JAHRESBERICHT 2000/2001 - Fritz Thyssen Stiftung

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disease progression. – In: The Journal of Biological Chemistry. 275.
2000. S. 9620–9627.
Schwarz, G., et al.: The molybdenum cofactor biosynthetic protein
Cnx1 complements molybdate-repaireable mutants, transfers molybdenum
to the metal binding pterin, and is associated with the cytoskeleton.
– In: Plant Cell. 12. 2000. S. 2455–2472.
Shmueli, O., et al.: DCX in PC12 cells. Downregulation of CREB –
mediated transcription. – In: Hum Mol Genet. 10. 2001. S. 1061–1070.
Sun, C., et al.: Deletion of AZFa (Azoospermia Factor a) region of
human Y chromosome caused by recombination between HERV15
proviruses. – In: Hum Mol Genet. 15. 2000. S. 2291–2296.
Stucka, Rolf, et al.: A modified alignment of human and rodent 5’
untranslated sequences of the acetylcholine receptor epsilon subunit
gene reveals additional regions of high homology. – In: Neuromuscular
Disorders. 10. 2000. S. 213/214.
Taubert, Helge, et al.: The p53 status in juvenile chronic arthritis and
rheumatoid arthritis. – In: Clin Exp Immunol. 123. 2001. S. 147–154.
Thermann, Rolf, et al.: Binary specification of nonsense codons by
splicing and cytoplasmic translation. – In: The EMBO Journal. 17.
1998. S. 3484–3494.
Touraine, Renaud L., et al.: Neurological phenotype in Waardenburg
type 4 syndrome correlates with novel SOX10 truncating mutations
and expression in developing brain. – In: Am J Hum Genet. 66.
2000. S. 1496–1503.
Utech, Markus, et al.: Accumulation of RhoA, RhoB, RhoG, and Rac 1
in fibroblasts from Tangier disease subjects suggests a regulatory
role of Rho family proteins in cholesterol efflux. – In: Biochemical
and Biophysical Research Communications. 280. 2001. S. 229–236.
Vetter, B., et al.: Dominant �-thalassaemia. A highly unstable haemoglobin
is caused by a novel 6bp deletion of the �-globin gene. –
In: British Journal of Haematology. 108. 2000. S. 176–181.
Waltenberger, Johannes, et al.: Functional upregulation of the vascular
endothelial growth factor receptor KDR by hypoxia. – In: Circulation.
94. 1996. S. 1647–1654.
Waltenberger, Johannes: Gentechnologie und Molekularbiologie in
der Kardiologie : Update 2000. – In: J Kardiol. 7(6). 2000. S. 246–251.
Waltenberger, Johannes: Modulation of growth factor action. Implications
for the treatment of cardiovascular diseases. – In: Circulation.
96. 1997. S. 4083–4094.
Waltenberger, Johannes, et al.: Neovascularization in the human
heart is associated with expression of VEGF-A and its receptors