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Friday, June 26th, 2009<br />

12:00 - 13:15<br />

Plenary sessions<br />

PS-PL03 Sport, Body and Mind: Towards a New Understanding?<br />

EVIDENCE FROM RANDOMIZED TRIALS: EFFECTS ON MENTAL DISEASE OUTCOMES<br />

DUNN, A.L.<br />

KLEIN BUENDEL<br />

Recent randomized clinical trials demonstrate the role <strong>of</strong> exercise and physical activity in the treament <strong>of</strong> a wide variety <strong>of</strong> mental pathologies.<br />

Using depression illness as a primary focus and severe mental illness as a secondary focus, this discussion will highlight how<br />

exercise could be used in the treatment <strong>of</strong> depression and severe mental disorders.<br />

A number <strong>of</strong> randomized trials suggest that exercise could serve multiple functions in the treatment <strong>of</strong> depression disorders and severe<br />

mental illness. One randomized trial examining different doses <strong>of</strong> exercise to treat diagnosed Major Depressive Disorder (MDD) as a<br />

monotherapy found that an amount equivalent to public health guidelines for physical activity was effective in reducing symptoms, while<br />

a lower dose was not. Furthermore, several studies now show response and remission rates <strong>of</strong> exercise to treat depression disorders<br />

appear to be equivalent to standard therapies <strong>of</strong> antidepressant medication and cognitive behavioural therapy (CBT). Exercise could also<br />

be used to augment pharmacotherapy or CBT in patients who might not be achieving complete remission <strong>of</strong> symptoms, and there is<br />

some evidence to suggest the use <strong>of</strong> exercise to augment traditional treatments is similar to findings from multi-site trials. Finally, recent<br />

trials demonstrate that exercise could be used as adjunctive therapy with both depression and severe mental disorders to improve<br />

health outcomes related to co-morbid diseases, like cardiovascular disease and diabetes.<br />

Most studies <strong>of</strong> exercise have had relatively small sample sizes in comparison to standard treatment studies <strong>of</strong> pharmacotherapy and<br />

CBT. There is a need to demonstrate the efficacy and effectiveness <strong>of</strong> exercise treatments in well controlled studies with larger sample<br />

sizes <strong>of</strong> diagnosed patients. Further, there is a need for better understanding <strong>of</strong> the potential mechanisms underlying treatment effects in<br />

both primary and secondary prevention studies. This includes understanding: 1) short-term treatment effects <strong>of</strong> acute bouts <strong>of</strong> exercise; 2)<br />

long term treatment effects to prevent relapse; 3) key factors that might influence response to treatment, e.g., why do some individuals<br />

respond to exercise treatment; 4) the role <strong>of</strong> exercise in preventing the onset <strong>of</strong> a wide variety <strong>of</strong> mental disorders; and, 5) development <strong>of</strong><br />

programs that will enhance motivational factors for individuals with mental disorders to engage in regular exercise.<br />

THE BRAIN : HOW DOES THE INTEGRATION OF SIGNALS OCCUR ?<br />

MEEUSEN, R.<br />

VRIJE UNIVERSITEIT BRUSSEL<br />

Brain research is a rapidly developing field and new understandings emerge on the interaction between the brain, motor action, and<br />

even motivation and emotion.<br />

Recently there has been more interest in the effects <strong>of</strong> exercise on brain processes. These new experiments arise mostly from animal<br />

studies where the basic knowledge on possible underlying physiological mechanisms are explored. Much progress has been made in<br />

identifying the various hormonal and neural mechanisms by which the brain informs itself about signals from the periphery, and, in turn,<br />

generates behavioural, autonomic, and endocrine output. Several brain nuclei play crucial roles in homeostatic functions such as food<br />

intake, and in processing prior experience with food, reward, exercise etc. From these animal studies it is known that exercise might be a<br />

‘natural’ antidepressant, and that some neurotransmitters are influenced by regular exercise. Training will induce other neurochemical<br />

reactions, that might explain possible underlying neurophysiological arguments for the use <strong>of</strong> training in degenerative neurological<br />

diseases such as Parkinson’s disease and Alzheimer disease.<br />

Exercise can be a powerful tool in the treatment <strong>of</strong> neurological diseases. There have been a number <strong>of</strong> studies that examined the exercise<br />

effect on a wide variety <strong>of</strong> pathological situations, and it seems that exercise works, although there is a general need for controlled<br />

trials.<br />

14:15 - 15:15<br />

Poster presentations<br />

PP-PH07 Physiology 7<br />

EFFECT OF REST DURATION BETWEEN RESISTANCE TRAINING SETS ON MARKERS OF MUSCLE DAMAGE IN TRAINED<br />

MALES<br />

GHADERI, M., RAHIMI, R., AGHA ALINEJAD, H.<br />

AZAD UNIVERSITY OF MAHABAD<br />

Abstract:<br />

The purpose <strong>of</strong> the present study was to evaluate the effects <strong>of</strong> rest duration between resistance training sets on markers <strong>of</strong> muscle<br />

damage in trained men. Therefore, ten male athletes (Mean ± SD, age=20.37± 2.24 years, body mass= 65.5 ± 26.70 kg) voluntarily<br />

participated in four sessions with 48 h recovery time who were performing different resistance trainings. At the first session, one repetition<br />

maximum (1RM) <strong>of</strong> participants evaluated. The subjects from the second session up to the fourth, each session, performed four sets <strong>of</strong><br />

squat and bench press to failure with 85 % <strong>of</strong> 1RM and one <strong>of</strong> rest intervals <strong>of</strong> 60s, 90s and 120s between the sets used randomly. Also,<br />

there were taken three blood samples from the subjects before, immediately after, and 30 min after the training, to determine serum<br />

OSLO/NORWAY, JUNE 24-27, 2009 347

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