Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 136
c- Most damage is slowly progressive, and only become or is perceived as sicknesses after many
years of progression. There are many small glaucomatous damage (glaucomatous Optic neuropathy)
that still have not the size to be denominated as Glaucoma. After more some years, whether the damage
increases, then it will be classified as Glaucoma. However, the sickness has begun many years
before the classification.
Under the effect of a strong etiology, anyone patient can present the respective sickness suddenly, at
any age.
Typical is the age evolution of the signs and symptoms from the beer intolerance:
- On teenagers and at the twenties, the main manifestation of beer drinks is hangover.
- After some more years, beer drinks cause red eyes and headaches.
- After the fifties, the main manifestations are tearfulness, neck pain, cluster and tension migraines.
- After more some years of beer drinks, the result is the glaucoma.
Aging is an etiology that we cannot control, and it is associated with every other etiology. The signs
and symptoms of the Fluids Hypertension Syndromes that happen mainly in aged people are denominated
by some doctors as “late-life migrainous accompaniments”.
We surely would like it, but it is still impossible to say to the patient “You turn yourself younger 20
years from now”. I want to be younger again, too.
We conclude that aging is an etiology to the Fluids Hypertension Syndromes and to the caffeine
intolerance, which reduces the signs and symptoms and increases the definitive damage and sicknesses.
XI- 32) – Very low arterial pressure when sleeping or in surgeries:
When the patient is sleeping, whether the blood arterial pressure falls too much, and the perfusion
pressure becomes lower than the intraocular pressure, it can cause an ischemic “low-tension” glaucomatous
damage. This is one etiology to glaucoma, and not to the fluid's hypertension.
“Patients who had visual field loss progression showed significantly lower nocturnal blood pressure
variables, with the dips of the systolic, diastolic, and mean arterial pressure significantly larger. They
also had a greater history of disk hemorrhages. The nocturnal reduction in blood pressure may, therefore,
be an additional risk factor in glaucoma patients”. (Graham S L, and Drance S M).
Similar ischemic damage can happen in the brain, whether the blood perfusion pressure falls too
much, to values lesser than the Cerebrospinal fluid pressure.
XI- 33) – Vasoconstrictors, vasodilators, and others: The following etiologies, besides others, can
cause cranial vasodilation, or initial vasoconstriction followed by a rebound vasodilation, and consequent
migraines and headaches:
1) Adrenaline (Epinephrine); it is the main stress hormone: it is a vasoconstrictor.
2) Alcoholic (ethanol) drink: it is a vasodilator, and in high doses it causes arterial diastolic hypertension.
3) Analgesics: Most of them have caffeine, which is a vasoconstrictor, in the tablet. The caffeine withdrawal
causes brain vasodilation.
4) Amphetamines: they are vasoconstrictors.
5) Caffeine: it is a brain vasoconstrictor.
6) Calcitonin gene-related peptide (CGRP) causes dilatation of the cranial middle meningeal artery.
7) Carbachol: it is a cholinomimetic agonist, a cranial vasodilator.
8) Cardiac patent foramen ovale and pulmonary arterio-venous malformation: they cause vasodilation.
Their surgical closure can cause vasoconstriction and migraines after the surgery.
9) Cocaine: It is a strong vasoconstrictor. It causes much more lesions than the caffeine.
10) Cortisone (a stress hormone): it retains water and raises the fluids’ pressures.
11) Diving: it can cause breath with few oxygen and excessive carbonic gas; both are brain vasodilators.
12) Ergots: They are vasoconstrictors.
13) Fasting: It decreases the blood glucose and the arterial pressure, which cause vasodilation.