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Sorted By Test Name - Mayo Medical Laboratories

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FBIU<br />

90357<br />

9880<br />

FBAF<br />

91701<br />

BILEA<br />

200039<br />

> or =10 years: 22-29 mmol/L<br />

Clinical References: Tietz Textbook of Clinical Chemistry, Edited by Burtis and Ashwood.<br />

Philadelphia, PA, WB Saunders Company, 1999.<br />

Bicarbonate, Urine<br />

Reference Values:<br />

Normally: None Detected.<br />

<strong>Test</strong> Performed <strong>By</strong>: NMS Labs<br />

3701 Welsh Road<br />

PO Box 433A<br />

Willow Grove, PA 19090-0437<br />

Bielschowsky Stain<br />

Reference Values:<br />

The laboratory will provide a pathology consultation and stained slide.<br />

Bile Acids, Fractionated and Total<br />

Reference Values:<br />

Cholic Acid 3.1 or less umol/L<br />

Deoxycholic Acid 7.3 or less umol/L<br />

Chenodeoxycholic Acid 9.9 or less umol/L<br />

Total Bile Acids 4.5-19.2 umol/L<br />

<strong>Test</strong> Performed <strong>By</strong>: Quest Diagnostics Nichols Institute<br />

33608 Ortega Highway<br />

San Juan Capistrano, CA 92690<br />

Bile Acids, Total, Serum<br />

Clinical Information: Bile acids are formed in the liver from cholesterol, conjugated primarily to<br />

glycine and taurine, stored and concentrated in the gallbladder, and secreted into the intestine after the<br />

ingestion of a meal. In the intestinal lumen, the bile acids serve to emulsify ingested fats and thereby<br />

promote digestion. During the absorptive phase of digestion, approximately 90% of the bile acids are<br />

reabsorbed. The efficiency of the hepatic clearance of bile acids from portal blood maintains serum<br />

concentrations at low levels in normal persons. An elevated fasting level, due to impaired hepatic<br />

clearance, is a sensitive indicator of liver disease. Following meals, serum bile acid levels have been<br />

shown to increase only slightly in normal persons, but markedly in patients with various liver diseases,<br />

including cirrhosis, hepatitis, cholestasis, portal-vein thrombosis, Budd-Chiari syndrome, cholangitis,<br />

Wilson's disease, and hemochromatosis. No increase in bile acids will be noted in patients with intestinal<br />

malabsorption. Metabolic hepatic disorders involving organic anions (eg, Gilbert's disease, Crigler-Najjar<br />

syndrome, and Dubin-Johnson syndrome) do not cause abnormal serum bile acid concentrations.<br />

Useful For: An aid in the evaluation of liver function Evaluation of liver function changes before the<br />

formation of more advanced clinical signs of illness such as icterus An aid in the determination of hepatic<br />

dysfunction as a result of chemical and environmental injury An indicator of hepatic histological<br />

improvement in chronic hepatitis C patients responding to interferon treatment An indicator for<br />

intrahepatic cholestasis of pregnancy<br />

Current as of January 4, 2013 7:15 pm CST 800-533-1710 or 507-266-5700 or <strong>Mayo</strong><strong>Medical</strong><strong>Laboratories</strong>.com Page 263

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