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Table of Contents - WOC 2012

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IS-CAT-SA 186 (5)<br />

Management <strong>of</strong> Multifocal IOL Complications<br />

Akaishi Leonardo (1)<br />

1. Instituto de Catarata de Brasilia<br />

Abstract not available<br />

Classic Signs in Neuro-Ophthalmology<br />

Sat 18 Feb 10:30 - 12:00 Conference Room A1<br />

IS-NEO-SA 187 (1)<br />

Optic Disc Edema<br />

Arnold Anthony (1)<br />

1. Jules Stein Eye Institute<br />

Presentation reviews features which distinguish true optic disc edema from<br />

pseudo-edema and outlines differential diagnosis <strong>of</strong> both unilateral and<br />

bilateral optic disc edema, with management plans for the most common<br />

entities. Primary diagnoses discussed include papilledema, papillitis, anterior<br />

ischemic optic neuropathy, Leber hereditary optic neuropathy, and retinal<br />

vascular causes.<br />

IS-NEO-SA 187 (2)<br />

Dilated Pupil<br />

Aydin Pinar (1)<br />

1. ICO<br />

Unilateral or bilateral dilated pupils are always a disturbance for both patients<br />

and doctors. A practical guideline will be given for the differential diagnosis and<br />

management <strong>of</strong> dilated pupils.<br />

IS-NEO-SA 187 (3)<br />

Abduction Deficit, Differential Diagnosis and Work Up<br />

Alobailan Majed (1)<br />

1. King Saud University<br />

Objectives: At the conclusion <strong>of</strong> this course, attendees will be able to provide<br />

management solutions to patients with abduction deficit. The presentation will<br />

cover the different clinical presentations <strong>of</strong> abduction deficit with the differential<br />

diagnosis and the need for testing. The attendee will know when and what<br />

studies, treatments, and referrals to initiate for distinct diagnoses.<br />

Summary <strong>of</strong> Content: A practical approach to the patient with abduction deficits<br />

will be presented. The discrimination <strong>of</strong> differentials diagnoses symptoms and<br />

signs will be emphasized, utilizing video examples <strong>of</strong> methods <strong>of</strong> assessing<br />

ocular alignment, cranial nerve palsy, internuclear ophthalmoplegia, fatigable<br />

ptosis and ophthalmoplegia, and ice testing.<br />

Target Audience: General ophthalmologists and residents<br />

Educational Level: Beginning to intermediate<br />

IS-NEO-SA 187 (4)<br />

Optic Atrophy<br />

Chung Sophia (1)<br />

1. Saint Louis University School <strong>of</strong> Medicine<br />

Optic atrophy is the morphologic endpoint <strong>of</strong> diseases that cause axonal loss<br />

in the anterior visual system, i.e, the retinogeniculate pathway. The etiologies<br />

vary considerably but a detailed history and examination can <strong>of</strong>ten guide the<br />

ophthalmologist to the appropriate diagnosis. However, the diagnosis can<br />

be elusive and laboratory evaluation is required. The presentation will focus<br />

on the sometimes unique features <strong>of</strong> cause-specific optic atrophy and the<br />

recommended clinical approach and evaluation <strong>of</strong> optic atrophy.<br />

IS-NEO-SA 187 (5)<br />

Internuclear Ophthalmoplegia<br />

Kashii Satoshi (1)<br />

1. Aichi Shukutoku University<br />

<strong>WOC</strong><strong>2012</strong> Abstract Book<br />

An ipsilateral adduction deficit with abduction nystagmus <strong>of</strong> the contralateral<br />

eye is a classic sign <strong>of</strong> internuclear ophthalmoplegia (INO) that is caused<br />

by lesions affecting the medial longitudinal fasciculus (MLF). Preserved<br />

convergence suggests a caudal lesion with presumed preservation <strong>of</strong> the<br />

medial rectus motorneurons. The diagnosis is fairly straightforward, but that<br />

is not all, folks. INO causes various disturbances <strong>of</strong> extraocular movements<br />

as the MLF contains the pathways mediating the vertical vestibuloocular reflex<br />

and those connecting between vestibular nuclei and the interstitial nucleus <strong>of</strong><br />

Cajal.<br />

IS-NEO-SA 187 (6)<br />

Ptosis<br />

McCulley Timothy (1)<br />

1. Johns Hopkins School <strong>of</strong> Medicine<br />

Ptosis <strong>of</strong> the eyelids is <strong>of</strong>ten assumed to be benign. This is true in the majority<br />

<strong>of</strong> patients, with the most common cause being involutional changes <strong>of</strong> the<br />

levator aponeurosis and muscle. Before jumping to this conclusion, other<br />

etiologies should be considered. Blephaorptosis may indicate more a serious<br />

underlying disease; such as myasthenia gravis, Horner syndrome, oculomotor<br />

nerve abnormalities and increased orbicularis oculi tone. This talk will focus<br />

on diagnostic clues to aid in identifying underlying causes <strong>of</strong> blepharoptosis.<br />

Uveal Melanoma<br />

Sat 18 Feb 10:30 - 12:00 Conference Room A2<br />

IS-ONC-SA 188 (1)<br />

Clinical Features<br />

Shields Jerry (1)<br />

1. Wills Eye Institute<br />

Uveal melanoma can occur in the iris, ciliary body or choroid. Ophthalmologists<br />

must recognize their clinical variations to provide the best patient care. Iris<br />

melanoma can be nodular or diffuse. Ciliary body melanoma occurs in an<br />

occult location and may attain a large size before clinical recognition, but<br />

certain external signs may assist in recognition. Choroidal melanoma can be<br />

nodular or diffuse and has several clinical variations. Melanoma in any location<br />

can be nonpigmented, making the diagnosis more difficult.<br />

IS-ONC-SA 188 (2)<br />

Brachytherapy <strong>of</strong> Uveal Melanomas<br />

Bornfeld Norbert (1)<br />

1. University <strong>of</strong> Essen<br />

Local radiation with episcleral radioactive plaques as introduced by Stallard is<br />

now the most frequently used treatment modality in the armamentarium for<br />

eye salvaging treatment <strong>of</strong> uveal melanomas. Based on a series <strong>of</strong> 1600<br />

cases evaluated current techniques in the brachytherapy <strong>of</strong> uvealmelanomas<br />

with gamma-ray (iodine-125) and beta-ray sources (ruthenium-106) will be<br />

presented. Special emphasis will be laid on the design <strong>of</strong> plaques using both<br />

radiation sources, limits and complications <strong>of</strong> brachytherapy with special<br />

respect to large tumors and tumors at the posterior pole, ancillary treatment<br />

modalities like intravitreal treatment (corticosteroids, anti-VEGF drugs) and<br />

transpupillary thermotherapy and the management <strong>of</strong> treatment related<br />

complications like exudative retinal and choroidal detachment, radiation<br />

retinopathy and scleral necrosis.<br />

193

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