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Preventing Childhood Obesity - Evidence Policy and Practice.pdf

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Chapter 33<br />

Finally, within a given individual, namely a malnourished<br />

young child, there can be a quick transition,<br />

over a few years or even months, from being<br />

wasted (underweight for height) to being overweight<br />

or even obese (excess weight for height). Moreover, in<br />

many cases, these children remain stunted (low height<br />

for age), making them more vulnerable in an urban<br />

setting to obesity <strong>and</strong> diabetes, both of which are<br />

linked to excess body fat resulting from sedentary lifestyles<br />

<strong>and</strong> consumption of high - energy density diets.<br />

In fact, the model of the malnourished child during<br />

recovery serves to illustrate many of the features of the<br />

nutrition transition mirroring the rapid shift in diet<br />

from low - to high - energy density food, as well as a<br />

progressively sedentary life style, that moves stunted<br />

populations from underweight to overweight <strong>and</strong><br />

obesity. 12<br />

More recently, the influence of postnatal growth on<br />

later development of obesity <strong>and</strong> nutrition - related<br />

chronic disease has been documented. Postnatal<br />

growth is clearly related to prenatal growth with some<br />

of the metabolic changes associated with prenatal<br />

nutritional sufficiency affecting postnatal physiology<br />

13 – 15<br />

<strong>and</strong> behavior which, in turn, affect growth. In<br />

2005, Stein et al reviewed the evidence linking child<br />

growth <strong>and</strong> chronic diseases in five cohorts from transitional<br />

countries (China, India, Guatemala, Brazil<br />

<strong>and</strong> the Philippines) concluding that growth failure in<br />

early childhood <strong>and</strong> increased weight gain in later<br />

childhood were associated with increased prevalence<br />

of risk factors for cardiovascular disease (i.e. body<br />

composition, blood pressure, glucose metabolism). 16<br />

These results correspond to studies from cohorts that<br />

originated in the 1970s <strong>and</strong> early 1980s. The clear<br />

progression of the obesogenic environment in most<br />

countries around the world suggests that the present<br />

impact of these early life factors in determining<br />

the rise in adult chronic diseases is likely to be<br />

underestimated.<br />

Postnatal growth, on the other h<strong>and</strong>, is also associated<br />

with later adult disease independent of prenatal<br />

growth. Studies from developed countries show a consistent<br />

positive association between infant size <strong>and</strong><br />

later body size but inconsistent associations with later<br />

disease. 17 In transitional countries, it is important to<br />

clarify the relative importance of growth in different<br />

postnatal periods in order to implement interventions.<br />

In Brazil, India <strong>and</strong> Guatemala, growth in<br />

infancy (0 – 1 years) <strong>and</strong> late childhood (over 2 years)<br />

were described as critical periods predicting adult<br />

body composition. Weight gain in infancy was related<br />

to higher fat - free mass in adulthood, while rapid<br />

weight gain in late childhood was associated with a<br />

higher acquisition of fat mass. In India, greater<br />

increases in body mass index ( BMI ) between 2 <strong>and</strong> 12<br />

years were strongly associated with glucose intolerance<br />

<strong>and</strong> Type 2 diabetes in adulthood. 15<br />

Access to f ood, p overty <strong>and</strong><br />

c hildhood o besity<br />

Income level is the main determinant of access to<br />

food. The proportion of family income that is spent<br />

on food serves to define the population income categories<br />

in most developing countries. If family income<br />

is less than the cost of one basic food basket the family<br />

is defined as indigent; those with income below the<br />

cost of two food baskets are classified as poor, in this<br />

case they actually spend more than 50% of their<br />

income on basic foods. 18<br />

Food security depends on the households ’ access to<br />

food rather than overall availability of food. In rural<br />

areas, household food security depends mainly on<br />

access to l<strong>and</strong> <strong>and</strong> other agricultural resources which<br />

facilitate domestic production. In urban areas,<br />

however, food is mainly purchased in the market. A<br />

variety of foods, therefore, needs to be available <strong>and</strong><br />

affordable in urban markets for adequate food security.<br />

In most developing regions there is plenty of food<br />

in the stores, yet families living under poverty conditions<br />

are unable to buy food of sufficient quantity <strong>and</strong>/<br />

or quality <strong>and</strong> thus will be food insecure. 19,20<br />

Poverty <strong>and</strong> food insecurity go h<strong>and</strong> in h<strong>and</strong>. Food<br />

security in poor households often fluctuates dramatically,<br />

depending on changes in agricultural production<br />

in response to seasonal <strong>and</strong> environmental<br />

conditions. Fluctuating market prices affect poor producers<br />

as well as the urban or rural poor. In an open<br />

market economy, farmers <strong>and</strong> their families are also<br />

affected by falling global market prices for food commodities.<br />

In most cases, they depend on products they<br />

place in the market <strong>and</strong> this means that their income<br />

will vary with commodity prices which are beyond<br />

their control. In the urban setting, the poor rely on<br />

food purchases that are commonly affected by<br />

rampant inflation. This income instability means that<br />

286

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