Fluids Hypertension Syndromes: Migraines, Headaches, Normal ...

Fluids Hypertension Syndromes – Dr. Leonardo Izecksohn – page 160
The patient with cardiac patent foramen ovale is lifelong with this risk factor to migraines. Whether
she is a woman, her estrogens are one more risk factor. Whether she drinks caffeine, or beer, or excessive
water, the adding up of all simultaneous risk factors become etiologies and cause migraines.
We conclude that to feel migraines the patient must have two or more risk factors simultaneously.
The research that isolate the patient from his daily drinks and give him only one risk factor, usually
do not obtain any migraine, unless this risk factor is taken in a big dose.
Even the caffeine needs some individual predisposition to cause its bad effect: “Available evidences
showed that caffeine had different effects on intraocular pressure in different groups of individuals. For
normal individuals, intraocular pressure was not changed by ingestion of caffeine, while for patients
with glaucoma or ocular hypertension, intraocular pressure increased significantly.” (Li M, and others).
XII r) Allodynia, Neural referred (reflexes) pain, neuropathic pain, muscle tenderness, other
signs and symptoms:
Many migraines, variants signs and symptoms are worsened by the central nervous system spreading
reflex orders to other nerves that ache, or cause vasodilation, or vasoconstriction, or increase secretions,
or cause muscular tenderness, or cause other sign or symptom.
These reflexes are the nervous system reacting to defend himself, and also warning the conscious individual
that his body is suffering and it needs help. The aching nerve can be other than the stretched
nerve, and this is one way that the central nervous system manifests its sufferings.
Many aches are caused by the Allodynia. “Cutaneous allodynia is pain resulting from a nonnoxious
stimulus to normal skin.” “In 79% of the patients, migraine was associated with cutaneous allodynia.”
(Burstein R, and others).
Many hemicranias are allodynia (physiologic sensations transformed in pain) and muscle tenderness:
On “25 patients with strictly unilateral migraine and 25 healthy subjects…pressure pain thresholds in
the neck there were significant differences between groups and sides,… while pressure pain thresholds
in the cephalic point showed differences between groups, but not sides… Patients had lower pressure
pain thresholds and increased pericranial tenderness on the symptomatic side… , whereas no significant
differences were identified between the non-symptomatic side and controls. The enhancement of
local tenderness scores was related to hyperesthesia of specific muscles (sternocleidomastoid, suboccipital,
and temporalis) rather than a generalized pericranial tenderness.” (Fernández-de-Las-Peñas C,
and others).
The allodynia in migraineurs with aura is progressive during an attack: “During a migraine attack in a
42-year-old male... Prior to the headache, he experienced visual, sensory, motor and speech aura. During
the headache, he experienced photo-, phono- and odour-phobia, nausea and vomiting, worsening of
the headache by coughing or moving his head, and cutaneous pain when shaving, combing his hair or
touching his scalp…
(i) After 1 h, mechanical and cold allodynia started to develop in the ipsilateral head.
(ii) After 2 h, this allodynia increased on the ipsilateral head and spread to the contralateral head and ipsilateral
forearm.
(iii) After 4 h, heat allodynia was also detected while mechanical and cold allodynia continued to increase.”
(Burstein R, and others).
We conclude that there are at least five types of neural signs and symptoms from the Fluids Hypertension
Syndromes and the caffeine intoxication:
1- Primary ache: It is secondary to a structure or nerve stretched by the Fluid's Hypertension or intoxicated
by the caffeine, and it aches. There are no primary aches (pain) without etiologies: all
aches and migraines are secondary to some disturb. The denomination as “primary” means that the
ache is the first symptom, and not that it has no etiology.