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[18] Lingyan Xu, Xinran Ma, Jieli Li, Xiaoying Li, Jianming Xu, Shu Wang, Guang Ning. SRC-3deficient mice developed fat redistribution under high-fat diet. Endocrine.2010; 38(1): 60-66[19] Menglei Chao, Weiqing Wang, Yifei Wang, Xi Lu, Jin Meng, Guang Ning. Bioequivalencebetween two human insulin analogs in Chinese population: Glulisine and LisproEndocrine.2010; 38: 48-52.[20] Liu RX, Wang WQ, Ye L, Bi YF, Fang H, Cui B, Zhou WW, Dai M, Zhang J, Li XY, NingG. p21-activated kinase 3 is overexpressed in thymic neuroendocrine tumors (carcinoids)with ectopic ACTH syndrome and participates in cell migration. Endocrine.2010; 38(1):38-47. 1.278[21] Ling-Yan Xu, Xin-Ran Ma, Jie-Li Li, Xiao-Ying Li, Jian-Ming Xu, Shu Wang and GuangNing. Ablation of steroid receptor coactivator-3 in mice impairs adipogenesis and enhancesenergy expenditure. Frontiers of Medicine in China.2010; 4(2): 229-234[22] Min Xu, Xiaoying Li, Jiguang Wang, Xiajuan Wang, Yun Huang, Qi Cheng, He Huang, R Li,Jie Xiang, Jiaorong, Tan, Meng Dai, Guang Ning. Retinol-binding protein 4 is associatedwith impaired glucose regulation and microalbuminuria in a Chinese population Diabetologia.2009; 52: 1511-1519.[23] Cao Y, Liu R, Jiang X, Lu J, Jiang J, Zhang C, Li X, Ning G. Nuclear-cytoplasmic shuttlingof menin regulates nuclear translocation of β-catenin. Mol Cell Biol. 2009; 29: 5477-5487[24] Bi YF, Liu RX, Ye L, Fang H, Li X, Wang W, Zhang J, Wang KK, Jiang L, Su T,Chen ZY,Ning G. Gene expression profiles of thymic neuroendocrine tumors(carcinoids) with ectopicACTH syndrome reveal novel molecular mechanism. Endocr Relat Cancer. 2009; 16(4):1273-1282.[25] Ma X, Xu L, Wang S, Chen H, Xu J, Li X, Ning G. Loss of steroid receptor co-activator-3attenuates carbon tetrachloride-induced murine hepatic injury and fibrosis. Lab Invest. 2009;89: 903-914[26] Jie Hong, Yuwen Zhang, Huijie Zhang, Huiyinig Jia, Yu Zhang, Xiaoyi Ding, Danyang Zhou,Huiping Chen, Xiaohua Jiang, Bin Cui, Xiaoying Li, Guang Ning. The novel compoundheterozygous mutations, V434del and W666X, in WFS1 gene causing the Wolframsyndrome in a chinese family Endocrine. 2009; 35: 151-157[27]Yuwen Zhang, Huiying Jia, Jie Hong, Yan Ge, Huijie Zhang, Chunfang Shen, Lei Ye, Bin Cui,Xiaoying Li, Weiqiong Gu, Yifei Zhang, Weiqing Wang, Guang Ning. Clinical and geneticanalysis for four Chinese families with Prader-Willi syndrome Endocrine. 2009; 36: 37-44.[28] Zhang Y, Hong J, Gu W, Gui M, Chen Y, Zhang Y, Chi Z, Wang W, Li X, Ning G. Impact ofthe metabolic syndrome and its individual components on risk andseverity of coronary heartdisease. Endocrine. 2009; 36: 233-238.[29] Yan Q, Gu WQ, Hong J, Zhang YF, Su YX, Gui MH, Zhang Y, Chi ZN, Zhang YW, Li XY,Ning G. Coronary angiographic studies of impaired glucose regulation and coronary arterydisease in Chinese nondiabetic subjects. Endocrine. 2009; 36: 457-463.[30] Chao M, Zou D, Zhang Y, Chen Y, Wang M, Wu H, Ning G, Wang W Improving insulinresistance with traditional Chinese medicine in type 2 diabetic patients. Endocrine. 2009; 36:268-274.[31] Xiang J, Li XY, Xu M, Hong J, Huang Y, Tan JR, Lu X, Dai M, Yu B, Ning G. Zinctransporter-8 gene (SLC30A8) is associated with type 2 diabetes in Chinese. J ClinEndocrinol Metab. 2008; 93(10): 4107-4112- 9 -