Tobacco and Public Health - TCSC Indonesia

Table 31.1 (continued) Epidemiological studies of cigarette smoking and endometrial cancer risk: current, former, and never smokers
First author, Study design Cases/ Age Ever Former Current Adjustment for:*
study year controls range at smoking vs. smoking smoking
(# in Cohort) recruitment never vs. never vs. never
OR (95% CI) OR (95% CI) OR (95% CI)
Smith et al. Population case– 70/612 20–54 0.8 (0.4–1.5) – – Age, BMI, HRT, parity, age
1984 control at menopause
Tyler et al. 1985 Population case– 437/3200 20–54 0.9 (0.7–1.1) 1.0 (0.7–1.4) 0.8 (0.7–1.1) Age, Weight, HRT, age at
control menopause
Weiderpass and Population case– 709/3368 50–74 – 0.9 (0.7–1.1) 0.6 (0.5–0.8) Age, BMI, HRT, parity, diabetes,
Baron, 2001 control age at menopause
Austin et al. Hospital case– 168/334 40–82 – 0.8 (0.5–1.5) 0.7 (0.4–1.2) Age, BMI, HRT, parity, diabetes
1993 control
Kelsey et al. Hospital case– 167/903 45–74 0.8 (NO CI) – – Age, HRT, age at menopause
1982 § control
Koumantaki et al. Hospital case– 83/164 40–79 – – 0.5 (0.2–1.3) Age, height, weight, HRT,
1989 control parity, age at menopause
Lesko et al. Hospital case– 510/727 18–69 – 0.9 (0.6–1.2) 0.8 (NO CI) † Age, BMI, HRT, age at
1985 control menopause, parity, diabetes
Levi et al. 1987 Hospital case– 357/1122 31–74 – 0.9 (0.5–1.5) 0.5 (0.3–0.7) Age, BMI, HRT, parity, age
control at menopause
Stockwell and Hospital case– 1374/3921 – – 0.6 (0.5–0.8) 0.7 (NO CI) † Age
Lyman 1987 control
*Covariates considered here are: age, body mass index (BMI), hormone replacement therapy (HRT), age at menopause, parity, diabetes.
† Crude measures of association were calculated from the data provided.
‡ Results shown are for women not taking unopposed estrogens. Among estrogen users, smokers were also at higher risk than nonsmokers.
§ Results reported in Baron (1984).
Former smokers were combined with never smokers in this analysis.
Note: The table was originally published in Lancet Oncology 2002;3:470–80 and has been reproduced with permission.