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Effet chez le porcelet d'une exposition à un régime co-contaminé en ...

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INTRODUCTIONpolarity. Pujol et al., (1999) <strong>co</strong>ncluded that steeping <strong>co</strong>rn kernels in 0.2% SO 2 solution at 60°C for 6 his very effective to decrease the amo<strong>un</strong>t of FB1.Ochratoxin A is distributed differ<strong>en</strong>tly in the <strong>co</strong>rn steeping process, and the toxin remains in thesteeped <strong>co</strong>rn, and do not reach into the steep water (Wood, 1982).2) Inactivation in <strong>co</strong>ntaminated maizea) Heat treatm<strong>en</strong>tMost of my<strong>co</strong>toxins are heat-stab<strong>le</strong> within the range of <strong>co</strong>nv<strong>en</strong>tional food/feed processingtemperatures (80-121°C), so litt<strong>le</strong> or no destruction occurs <strong>un</strong>der normal <strong>co</strong>oking <strong>co</strong>nditions such asboiling and frying, or ev<strong>en</strong> following pasteurization. The s<strong>en</strong>sitivity of my<strong>co</strong>toxins to heat treatm<strong>en</strong>t isaffected by many differ<strong>en</strong>t factors including moisture, pH, and ionic str<strong>en</strong>gth of food. Degradation byheat treatm<strong>en</strong>t also dep<strong>en</strong>ds on the type of my<strong>co</strong>toxin, its <strong>co</strong>nc<strong>en</strong>tration, the ext<strong>en</strong>t of bindingbetwe<strong>en</strong> the my<strong>co</strong>toxin and the food <strong>co</strong>nstitu<strong>en</strong>ts, the degree of heat p<strong>en</strong>etration, and theprocessing time (Kabak et al., 2006). A heat treatm<strong>en</strong>t which has be<strong>en</strong> widely studied for thedestruction of some naturally occurring food toxins, is extrusion <strong>co</strong>oking process. The reduction ofthe my<strong>co</strong>toxin in the finished products dep<strong>en</strong>ds on several factors including extruder temperature,screw speed, moisture <strong>co</strong>nt<strong>en</strong>t of the extrusion mixture, and transit time in the extruder. Of thesefactors, extrusion temperature and transit time seem to have the greatest effect. The highestreductions in my<strong>co</strong>toxin <strong>co</strong>nc<strong>en</strong>trations in extruded products occur at temperatures of 160°C orgreater, and wh<strong>en</strong> time of transit is increased. During extrusion <strong>co</strong>oking, the raw material issubjected to high temperatures, high pressure, and severe shear forces. Two major types of extruderare used in the food industry : sing<strong>le</strong>-screw and twin-screw extruders.Hameed (1993) showed that extrusion alone was ab<strong>le</strong> to reduce aflatoxins <strong>co</strong>nt<strong>en</strong>t in naturally<strong>co</strong>ntaminated<strong>co</strong>rn by 50-80%, and with addition of ammonia, either as hydroxide (0.7 and 1.0%) oras bicarbonate (0.4%) the aflatoxin reduction achieved was greater than 95%. However, Cazzaniga etal., (2001) emphasizes the importance of the extruder design, which <strong>co</strong>uld be a significant factoraffecting the resid<strong>en</strong>ce time and the degree of mixing.In addition to this heat process, roasting is also effective in the detoxification of aflatoxin<strong>co</strong>ntaminated<strong>co</strong>rn. Reduction in aflatoxin <strong>co</strong>nt<strong>en</strong>t was higher wh<strong>en</strong> roasting was <strong>co</strong>mbined withammonia treatem<strong>en</strong>t (Conway et al., 1978).Extrusion appears to be an effective method for reducing fumonisins <strong>le</strong>vels in <strong>co</strong>rn (Castelo et al.,1998a; Katta et al., 1999). In the study of Katta (1999), the FB1 losses in <strong>co</strong>rn grits at differ<strong>en</strong>t57

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