Sorted By Test Name - Mayo Medical Laboratories

80326
BDIAL
83094
night sweats. It commonly spreads to the skin, bone, or internal genitalia where suppuration and
granulomas are typical. Occasionally, primary cutaneous lesions after trauma are encountered; however,
this type of infection is uncommon. Central nervous system disease is uncommon.
Useful For: Detection of antibodies in patients having blastomycosis
Interpretation: A positive result is suggestive of infection, but the results cannot distinguish between
active disease and prior exposure. Furthermore, detection of antibodies in cerebrospinal fluid may reflect
intrathecal antibody production, or may occur due to passive transfer or introduction of antibodies from
the blood during lumbar puncture. Routine fungal culture of clinical specimens (eg, cerebrospinal fluid) is
recommended in cases of suspected blastomycosis involving the central nervous system.
Reference Values:
Negative
Clinical References: Kaufman L, Kovacs JA, Reiss E: Clinical Immunomycology. In Manual of
Clinical and Laboratory Immunology. Edited by NL Rose, E Conway-de Macario, JD Folds, et al.
Washington, DC, American Society for Microbiology, 1997, pp 588-589
Bleach Stain
Useful For: Method for removal of melanin pigment
Reference Values:
The laboratory will provide a pathology consultation and stained slide.
Bleeding Diathesis Profile, Limited
Clinical Information: Bleeding problems may be associated with a wide variety of coagulation
abnormalities or may be due to problems not associated with coagulation (trauma and surgery as obvious
examples). A partial listing of causes follows. -Deficiency or functional abnormality (congenital or
acquired) of any of the following coagulation proteins: fibrinogen (factor I), factor II (prothrombin),
factor V, factor VII, factor VIII (hemophilia A), factor IX (hemophilia B), factor X, factor XI (hemophilia
C; bleeding severity not always proportionate to factor level), factor XIII (fibrin-stabilizing factor), von
Willebrand factor (VWF antigen and activity), and alpha-2 plasmin inhibitor and plasminogen activator
inhibitor (PAI-I; severe deficiency in rare cases). Neither alpha-2 plasmin inhibitor nor PAI-I are included
as a routine bleeding diathesis assay component, but either can be performed if indicated or requested.
-Deficiency (thrombocytopenia) or functional abnormality of platelets such as congenital (Glanzmann
thrombasthenia, Bernard-Soulier syndrome, storage pool disorders, etc) and acquired (myeloproliferative
disorders, uremia, drugs, etc) disorders. Platelet function abnormalities cannot be studied on mailed-in
specimens. -Specific factor inhibitors (most commonly directed against factor VIII); factor inhibitors
occur in 10% to15% of the hemophilia population and are more commonly associated with severe
deficiencies of factor VIII or IX (antigen