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泌尿、生殖、内分泌系统 - 国家自然科学基金委员会医学科学部

泌尿、生殖、内分泌系统 - 国家自然科学基金委员会医学科学部

泌尿、生殖、内分泌系统 - 国家自然科学基金委员会医学科学部

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Chinese patients with anti-GBM disease.Clin Immunol. 2009 ;133(2):245-50.[8]Cui Z, Zhao MH, Segelmark M, Hellmark T. Natural autoantibodies to myeloperoxidase,proteinase 3, and the glomerular basement membrane are present in normal individuals.Kidney Int. 2010;78(6):590-7.[9]Chen M, Cui Z, Zhao MH. ANCA-associated vasculitis and anti-GBM disease: theexperience in China. Nephrol Dial Transplant.2010 ;25(7):2062-5.[10]Xing GQ, Chen M, Liu G, Zheng X, E J, Zhao MH. Differential deposition of C4d andMBL in glomeruli of patients with ANCA-negative pauci-immune crescenticglomerulonephritis.J Clin Immunol.2010;30(1):144-56.[11]Xu PC, Chen M, Cui Z, Zhao MH. Influence of myeloperoxidase by anti-myeloperoxidaseantibodies and its association with the disease activity in microscopic polyangiitis.Rheumatology (Oxford). 2010;49(11):2068-75.[12]Xing GQ, Chen M, Liu G, Wang SX, Zhao MH. Renal neutrophils infiltration inantineutrophil cytoplasmic antibodies-negative pauci-immune crescentic glomerulonephritis.Am J Med Sci.2010 ;340(6):474-80.[13]Lin W, Chen M, Cui Z, Zhao MH. The immunopathological spectrum of crescenticglomerulonephritis: a survey of 106 patients in a single Chinese center. Nephron ClinPract.2010;116(1):c65-74.[14]Luo H, Chen M, Cui Z, Yang R, Xu PC, Zhou XJ, Zhao MH. The association ofHLA-DQB1, -DQA1 and -DPB1 alleles with anti- glomerular basement membrane (GBM)disease in Chinese patients. BMC Nephrol.2011; 13:12-21.[15]Luo H, Chen M, Yang R, Xu PC, Zhao MH. The association of HLA-DRB1 alleles withantineutrophil cytoplasmic antibody-associated systemic vasculitis in Chinese patients. HumImmunol.2011;72(5):422-5.[ 16 ] Cui Z, Zhao J, Jia XY, Zhu SN, Zhao MH. Clinical features and outcomes ofanti-glomerular basement membrane disease in older patients. Am J Kidney Dis.2011;57(4):575-82.[17]Cui Z, Zhao J, Jia XY, Zhu SN, Jin QZ, Cheng XY, Zhao MH. Anti-glomerular basementmembrane disease: outcomes of different therapeutic regimens in a large single-centerChinese cohort study.Medicine (Baltimore). 2011;90(5):303-11.[18]Tan Y, Yu F, Qu Z, Su T, Xing GQ, Wu LH, Wang FM, Liu G, Yang L, Zhao MH.Modified C-reactive protein might be a target autoantigen of TINU syndrome. Clin J Am SocNephrol. 2011;6(1):93-100.[19]Xu PC, Cui Z, Chen M, Hellmark T, Zhao MH. Comparison of characteristics of naturalautoantibodies against myeloperoxidase and anti-myeloperoxidase autoantibodies frompatients with microscopic polyangiitis. Rheumatology (Oxford). 2011;50(7):1236-43.[20]Jia XY, Qu Z, Cui Z, Yang R, Zhao J, Zhao MH. Circulating anti-glomerular basementmembrane autoantibodies against α3(IV)NC1 undetectable by commercially availableenzyme-linked immunosorbent assays. Nephrology (Carlton). 2012;17(2):160-6.[21]Xu PC, Chen M, Zhao MH. High potential to reverse the inhibition of myeloperoxidase byceruloplasmin of anti-myeloperoxidase autoantibodies of IgG3 subclass.Autoimmunity.2012;45(3):218-25.会 议 报 告 : 国 际 大 会 2 分 组 0 国 内 大 会 0 分 组 0- 2 -

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