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Thursday, June 25th, 2009<br />

NON-LINEAR INCREASE IN RPE DURING CONSTANT LOAD EXERCISE TO VOLITIONAL EXHAUSTION<br />

RENFREE, A., GURNEY, T.<br />

UNIVERSITY OF WORCESTER<br />

It has been proposed that during constant load exercise to exhaustion, the increase in RPE throughout is linear and that termination<br />

usually occurs prior to attainment <strong>of</strong> the highest possible RPE score (Noakes 2004). This suggests that the rate <strong>of</strong> increase in RPE during<br />

constant load exercise, in addition to the highest RPE that can be sustained, determines the duration that a constant workload can be<br />

maintained for. This study models the increase in RPE during constant load exercise to exhaustion at supra lactate threshold intensity<br />

using both the Borg Category (C20) and Category-Ratio (CR10) scales.<br />

Ten recreationally active male participants performed an incremental exercise test on an electronically braked cycle ergometer to allow<br />

visual identification <strong>of</strong> the workload that coincided with the lactate threshold (LT). Participants then performed two trials to volitional exhaustion<br />

at a workload 60W above LT. RPE was recorded at 60s intervals using either the C20 or CR10 scale. Participants were familiarised<br />

with the scales in advance, and the order was randomised. For both scales, RPE data was subsequently scaled as a percentage <strong>of</strong><br />

total exercise duration for each participant.<br />

Total exercise duration was similar regardless <strong>of</strong> whether participants reported RPE using the C20 (16.7+5.3min) or CR10 (17.1+8.1min)<br />

scales (P>0.05). During every individual trial, participants reported achieving maximum possible RPE regardless <strong>of</strong> the scale used. When<br />

reporting RPE using C20, termination coincided with achievement <strong>of</strong> maximal RPE in 30% <strong>of</strong> participants. When reporting RPE using CR10,<br />

on no occasion did termination coincide with achievement <strong>of</strong> a maximal RPE. As a group, participants spent 17+14.2 % <strong>of</strong> total exercise<br />

duration at maximal RPE when using the C20 scale, and 21 +12.0% when using the CR10 scale.<br />

These results suggest that in recreationally active participants, the rate <strong>of</strong> increase in RPE during constant load exercise is not linear and<br />

that termination does not always coincide with attainment <strong>of</strong> the highest possible values. Therefore, regardless <strong>of</strong> the RPE scale used, the<br />

findings <strong>of</strong> this study disagree with the contention <strong>of</strong> Noakes (2004) that RPE is the real determinant <strong>of</strong> the fatigue point during prolonged<br />

sub maximal exercise at a fixed work rate. It is possible, however, that the observed relationship between RPE and termination <strong>of</strong> exercise<br />

may depend to some extent on the training status and experience <strong>of</strong> participants in these studies.<br />

Reference<br />

Noakes, T. D. (2004) Linear relationship between the perception <strong>of</strong> effort and the duration <strong>of</strong> constant load exercise that remains, J Appl.<br />

Physiol. (96) 1571-1573.<br />

TASK FAILURE DURING HIGH-INTENSITY EXERCISE IS ASSOCIATED WITH A CRITICAL REDUCTION IN TISSUE OXY-<br />

GENATION<br />

BISHOP, D., BORTOLOTTI, S., FERRI, A.<br />

UNIVERSITÀ DEGLI STUDI DI VERONA<br />

Introduction: Task failure is an important feature <strong>of</strong> performance, ageing and many diseases. Three popular models suggest that task<br />

failure may be due to: a) a failure <strong>of</strong> skeletal muscle motor unit recruitment (1); b) depletion <strong>of</strong> a fixed, critical, muscle energy store (2); or c)<br />

a critical reduction in tissue oxygenation (3). The purpose <strong>of</strong> this study was to assess the hypothesis that task failure during high-intensity,<br />

whole-body exercise may be associated with a critical reduction in tissue oxygenation.<br />

Methods: Six well-trained cyclists (VO2max: 67.6 +/- 6.3 mL/kg/min) performed an incremental test to exhaustion on a cycle ergometer.<br />

They then performed a familiarisation session, followed by, in a random order, constant-load exercise tests at 100, 110 and 120% <strong>of</strong><br />

VO2max until task failure. During these trials, near-infrared spectroscopy (NIRS) was used to monitor changes in the concentration<br />

(&#956;M) <strong>of</strong> oxygenated haemoglobin (Hb) and myoglobin (Mb) (delta[02Hb]), deoxygenated Hb+Mb (delta [HHb]) in the right vastus<br />

lateralis muscle and the frontal cerebral cortex. Oxygen uptake (VO2) and rating <strong>of</strong> perceived exertion (RPE) were also monitored during<br />

all 3 trials. The study had ethics approval and all subjects signed a statement <strong>of</strong> informed consent prior to the study.<br />

Results: At task failure, there was no significant difference between the three conditions for delta[HHb] (40 +/- 5 vs 39 +/- 8 vs 37 +/- 7<br />

&#956;M; P>0.05), delta[02Hb] (23 +/- 5 vs 26 +/- 6 vs 22 +/- 3 &#956;M; P>0.05), VO2 (67.6 +/- 4.5 vs 67.4 +/- 3.8 vs 64.7 +/- 4.1<br />

mL/kg/min) or RPE (20 vs 20 vs 20; P>0.05). There were also no significant differences between trials for cerebral oxygenation levels.<br />

Discussion: Our delta[02Hb] and delta[HHb] values, for both the right vastus lateralis muscle and the frontal cerebral cortex, are similar to<br />

those previously reported in the literature (3). We report for the first time however, that task failure during 3 different exercise intensities<br />

occurred at similar tissue oxygen (and oxygen consumption) levels. While further research is required, our results raise the possibility that<br />

task failure during high-intensity exercise (at sea level) may be associated with an inability to further increase the rate <strong>of</strong> aerobic energy<br />

supply.<br />

References<br />

1. Davis, J. M. and S. P. Bailey. Med Sci Sports Exerc. 29:45-57, 1997.<br />

2. Monod, H. and J. Scherrer. Ergonomics. 8:329-338, 1965.<br />

3. Subudhi, A. W., A. C. Dimmen, and R. C. Roach. J Appl Physiol. 103:177-183, 2007.<br />

CARBOHYDRATE SUPPLEMENTATION PREVENT PROTEIN METABOLISM IN A LONG DISTANCE MOUNTAIN BIKE<br />

TREADMILL<br />

MOTA, M.P., SOARES, J., TEIXEIRA, R., JOÃO, P.<br />

UNIVERSIDADE DE TRÁS-OS-MONTES E ALTO DOURO<br />

Introduction: As a consequence <strong>of</strong> its intensity and duration, Mountain Bike (MTB) can induce an increase in serum urea concentration as<br />

the final product <strong>of</strong> protein metabolism. This may be explained by the significant reduction in muscle glycogen with the duration <strong>of</strong> the<br />

event. In order to prevent muscle glycogen depletion and afterward protein metabolism, we searched if carbohydrate ingestion during a<br />

long distance MTB treadmill can prevent protein metabolism.<br />

Methods: Ten male athletes aged between 15 and 46 yrs took part in this study (average=32±8.7 yrs). All the subjects performed a 59.14<br />

km MTB treadmill in 5.71 hours (±0.76) during which a carbohydrate supplementation (glucose concentration <strong>of</strong> 0,7g/Kg/h) was given to<br />

each athlete and heart rate was monitored through Polar S625X.<br />

Results/Discussion: Bearing in mind heart rate data, our results revealed that the MTB treadmill was performed with an average intensity<br />

<strong>of</strong> 82.4%. Although relative values <strong>of</strong> serum urea concentration were significantly increased (15.55%; p=0,012) at the end <strong>of</strong> the MTB<br />

OSLO/NORWAY, JUNE 24-27, 2009 249

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