Candida albicans-induzierte Genexpression in primären ... - OPUS

13.2 Publizierte Abstracts
Müller, V., Viemann, D., Schmidt, M., Tenbrock, K., Schmid, S., Klimmek, K., Ludwig,
S., Roth, J. and Goebeler, M. (2006) Nickel triggers a unique inflammatory and
proangiogeneticgene expression pattern via activation of NF-κB and HIF-1α. J Invest
Dermatol, 126, Supplement 3, s93
Viemann, D., Schmid, S., Tenbrock, K., Klimmek, K., Müller, V., Schmidt, M., Roth, J.
and Goebeler, M. (2006) Dissection of nickel-induced intracellular signal transduction
pathways and identification of novel target genes in primary human endothelial cells.
Exp Dermatol, 15, 257
Müller, V., Viemann, D., Schmidt, M., Leverkus, M., Roth, J. and Goebeler, M. (2005)
Gene expression profiling of human primary endothelial cells after infection with
Candida albicans. J Invest Dermatol, 125, Supplement 1s, A83
Müller, V., Ossadnik, M., Toksoy, A., Viemann, D., Roth, J., Ludwig, S., Bröcker,
E.B., Goebeler, M. and Leverkus, M. (2005) Differential roles of IKK1 and IKK2 for C.
albicans-induced NF-κB activation. Arch Dermatol Res, 296, 431 (A)
Müller, V., Schmid, S., Viemann, D., Roth, J., Ludwig, S., Bröcker, E.B., Leverkus, M.
and Goebeler, M. (2004) C. albicans activates human primary endothelial cells by
IKK2/IκBα/NF-κB- and p38-MAP Kinase-dependent intracellular signalling pathways.
J Invest Dermatol, 123, A71
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