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CHAPTER 9 The Kidney and Urinary Tract 329

A

B

FIG. 9.26 Xanthogranulomatous Pyelonephritis. (A) Sagittal sonogram shows an enlarged hypoechoic kidney and a large showing staghorn

calculus. (B) Corresponding contrast-enhanced CT demonstrates a diffusely enlarged left kidney, multiple intrarenal abscesses, and a large, central

obstructing calculus.

A

B

FIG. 9.27 Papillary Necrosis. (A) Sagittal and (B) transverse sonograms show swollen bulbous papillae.

Sonographic Findings of Papillary Necrosis

Swollen pyramids

Papillary cavitation

Adjacent clubbed calix

Sloughed papilla in collecting system that can calcify and

simulate a stone

Sloughed papilla may cause obstruction

Hydronephrosis may develop if the sloughed papilla obstructs

the ureter.

Tuberculosis

Urinary tract tuberculosis (TB) occurs with hematogenous seeding

of the kidney by Mycobacterium tuberculosis from an extraurinary

source (typically lung). Urinary tract TB usually manifests 5 to

10 years ater the initial pulmonary infection. Chest radiographs

may be normal (35%-50% of patients) or may show active TB

(10%) or inactive healed TB (40%-55%). Most patients present

with lower urinary tract signs and symptoms that include frequency,

dysuria, nocturia, urgency, and gross or microscopic

hematuria, whereas 10% to 20% of patients are asymptomatic. 48

Urinalysis indings include sterile pyuria, microscopic hematuria,

and acidic pH. TB is deinitively diagnosed with acid-fast bacilli

urine cultures; however, this usually requires 6 to 8 weeks for

growth.

Although both kidneys are seeded initially, clinical manifestations

of urinary tract TB are typically unilateral. he early or

acute changes include development of multiple small bilateral

tuberculomas. Das et al. 49 found that the most frequently

encountered sonographic abnormality was focal renal lesions

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