european college of sport science

PP-PH14 Physiology 14
THE ACUTE EFFECT OF TAURINE INGESTION ON 3KM RUNNING PERFORMANCE AND FORCE IN TRAINED MIDDLE
DISTANCE RUNNERS
BALSHAW, T.G., BAMPOURAS, T.M., BARRY, T., SPARKS, S.A.
UNIVERSITY OF CUMBRIA, LANCASTER
Introduction: Taurine (TA) can increase force production of isolated mammalian skeletal muscle tissue while depletion of it decreases
muscular function (Bakker and Berg, 2002; Hamilton et al., 2006). Although prolonged TA administration appears to benefit endurance
performance in trained endurance athletes (Lee et al, 2003), little research exists examining the acute effects of TA on performance
(Rutherford et al, 2006).The current study investigated the acute effect of 1000mg of TA on maximal 3km time trial (3KTT) performance and
lower limb muscular force production (MFP) in well-trained, competitive middle distance runners (MDR).
Methods: Six male MDR (age: mean 20.10, s = 1.11 years; 800m personal best times: mean 123.03, s = 5.18 seconds) participated in a
double blind, randomised, crossover designed study. Following a standardized warm-up, participants completed a laboratory-based
self-paced, maximal 3KTT on a treadmill ingesting either TA or placebo (PL), two hours prior to testing. Capillary blood lactate (LAC), glucose
(GLU) and MFP during three countermovement jumps (without arm swing) were measured pre- and post-3KTT. Respiratory exchange
ratio (RER), heart rate (HR), rating of perceived exertion (RPE), and split times were recorded at 500m intervals. Wilcoxon’s test was
used to analyse overall 3KTT performance. All other variables were analysed using Friedman’s test, followed by Wilcoxon’s test with
Bonferroni adjustment, if differences were found.
Results: No significant differences (P > 0.05) were found for any of the variables between conditions. However, the TA condition demonstrated
better performances in 3KTT but not MFP (TA: mean 650.00, s = 56.68 seconds; PL: mean 655.50, s = 68.22 seconds and TA:
mean 1196.37, s = 133.08 N; PL: mean 1289.53, s = 243.08 N, respectively).
Discussion: The failure to detect significant differences in performance between conditions indicates acute ingestion of TA may have no
effect on 3KTT performance or peak lower limb MFP of well trained endurance runners in middle distance events. The findings of this
study provide an insight into the acute effects of TA on both endurance and MFP performance, as previous studies indicated TA benefiting
performance in longer duration exercise trials and after prolonged administration (Lee et al., 2003). The results also raise questions over
the potential influence of TA on MFP in vivo (Bakker and Berg, 2002). More research is required to establish potential effects of different TA
dosages on endurance performance of different duration.
References
Bakker AJ & Berg HM. (2002). Journal of Physiology, 538, 185-194.
Lee HM, Paik IY, Park TS. (2003). Korean Journal of Nutrition, 36(7), 711-719.
Hamilton EJ, Berg HM, Easton CJ, Bakker AJ. (2006). Amino Acids, 31, 273-278.
Rutherford J, Stellingwerff T, Lawrence L. (2006). Medicine and Science in Sports and Exercise, 38(5), S127.
EFFECTS OF A 24-H FASTING AND A SUBSEQUENT MODERATE AEROBIC CYCLING ON BLOOD ADIPONECTIN CON-
CENTRATIONS AND NEUTROPHIL RESPONSES
LI, T.L., LIN, C.L., CHEN, Y.C.
NATIONAL DONG HWA UNIVERSITY
The combination of exercise and fasting is a popular way in weight reduction. Adiponectin is produced by adipose tissue and has been
shown to associate with body mass index (BMI), insulin resistance, and inflammation. The purpose of this study was to investigate the
influence of a 24-h fasting and a subsequent moderate aerobic cycling on blood adiponectin levels and neutrophil responses.
With local ethics committee approval, ten male volunteers (age 22.0±0.6 years, height 1.69±0.02 m, body mass 72.7±2.5 kg, BMI
25.2±0.6, VO2max 35.15±2.91 mL•kg-1•min-1; means ± SEM) participated in this study. After a preliminary trial of VO2max measurement,
subjects completed two main trials (fasting and control) in a counterbalanced order. Subjects dined a standardized dinner
(1082±38 kcal•kg-1: CHO 137±5 g, protein 53±2 g, fat 36±1 g) between 18:00-18:30 in both trials. Subjects kept fasted status henceforward
in fasting trial, whereas subjects ate standardized breakfast at 09:00 (216 kcal•kg-1: CHO 54 g, protein 25 g, fat 16 g) and lunch at
12:00 (identical meal to dinner) in control trial, before completing a 1-h cycling at 50% VO2max between 18:30-19:30 the following day.
Blood samples were taken at pre-dinner (Pre-DN), pre-exercise (Pre-EX), and post-exercise (Post-EX). Haematological analysis was performed
using an automated cell counter. Plasma adiponectin levels were determined using ELISA kits. fMLP induced neutrophil oxidative
burst (OB) activity was measured using a chemiluminescence assay. Results were analysed using a two-way repeated measures ANOVA
with post hoc Tukey test. Statistical significance was accepted at P