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PAHD<br />

82786<br />

include high phosphorous diet, parathyroid hormone, extracellular volume expansion, low dietary<br />

potassium intake and proximal tubule defects (eg Fanconi Syndrome, X-linked hypophosphatemic<br />

Rickets, tumor-induced osteomalacia). Factors which decrease, or are associated with decreases in,<br />

urinary phosphorous excretion include low dietary phosphorous intake, insulin, high dietary potassium<br />

intake, and decreased intestinal absorption of phosphorous (eg phosphate-binding antacids, vitamin D<br />

deficiency, malabsorption states). A renal leak of phosphate has also been implicated as contributing to<br />

kidney stone formation in some patients.<br />

Useful For: Evaluation of hypo- or hyper-phosphatemic states. Evaluation of patients with<br />

nephrolithiasis.<br />

Interpretation: Interpretation of urinary phosphorous excretion is dependent upon the clinical<br />

situation, and should be interpreted in conjunction with the serum phosphorous concentration.<br />

Reference Values:<br />

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