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Table 9 : Outcome of yeast cell walls effects in mycotoxin contaminated diets in different speciesAnimalspeciesmycotoxinconcentrationAdditivesconcentrationChickens AF (5 ppm) 0.05-0.1%Chickens AF (2 ppm) 0.05-0.1%Chickens AF (2 ppm) 0.075 %Chickens T-2 (8.1 ppm) 0.1%PigsPigsT-2 (up to 2.1ppm)DON (4.44ppm)0.2%0.2 %Outcome of feed additive effectsPerformance: improvement in body weightOrgans: restoration of relative weights of liver, proventriculus and heartSystemic response: positive effects on serum biochemical parametersPerformance : no dataOrgans : decline in the number and severity of pathological lesions in liver, bursaof Fabricius, thymus and spleenSystemic response : no dataPerformance: no dataOrgans: Subacute dietary intake of AF altered nonenzymatic components (CP, alb,vit A, â-carotene, Vit E) of antioxidant defense systems.Systemic response: yeast glucomannan was not sufficient to ameliorate theoxidative damagePerformance: no dataOrgans: partial protection of the feed additive alone and further protection incombinaison with organic selenium against antioxidant depletion and lipidperoxydation in liverSystemic response: no dataPerformance: no difference on performance whatever the dietsOrgans: no lesions were observed regardless of T-2 toxin dose or glucomannansupplementationSystemic response: protective effect against AFB 1 immunotoxicity during thevaccinal responsePerformance: Feeding of the mycotoxin-contaminated diets resulted in a decreasein feed intake and live weight gain by 28% and 14% when compared to the controlgroup.Organs: no dataSystemic response: The addition of glucomannan mycotoxin adsorbent to pigletReferencesStanley etal. (1993)Karaman etal. (2005)Çinar et al.(2008)Dvorska etal. (2007)Meissonnieret al. (2009)Dänicke etal. (2007)
Cows AF (100 ppb) 1%, 5%Cows AFB 1 (112ppb)0.56%diets which are mainly contaminated with DON could not be recommendedPerformance: evaluation of milk aflatoxin concentrations in lactating Holsteincows with the inclusion of non-digestible yeast oligosaccharidesOrgans: no dataSystemic response: No significant changes (P>0.25) in AFM 1 concentrations inresponse to non-digestible yeast oligosaccharides were verified.Performance: reduction of AFM1 concentrations in dairy cowsOrgans: no dataSystemic response: the commercial product was not effective in reducing milkAFM1 concentrations (4%), AFM1 excretion (5%), or AF transfer from feed to milk(2.52%)Hopkins etal. (2008)Kutz et al.(2009)Chickens included broiler chicks, laying hens.Rats included male and female.Pigs included piglets, sow, barrow.
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AUTEUR : Bertrand GRENIERTITRE : Ef
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REMERCIEMENTSLe travail ici présen
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J’ai une pensée particulière po
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Grenier, B., Loureiro-Bracarense, A
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TABLE DES MATIERES ................
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LISTE DES ABREVIATIONSAFAflatoxineD
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FIGURES ET TABLEAUXFigure 1 : Mycot
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INTRODUCTION8
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INTRODUCTIONCONTEXTE DE L’ETUDELe
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INTRODUCTIONElle se présentait sou
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INTRODUCTIONl’utilisation de ce m
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INTRODUCTION1. Les mycotoxines : g
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INTRODUCTIONc) La zéaralénone (vo
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INTRODUCTIONleucoencéphalomalacies
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INTRODUCTIONMycotoxins co-contamina
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INTRODUCTIONINTRODUCTIONFood safety
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INTRODUCTIONCHARACTERIZATION OF THE
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Quail(140 d)AF-FBRabbit(21 d)AF-FBR
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INTRODUCTIONdue to the ingestion of
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Table 2 : Interaction between Aflat
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(49 d) - RW-L ↗- Ab SRBC ↘- RW-
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INTRODUCTIONb) effects AF and OTA o
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INTRODUCTIONThe depletion of lympho
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AF-T2Chicken 0.3 - 3.0(35 d)AF-T2Ra
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INTRODUCTIONonly seen in birds give
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(30 d) 2AF-RUBGuinea pig 0.02 bw -(
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INTRODUCTIONbetween AF and CPA on t
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(21 day) - RBC, hemoglobin ↗- AST
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INTRODUCTIONII. INTERACTIONS BETWEE
Page 60 and 61: INTRODUCTIONwhereas in the experime
Page 62 and 63: INTRODUCTION2.2) TCT type A and BTw
Page 64 and 65: Table 6 : Interaction between Ochra
Page 66 and 67: T2-CPAChicken(28 d)T2-CPAChicken(21
Page 68 and 69: INTRODUCTIONexposed group (Brown et
Page 70 and 71: INTRODUCTION2.3) Interaction betwee
Page 72 and 73: INTRODUCTIONeffects in comparison t
Page 74 and 75: INTRODUCTION3. Procédés de décon
Page 76 and 77: INTRODUCTIONPhysical and chemical m
Page 78 and 79: INTRODUCTIONINTRODUCTIONConsumption
Page 80 and 81: INTRODUCTIONmm screen shows that fr
Page 82 and 83: INTRODUCTIONseparate the grain into
Page 84 and 85: Table 7 : toxicological evaluation
Page 86 and 87: INTRODUCTIONextrusion parameters ra
Page 88 and 89: INTRODUCTION- an important loss of
Page 90 and 91: INTRODUCTIONal., 2005). The fate of
Page 92 and 93: INTRODUCTIONby a steeping period. S
Page 94 and 95: INTRODUCTIONon cell tissue cultures
Page 96 and 97: INTRODUCTIONCONCLUSIONDetoxificatio
Page 98 and 99: INTRODUCTIONMycotoxin Reduction in
Page 100 and 101: INTRODUCTIONINTRODUCTIONMycotoxin-p
Page 102 and 103: INTRODUCTIONI. ADSORPTION OF MYCOTO
Page 104 and 105: Rats3000 ppb 0.5 %2500 ppb 0.5 %250
Page 106 and 107: INTRODUCTIONIn studies of the poten
Page 108 and 109: INTRODUCTIONSubstances investigated
Page 112 and 113: INTRODUCTIONFurthermore, cholestyra
Page 114 and 115: INTRODUCTIONwall peptidoglycans and
Page 116 and 117: INTRODUCTIONII. BIOLOGICAL DETOXIFI
Page 118 and 119: INTRODUCTIONdecrease in toxin-conce
Page 120 and 121: INTRODUCTIONCONCLUSIONPrevention an
Page 122 and 123: TRAVAILEXPERIMENTAL90
Page 124 and 125: TRAVAIL EXPERIMENTALtoxicité gén
Page 126 and 127: TRAVAIL EXPERIMENTALRESUME DES ETUD
Page 128 and 129: These are not the final page number
Page 140 and 141: TRAVAIL EXPERIMENTALChronic ingesti
Page 142 and 143: TRAVAIL EXPERIMENTALINTRODUCTIONMyc
Page 144 and 145: TRAVAIL EXPERIMENTALMATERIAL & METH
Page 146 and 147: TRAVAIL EXPERIMENTALthrough a grade
Page 148 and 149: Figure 5ABCDVilli height (µm)5432a
Page 150 and 151: TRAVAIL EXPERIMENTALRESULTS1) Histo
Page 152 and 153: Figure 8ILEUM2.01.6TNF-αbbb2.52.0I
Page 154 and 155: TRAVAIL EXPERIMENTAL3) Intestinal i
Page 156 and 157: TRAVAIL EXPERIMENTALtreated group t
Page 158 and 159: TRAVAIL EXPERIMENTALThe potencial e
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TRAVAIL EXPERIMENTALQuantification
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Figure 10 :(a) Réaction de deesté
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TRAVAIL EXPERIMENTALrévélé de l
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TRAVAIL EXPERIMENTALABSTRACTFumonis
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TRAVAIL EXPERIMENTALintestinal leve
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TRAVAIL EXPERIMENTAL3) Experimental
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TRAVAIL EXPERIMENTALthe importance
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Figure 11 : Effects of FB1 and HFB1
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Table 15 : Effects of FB1 and HFB1
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Table 17 : Effects of FB1 and HFB1
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TRAVAIL EXPERIMENTALDISCUSSIONThe a
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TRAVAIL EXPERIMENTALassociated lymp
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TRAVAIL EXPERIMENTALHartinger et al
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Figure 13 :(a) Transformation par v
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Figure 14 :Illustrations de la proc
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Figure 15 :Plan expérimental de la
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TRAVAIL EXPERIMENTAL4) Analyses du
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TRAVAIL EXPERIMENTAL9) Analyses sta
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TRAVAIL EXPERIMENTALRESULTATS1) Eff
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TRAVAIL EXPERIMENTAL2) Effet des ag
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TRAVAIL EXPERIMENTALb,cca,ba,b,da,d
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TRAVAIL EXPERIMENTALL’exposition
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Figure 21 : Effet de l’exposition
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1,41,21,00,80,60,40,20,0IL‐8a,cc
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TRAVAIL EXPERIMENTALDISCUSSIONLes o
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TRAVAIL EXPERIMENTALœdèmes pulmon
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DISCUSSIONGENERALE160
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DISCUSSION GENERALEDans ce travail
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DISCUSSION GENERALEdes vaches laiti
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Tableau 24 : Analyse de denrées ag
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DISCUSSION GENERALEet al., 2008b).
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DISCUSSION GENERALE• une réactio
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DISCUSSION GENERALE2. Les systèmes
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DISCUSSION GENERALEMycoplasma agala
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DISCUSSION GENERALEoptimale. Les au
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DISCUSSION GENERALEsphingomyéline
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DISCUSSION GENERALEsignalisation ce
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DISCUSSION GENERALEL’IMMUNITÉ IN
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DISCUSSION GENERALEplus spécifique
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DISCUSSION GENERALEsouligner que le
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DISCUSSION GENERALELA CARBOXYLESTER
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DISCUSSION GENERALEde HFB1. Toutefo
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CONCLUSIONS184
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CONCLUSIONSLes effets d’expositio
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CONCLUSIONSest utilisée lorsque la
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REFERENCES BIBLIOGRAPHIQUESAABDEL-W
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REFERENCES BIBLIOGRAPHIQUESBHANDARI
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REFERENCES BIBLIOGRAPHIQUESCASADO,
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REFERENCES BIBLIOGRAPHIQUESDEGIRMEN
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REFERENCES BIBLIOGRAPHIQUESETIENNE,
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REFERENCES BIBLIOGRAPHIQUESGRENIER,
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REFERENCES BIBLIOGRAPHIQUESHERZALLA
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REFERENCES BIBLIOGRAPHIQUESKERKADI,
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REFERENCES BIBLIOGRAPHIQUESKUMAR, M
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REFERENCES BIBLIOGRAPHIQUESMARZOCCO
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REFERENCES BIBLIOGRAPHIQUESODHAV, B
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REFERENCES BIBLIOGRAPHIQUESPFOHL-LE
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REFERENCES BIBLIOGRAPHIQUESREFAI, M
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REFERENCES BIBLIOGRAPHIQUESSCHWARTZ
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REFERENCES BIBLIOGRAPHIQUESSWANSON,
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REFERENCES BIBLIOGRAPHIQUESVEKIRU,
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REFERENCES BIBLIOGRAPHIQUESYAN, D.,
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