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Methods used to monitor pesticide poisoning Through this public-health network, pesticide poisoning is monitored at all levels (village, township, county, and province). In 1957, a reporting system for occupational diseases was established. Recording of pesticide poisonings began in 1961 in Shanghai and 1973 in Jiang-Su using the following procedures. Pesticide poisoning cases were first handled by the village "barefoot" doctors. After diagnosis and treatment, they were registered and reported directly to the county Health and Anti-Epidemic Station using a special index-card form. These cards were gathered and analyzed by the county station. Quarterly, they were sent with the occupational disease reports to the higher-level organization. The municipal or provincial station summarized the reports and calculated the incidence of poisoning cases and the resultant death rate. Etiologic and relevant factors were evaluated to modify programs and initiate special projects for further study. Research projects were undertaken to: survey hygienic conditions surrounding the use of pesticides in the field; assess exposure by measuring air concentration, skin contamination, blood cholinesterase activity and urinary changes; and observe any other adverse effects. Comparisons were made among sprayers in teams and among individual farmers. Organophosphates (e.g., dichiorvos, trichlorophon, dimethoate, parathion, methyl parathion, etc.) were measured in air samples collected in tubes containing silica gel. Cotton swab samples were used to assess dermal contamination by gas chromatography using flame photodetector (Wang and Du 1985). The activity of blood cholinesterase was determined by the modified Hestrin method. Chlordimeform in the air samples, cotton swabs, and urine samples was measured colorimetrically with chromogen naphthylene-ethyldiamine. Training courses to renew knowledge on prevention, diagnosis, and treatment of poisoning were delivered to personnel at various levels: physicians in county hospitals, hygienists in health stations, staff in township hospitals, and the rural paradoctors. These groups, in turn, educated rural people in preventive measures to be taken when using pesticides. As part of their training, "barefoot" doctors are required to complete a curriculum of courses and an internship of about 3 years. Diagnosis, treatment, and prevention of pesticide poisoning is one of the most important parts of their study. 231
Jiang-Su province Results and discussion By the late 1980s, the number of cases of work-related acute pesticide poisoning in Jiang-Su province had decreased gradually to half the number in the 1970s (Fig. 1). In 1981, the number of occupational poisonings tripled, but, because the number of people applying pesticides had increased five times, the overall incidence of poisoning cases was not elevated. However, after 1982, the number of nonoccupational poisonings remained steady at triple the 1970's rate. Fortunately, over the same period, the death rate due to nonoccupational poisoning decreased (Fig. 2). Thus, the overall mortality rate from pesticide poisoning did not increase at all. Of the 80 million people in the province of Jiang-Su, 70% live in rural areas; 70% of these people are engaged in agriculture as their main occupation. On average, 1.2-1.3 people/family are involved in the application of pesticides. The estimated rate of occupational poisoning among those exposed to pesticides is 0.4 per 1 000. The use of pesticides is an occupational hazard that has resulted in the loss of life, health, and economic productivity. Most of these incidents are easily preventable compared with other occupational hazards. In contrast, nonoccupational intoxication is a complex social and mentalhealth issue as most of the victims result from attempted suicide. Although Jiang-Su has a huge population, the incidence of nonoccupational intoxication was low. The crude mortality rate from nonoccupational pesticide poisoning 50 - Occupational Nonoccupational 0- 1973 1976 1979 1982 1985 1988 Fig. 1. Recorded cases of acute occupational and nonoccupational poisoning in Jiang-Su province, 1973-1988. 232
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Impact of pesticide use on health i
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IMPACT OF PESTICIDE USE ON HEALTH I
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©International Development Researc
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Contents Acknowledgments viii Forew
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Control of disease vectors: a curre
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Foreword From their first appearanc
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PART! EPIDEMIOLOGY OF PESTICIDE POI
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Although pesticides are used both i
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Table 1. Introduction of pesticides
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problem. The International Developm
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impact of pesticides on Canadian wi
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investigated further. For example,
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Conclusions The subject of this vol
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Jusoh Mamat, M.; Anas, A.N.; Heong,
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Health-risk assessment of pesticide
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especially in the absence of comple
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depends largely on external factors
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Specific situations may require spe
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that the best possible work is done
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doses (chronic poisoning) or delaye
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Table 1. Poisoning incidents in fou
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Pesticide research for public healt
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Current pest control practices in T
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800- 400- 200 - 1980 1981 1982 1983
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designed 30-40 years ago (Jusoh Mam
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and tore easily. One investigation
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education, mass-communication tools
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Increase and improve training activ
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Castaneda, C.P. 1988. Pesticide poi
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Ooi, A.C.P.; Heong, K.L.; Lim, B.K.
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Investigations into acute pyrethroi
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Level of exposure To assess the lev
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Table 2. Cases of acute pyrethroid
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(in 121 subjects), usually develope
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Deltamethrin was detectable in the
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pyrethroid poisoning. Therefore, th
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Gilliatt, R.W.; Willison, R.G. 1963
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Table 1. Value of imported pesticid
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Study two Twelve farmers participat
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All activities Left hand (73.2) Lef
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Acknowledgment- The three studies r
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Since the creation of Pakistan in 1
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Table 1. Incidents of pesticide poi
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Sandu, S.S.; Waters, M.D.; Simmon,
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and chlordane. Pest resistance to t
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young workers was 15.8%, 6.8%, and
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Storage of pesticides Although the
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Discussion The lowland regions and
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Pulmonary obstructive disease in a
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To investigate the presence and mea
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Age (years) Table 1. Distribution o
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was among paraquat users who smoked
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Levin, P.J.; Klaff, L.J.; Rose, A.G
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the delta. The total population of
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The successive and simultaneous rel
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on clinical studies and laboratory
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This made the collection of blood s
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Table 1. Relation between informati
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analysis, and engaging in various t
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Table 1. Value (million KSH) of pes
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To develop a health-education packa
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Storage site Table 3. Storage of ag
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Table 5. Causes of agrochemical and
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Methods A pilot field survey was un
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Conclusion The practices followed b
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manufactured or formulated locally,
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However, based on individual compla
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Table 4. Acetylcholinesterase activ
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Age did not appear to be an importa
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eactions to the pesticides in occup
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Bowers, M,B.; Goodman, E.; Sim, V.M
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Extent of exposure of farm workers
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Group III consisted of 31 farmers w
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25 - 0 Level of exposure J Group 1,
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Pesticides were stored everywhere c
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Table 2. Concluded. Question Answer
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PART II RELATING PESTICIDE DEVELOPM
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Toxicity Table 1. Requirements and
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Screening Process development Route
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Progress in improving safety Pestic
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type of formulation, and warning pr
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The pyrethroid, cypermethrin, is mo
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Studying the effects of pesticides
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of the risk is a quantitative issue
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egistry could be linked to the lice
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poisoned through mishandling of an
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no documented incident could be fou
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Region Table 3. Use of using pictog
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of fluorinated HDPE because fluorin
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Current initiatives, at the interna
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inding within their jurisdictions.
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Benefits of a hazard auditor From t
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different agencies take to be permi
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industry and of its serving to legi
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Anonymous. 1988a. FAO code of condu
Page 191 and 192: (Adam 1976; Fraser and Burrill 1979
Page 193 and 194: in the lower price categories tend
Page 195 and 196: Anas, N.; Jusoh Mamat, Md.; Heong,
Page 197 and 198: Zam, A.K. 1980. Bancian pengurusan
Page 199 and 200: Materials and methods Assessment of
Page 201 and 202: At the end of each day's operations
Page 203 and 204: Table 3. Number of garments chafing
Page 205 and 206: underlines the need to change attit
Page 207 and 208: Evaluation and management of pestic
Page 209 and 210: target and react with some crucial
Page 211 and 212: scientists on a contractual basis,
Page 213 and 214: pesticides in developing countries
Page 215 and 216: Regional information resources for
Page 217 and 218: Table 2. Number of enquiries receiv
Page 219 and 220: extent of this problem in developin
Page 221 and 222: Promoting the safe use of pesticide
Page 223 and 224: pests) is observed. Thus, the "quic
Page 225 and 226: is one of principal agencies respon
Page 227 and 228: Pretesting materials in training an
Page 229 and 230: of costly medical technologies and
Page 231 and 232: The agricultural sector has by far
Page 233 and 234: At the community level, the extent
Page 235 and 236: Neufeld, V. 1989. Community-based m
Page 237 and 238: Outline for learning module using o
Page 239 and 240: WHO (World Health Organization). 19
Page 241: In the past decade, tremendous effo
Page 245 and 246: As a result of deregulation, the nu
Page 247 and 248: Table 3. Proportion of workers usin
Page 249 and 250: Shih, J.H.; Wu, Z.Q.; Zhang, Y.X.;
Page 251 and 252: Pesticides and wildlife: a short gu
Page 253 and 254: Using target-specific products mini
Page 255 and 256: specifications. Other data are asse
Page 257 and 258: Formulation-specific concerns The e
Page 259 and 260: mirex, and chiordecone have been ba
Page 261 and 262: 1984). The base for pesticide granu
Page 263 and 264: e more stringently reviewed. In Can
Page 265 and 266: For example, searching for carcasse
Page 267 and 268: overlooked. In developing countries
Page 269 and 270: Establish monitoring programs to de
Page 271 and 272: Grue, C.E.; Fleming, W.J.; Busby, D
Page 273 and 274: Schafer, E.W., Jr; Brunton, R.B. 19
Page 275 and 276: Table 1. Prevalence and distributio
Page 277 and 278: By 1980, resistance to at least one
Page 279 and 280: tries. Japanese encephalitis has be
Page 281 and 282: to the targeted larvae of mosquitoe
Page 283 and 284: The role of the community Many prob
Page 285 and 286: from locally available sacking mate
Page 287 and 288: services. The project also includes
Page 289 and 290: Maxwell, C.A.; Curtis, C.F.; Haji,
Page 291 and 292: Botanical pesticides: optimizing pe
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neurotoxin but a cytotoxin, cross-r
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MoDuscicide- and pesticide-developm
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A development plan has been identif
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and environmental effects must be d
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Devitt, B.D.; Philogène, B.J.R.; H
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Endod, a potential natural pesticid
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University in Ottawa, and in other
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in the field (Lugt 1981), 137 500 m
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Hutchinson, J.; Dalziel, J.M. 1929.
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Community participation, including
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Acknowledgment- This research has b
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multiplication. The high temperatur
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Because pesticides are required in
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Africa and now East Africa. The suc
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Integrated pest management 1PM may
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to risk crop damage. Close involvem
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Ramos-Ocampo, V.E.; Magallona, E.D.
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esidues on fresh and cooked vegetab
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Grassy strip 108 N Farmer's Experim
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Table 1. Description and efficiency
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Table 3. Cost (PHP) of pest managem
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in the field. These pests were the
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PART V APPENDICES
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Ravindra Fernando, National Poisons
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Frank White, Pan-American Health Or
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ECHO abdominal ultrasonograph EEG e
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URT upper respiratory tract USAID U
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