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Thursday, June 25th, 2009<br />

mean head temperature was 1,7 ºC lower in CAP for boys and girls together. Mean heart frequency (HF) during rest periods was lower<br />

than during the exercise bouts, but was not statistically different between CAP and NOCAP. RPE and perception <strong>of</strong> thermal comfort (PTC)<br />

were not altered due to cap use, but increased during the exercise bouts in CAP and NOCAP. A positive correlation was observed between<br />

Vrun and VO2peak (r=0,64; p=0,002; r2=0,4). Conclusions: Cap use led to a decrease in mean head temperature. However, it<br />

was not sufficient to alter Vrun. Vrun decreases during all exercise bouts may have occurred due the action <strong>of</strong> a neural mechanism for<br />

homeostasis protection.<br />

LAMBERT, E.V., GIBSON, A St CLAIR and Noakes, T.D. Complex systems model <strong>of</strong><br />

fatigue: integrative homoeostatic control <strong>of</strong> peripheral physiological systems<br />

during exercise in humans. Br J Sports Med 2005; 39: 52-62.<br />

MUNDEL, T.; BUNN S, J.; HOOPER, P.L.; JONES, D.A. The effects <strong>of</strong> face cooling<br />

during hyperthermic exercise in man: evidence for a integrated thermal,<br />

neuroendocrine and behavioral response. Exp Physiol 2006; 92: 187-195.<br />

15:15 - 16:45<br />

Oral presentations<br />

OP-NU02 Nutrition 2<br />

EFFECTS OF EARLY-EVENING EXERCISE ON ACYLATED GHRELIN, LEPTIN AND PERCEIVED HUNGER MEASURED DURING<br />

A SUBSEQUENT SIMULATED NIGHT-SHIFT<br />

MORRIS, C., FULLICK, F., GREGSON, W., CLARKE, N., DORAN, D., MACLAREN, D., ATKINSON, G.<br />

LIVERPOOL JOHN MOORES UNIVERSITY<br />

Introduction: Shift-work, and particularly night-work, is common in Europe and is an independent risk factor for obesity (Atkinson et al.,<br />

2008). Shift-workers tend to ‘graze’ on high-fat foods during a night-shift and postprandial thermogenesis is lower at night, which, in<br />

combination, may compromise energy balance (Romon et al., 1993). Thus, a reduction in energy intake, by suppressing hunger could<br />

benefit the night-worker. Exercise in the late morning suppresses perceived hunger and associated regulators such as ghrelin (Broom et<br />

al., 2007) and leptin, for many hours. Although it is known that these hormones show circadian variation when measured at rest, no<br />

examination <strong>of</strong> the responses <strong>of</strong> these variables to exercise over a subsequent period <strong>of</strong> night-work has been attempted. We hypothesise<br />

that the responses <strong>of</strong> perceived hunger, ghrelin and leptin are different when awake and eating at night compared with previous<br />

studies undertaken during the day.<br />

Methods: Six healthy males (age: 30±8 y, body mass: 74.1±6 kg, height: 178±8 cm) completed two trials (exercise and control) in a random<br />

order. In the exercise trial, participants fasted from 10:00 h, consumed a test meal at 18:00 h and then cycled at 50% peak oxygen<br />

uptake between 19:00-20:00 h. Participants then remained awake until 05:00 h, consuming two identical meals at 22:00 and 02:00 h.<br />

Participants rested between 19:00 and 20:00 h in the control trial. During each trial, venous blood samples were drawn at 1-h intervals<br />

between 20:00-05:00 h for the determination <strong>of</strong> acylated ghrelin, leptin, insulin and glucose concentrations. Perceived hunger was also<br />

recorded at these times. Data were subtracted from baseline and analysed with two factor (trial x time) linear mixed models. Data are<br />

described as mean±SD.<br />

Results: Perceived hunger increased by 0.3±2.0 units throughout the night-work after exercise compared with a reduction <strong>of</strong> 1.2±2.3 units<br />

in the control trial (P0.05).<br />

Conclusions: We report, for the first time, that evening exercise increases perceived hunger and acylated ghrelin concentration during a<br />

subsequent night-shift. These data are in contrast to those from studies on diurnally-active individuals in which an exercise-mediated<br />

suppression <strong>of</strong> acylated ghrelin was observed (Broom et al., 2007). Therefore, the effects <strong>of</strong> exercise on appetite and its regulators appear<br />

different in the nocturnal period, which may help to explain the higher long-term risk <strong>of</strong> obesity in shift-workers.<br />

References<br />

Atkinson et al., (2008). Sports Med 38:671-85<br />

Broom et al., (2007). J Appl Physiol 102: 2165-2171<br />

Romon et al., (1993). Am J Clin Nutr 57: 467-480<br />

SHORT-TERM MEMORY IN POSTMENOPAUSE. WEIGHT OF DIFFERENT PREDICTORS VARIABLES<br />

DI BLASIO, A., DI DONATO, F., DI RENZO, D., GALLINA, S., NAPOLITANO, G., VALENTINI, P., RIPARI, P.<br />

G. D'ANNUNZIO UNIVERSITY, CHIETI<br />

Introduction: Literature widely shows the memory decline age-related, even if, on the other side, it documented the protective role <strong>of</strong><br />

physical exercise, diet, and both hormonal and anthropometric pattern on memory. Aerobic fitness protects and enhances memory<br />

through hippocampal neurogenesis related to the increase <strong>of</strong> cerebral blood flow exercise-dependent. The dietary habitudes play a<br />

fundamental on the memory functions through its quantitative and qualitative characteristics that can lead to diabetes, hypertension,<br />

stroke and excessive oxidative stress altering brain structure and functions. Both estrogens and leptin patterns have positive functional<br />

implications in memory processes, as excessive stress hormones have a negative role. So that, menopause-related hormonal modifications<br />

becomes important in memory decline. Because the aromatasic activity <strong>of</strong> adipose tissue may partially replace estrogens deficit<br />

after menopause, in this period, body fat (FM%) has been shown positively related to memory. The aim <strong>of</strong> this study was to investigate the<br />

influence <strong>of</strong> dietary, anthropometric, and physiological parameters on short-term memory (STM) in postmenopause.<br />

Methods<br />

OSLO/NORWAY, JUNE 24-27, 2009 257

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