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280<br />
ingrid holme<br />
Genetic sex determination in sports builds on <strong>the</strong> view of sex taken<br />
in endocrinology, where <strong>the</strong> female body is <strong>the</strong> default and inactive<br />
morphology, and <strong>the</strong> male body is actively responding to <strong>the</strong> action<br />
of <strong>the</strong> SRY gene. The concept of <strong>the</strong> male being <strong>the</strong> result of an active<br />
development away from <strong>the</strong> default female has a long history that has<br />
been explored within a historical context by Schiebinger (1987) and<br />
Laqueur (1990) and within a social context by Fausto-Sterling (2000).<br />
The idea that all vertebrates are inherently female can be seen popular<br />
science. One of <strong>the</strong> most interesting being <strong>the</strong> fi rst Jurassic Park fi lm, in<br />
which Ian Malcolm asks, “But again, how do you know <strong>the</strong>y’re all female?<br />
Does someone go into <strong>the</strong> park and, uh . . . lift up <strong>the</strong> dinosaurs’ skirts?”<br />
Dr. Henry Wu gives <strong>the</strong> answer, “We control <strong>the</strong>ir chromosomes. It’s<br />
not that diffi cult. All vertebrate embryos are inherently female anyway.<br />
It takes an extra hormone at <strong>the</strong> right developmental stage to create<br />
a male, and we simply deny <strong>the</strong>m that” (Kennedy, Molen, Spielberg,<br />
& Crichton, 1993). Through studying Intersex conditions researchers<br />
found that only a single gene from <strong>the</strong> Y chromosome was required to<br />
produce a male morphology in an individual with X-X karyotype. The<br />
DNA sequence of this gene has since identifi ed and labeled as <strong>the</strong> SRY<br />
(Sinclair et al., 1990). Thus, a confl ict exists between <strong>the</strong> view held in<br />
endocrinology and in genetics. In endocrinology sex is seen as a process<br />
that requires testosterone and o<strong>the</strong>r hormones to continually act as<br />
chemical messengers to maintain <strong>the</strong> active process of sex difference,<br />
while, <strong>the</strong> view within genetics that <strong>the</strong> SRY acts as a “master switch,”<br />
set to an on or off position by its presence or absence.<br />
The binary sex model is integral to <strong>the</strong> identity of <strong>the</strong> SRY gene. Its<br />
existence was fi rst suggested in 1927 by Danish geneticist Øjvind Winge<br />
(1927). He hypo<strong>the</strong>sized that those genes that were benefi cial to <strong>the</strong><br />
male sex and disadvantageous to <strong>the</strong> female sex tended to accumulate<br />
near a testis-determining factor (TDF) on <strong>the</strong> Y chromosome. Winge<br />
proposed <strong>the</strong> existence of TDF as a hypo<strong>the</strong>tical Mendelian gene with a<br />
set of expected characteristics based on <strong>the</strong> view of female and male as<br />
two distinct genetic entities. Through Y chromosome mapping studies<br />
<strong>the</strong> sequence was narrowed, and in <strong>the</strong> early 1990s a DNA sequence<br />
was identifi ed that fi t <strong>the</strong> characteristics of <strong>the</strong> hypo<strong>the</strong>sized TDF. The<br />
history of <strong>the</strong> TDF/SRY gene shows how established paradigms not only<br />
pose a certain set of questions but also frame new scientifi c knowledge<br />
in line with <strong>the</strong> existing model. Thus, one of <strong>the</strong> main feminist critiques<br />
of research into sex determination problematizes <strong>the</strong> focused on <strong>the</strong><br />
male as <strong>the</strong> active, and thus more interesting (Butler, 1990).<br />
Detailing <strong>the</strong> extent to which research questions and fi ndings have<br />
been hampered by <strong>the</strong> view of sex taken with genetics is outside <strong>the</strong> scope