suspect that the toxins absorbed by the digestion of unhealthy foods, and by the chronic feces
(bowel) constipation, can cause vascular disturbs in the brain and consequent migraines from the
Cerebrospinal Fluid Hypertension. We also suspect that after many years, this chronic intoxication can
cause neurological disorders, as Parkinson’s disease, Multiple Sclerosis, and Amyotrophic Lateral
Sclerosis.