Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...
Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...
Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...
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<strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong> – Dr. Leonardo Izecksohn – page 164<br />
- Whether the arterial blood has too few oxygen, the patient can suffer an ischemic brain stroke, or other<br />
damage.<br />
The patient with patent foramen ovale is lifelong prone to suffer the signs, symptoms and sicknesses<br />
of the <strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong>, more than other people without this cardiac damage.<br />
This risk factor (patent foramen ovale) causes more migraines when added with some condition<br />
which raise the venous blood pressure in the cardiac right atrium, which are:<br />
- Valsalva maneuver.<br />
- Breath with high-pressure air.<br />
- Pulmonary arteriovenous malformation.<br />
- Head-down positions.<br />
These are another risk factors. Together with the patent foramen ovale, they turn to be etiologies of<br />
migraines.<br />
In the patients with migraines, “Massive right-to-left shunt (patent foramen ovale) appeared in<br />
38.9% of migraine with aura and in 6.5% of migraine without aura. Migraine with aura patients identified<br />
at least one Valsalva-provoking activities as headache trigger in 45.8%.” (Tembl J, and others).<br />
The surgical closure of the cardiac patent foramen ovale and of the arterio-venous malformation improves<br />
or cures most migraines with aura:<br />
“Percutaneous transcatheter closure of patent interatrial communications results in significant amelioration<br />
of Migraine headache in 87% of patients (complete resolution in 24% and significant improvement<br />
in symptoms in 63%).” (Dubiel M, and others).<br />
Similar pathophysiology occurs with the Hereditary haemorrhagic telangiectasia, which “is a genetic<br />
disorder characterized by epistaxis, telangiectasia and visceral vascular manifestations. It is associated<br />
with migraine with aura due to pulmonary arteriovenous malformations. Lifetime prevalence of migraine<br />
was higher in Hereditary haemorrhagic telangiectasia patients (39.6%) than in controls<br />
(19.8%).” (Marziniak M, and others).<br />
XII t) Cardiac damage and hypothyroidism with and without blood shunting from right to left,<br />
and from left to right, cause the <strong>Fluids</strong> hypertension.<br />
“In 395 patients from the UCLA Adult Congenital Heart Disease Center,… the frequency of migraine<br />
headaches was 52% in the right-to-left shunt group, 44% in the left-to-right, and 38% in the no shunt<br />
group. In patients with a right-to-left shunt who underwent surgical repair, 47% had complete resolution<br />
of migraine headaches, whereas 76% experienced >50% reduction in headache days per month.”<br />
(Truong T, and others).<br />
The probable pathophysiology is that the congenital cardiac damage and hypothyroidism cause unstable<br />
supply of oxygenated blood to the brain, with occasional hypoxia (low O2), sometimes added<br />
with the accumulation of carbonic gas (CO2). The ischemia and/or the excess of carbonic gas causes<br />
the brain's vascular dilatation, the fluids’ pressures rises and downs, auras and migraines.<br />
XII u) Cranial (cerebral) venous sinus thrombosis and stenosis, and Jugular vein thrombosis<br />
cause the <strong>Fluids</strong> <strong>Hypertension</strong> <strong>Syndromes</strong>:<br />
The Cranial (cerebral) venous (dural) sinus thrombosis and the Jugular vein thrombosis are caused by<br />
cranial trauma, meningitis, and other etiologies. The venous sinus thrombosis can recover but it remains<br />
a sinus stenosis, verifiable by a magnetic resonance venography. The stenosis chronically difficult<br />
the venous blood return from the brain and eyes, causing chronic increased venous pressure and<br />
excessive fluids’ exudation. They can cause the Cerebrospinal Fluid’s and Ocular <strong>Hypertension</strong> syndromes,<br />
with respective migraines and variants:<br />
“Of patients with migraine, 6.7% had bilateral transverse sinus stenosis; 67.8% of these patients had<br />
idiopathic intracranial hypertension without papilledema”. (Bono F, and others).<br />
“Among the 198 patients with chronic tension-type headache who underwent magnetic resonance<br />
venography, 18 (9%) had bilateral transverse sinus stenosis. Thirteen of these 18 patients with bilateral<br />
transverse sinus stenosis underwent lumbar puncture, and nine (69.2%) had idiopathic intracranial hypertension<br />
without papilledema.”(Bono F, and others).