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Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

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<strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong> – Dr. Leonardo Izecksohn – page 166<br />

Scheme XII-2: The total intraocular pressure over the lamina cribosa increases in grams as increases<br />

its inner square millimeter surface, even with the same mmHg pressure. On a small Optic nerve disk<br />

with 2.15 square millimeter of area, an intraocular pressure of 16 mmHg causes 0.468 grams of pressure<br />

over the disk. On a glaucomatous disk, the same 16 mmHg of intraocular pressure causes more<br />

than 1.0 gram of pressure over the disk, and the lamina cribosa already is distended and weaker.<br />

We conclude that it is better to medicate the beginning of the raising intraocular pressure at the<br />

migraine phase and simultaneously to prevent the glaucoma, than years later to try to stop the<br />

progressive glaucomatous big cup already with visual lesion.<br />

XII y) Ocular <strong>Hypertension</strong> Syndrome. The patient who presents intraocular Aqueous Humor<br />

outflow or resorption deficiency has intraocular hypertension compared with the smaller<br />

Cerebrospinal fluid pressure at the other side. This causes the Optic Nerve’s disk squeeze from inside<br />

the eye towards the Optic Nerve. As bigger is this differential between the intraocular pressure and the<br />

Cerebrospinal fluid pressure, so bigger is the glaucomatous propensity. The patient feels this hydrodynamic<br />

squeeze mainly as Cluster (at the forehead or on the eyes) and Tension (at the nape) <strong>Migraines</strong><br />

or many other interchangeable signs or symptoms.<br />

This increased difference between the intraocular pressure and the Cerebrospinal fluid pressure on the<br />

Lamina cribosa's both sides, already was measured by lumbar puncture on 28 patients who had primary<br />

open-angle glaucoma:<br />

“The mean cerebrospinal fluid pressure was 13.0 mmHg in nonglaucoma patients and 9.2 mmHg in<br />

primary open-angle glaucoma patients. The cerebrospinal fluid pressure was lower in primary openangle<br />

glaucoma patients… Cup-to-disc ratio correlated independently with intraocular pressure,<br />

cerebrospinal fluid pressure, and the translaminar pressure difference... Larger cup-to-disc ratio was associated<br />

with lower cerebrospinal fluid pressure. Cerebrospinal fluid pressure is significantly lower in<br />

primary open-angle glaucoma patients compared with that in non-glaucomatous controls.” (Berdahl J P,<br />

and others).<br />

Whether this rise of intraocular pressure is too intense or remains too long, it causes ischemia of the<br />

Retinal ganglion cells and Optic Nerve’s fibers, it kills them and reduces the retinal nerve fiber layer<br />

thickness in the eyes, it increases the Optic nerve's cup and the pores visibility at the Lamina Cribosa,<br />

which results in visual field definitive partial loss, or say, in Glaucoma (Scheme IX-2, repeated).

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