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CHAPTER 14 The Peritoneum 521

10% to 15%; this increases to more than 50% in AIDS patients. 61

he peritoneum is a common site of extrapulmonary involvement,

62 but the chest radiograph will show evidence of pulmonary

TB in only 14% of these patients. herefore a high index of

suspicion, particularly in high-risk groups, and knowledge

of the common sonographic features allow for earlier diagnosis

of this potentially curable disease, thus reducing morbidity and

mortality.

here are no pathognomonic sonographic features for TB

peritonitis but, in the proper clinical setting, a difuse peritoneal

process may strongly suggest the diagnosis. Ascites is frequently

present and may be free or loculated. It may be anechoic or more

frequently particulate and may contain ine, mobile strands

composed of ibrin. hese strands may produce a latticelike

pattern. Irregular and nodular hypoechoic thickening of the

peritoneum, mesentery, and omentum is another feature (Fig.

14.35). 63 Associated lymphadenopathy in the mesentery and

retroperitoneum is a common feature and is more common than

in peritoneal carcinomatosis. 64-66 he nodes may be discrete or

conglomerate because of periadenitis. Caseation may give rise

to a hypoechoic center within the node, although a similar

appearance can be seen with metastatic lymph nodes undergoing

necrosis. Echogenic nodes caused by fat deposition may suggest

the diagnosis of TB. Sonographic assessment of the solid viscera

may show involvement, particularly hypoechoic masses in the

spleen. Ultrasound helps guide diagnostic paracentesis in TB

peritonitis and may also guide ine-needle aspiration of enlarged

nodes. 67 Sonography can also readily document response to

treatment.

Sclerosing Peritonitis

Sclerosing peritonitis is a major complication of continuous

ambulatory peritoneal dialysis and is characterized by the

formation of a connective tissue membrane covering the

peritoneum and eventually encasing and strangulating bowel

loops. 68,69 Patients initially complain of abdominal pain and loss

of ultrailtration. Ultimately, bowel obstruction occurs. Surgery is

oten diicult in these patients, and the prognosis is poor. Early

diagnosis of sclerosing peritonitis may be important in reducing

mortality.

Ultrasound is extremely helpful in the diagnosis. 70 Increased

peristalsis in multiple bowel loops is one of the earliest indings

in sclerosing peritonitis. Ascites, both free and loculated, is

common. With time, the luid becomes more complex with

stranding (Fig. 14.36). Bowel loops become matted together

and are tethered to the posterior abdominal wall by a characteristic

enveloping membrane. his membrane can be seen with ultrasound

as a uniformly echogenic layer measuring 1 to 4 mm in

thickness.

LOCALIZED INFLAMMATORY

PROCESS OF PERITONEAL CAVITY

he CT appearance and signiicance of inlamed peritoneal fat

are familiar to sonographers. If ultrasound is to be successful at

investigating patients with abdominal symptoms, the sonographic

appearance of inlamed fat must become just as familiar.

Inlamed perienteric fat appears as an echogenic “mass efect,”

with ultrasound frequently displacing bowel loops out of the

scanning plane. Compression sonography may greatly enhance

the detection of focally inlamed fat, and gentle palpation with

the transducer over this area will frequently show that it is the

site of the patient’s maximal tenderness. Frequently, an associated

underlying abnormality, such as an abnormal bowel segment,

can be identiied with ultrasound 71 (Fig. 14.37). Appendicitis

and diverticulitis are the most common acute processes giving

rise to focally inlamed fat. Other possibilities include inlammatory

bowel disease, pancreatitis, and complicated acute

cholecystitis. Progression to phlegmon typically shows development

of a hypoechoic region within the echogenic fat without

luid content (Fig. 14.38). If untreated, this may progress to abscess

formation. Color Doppler imaging frequently shows increased

blood low in the area of inlammation. 72

RIGHT-SIDED SEGMENTAL OMENTAL

INFARCTION

Right-sided segmental omental infarction is a rare clinical entity

that usually presents with right-sided abdominal pain and is

oten mistaken for appendicitis. It is important to make the correct

diagnosis because the condition is self-limiting and resolves

spontaneously with supportive measures. Omental infarction

occurs in all age groups and is thought to result from an embryologic

variant in the blood supply to the right inferior portion of

the omentum, leaving it prone to infarction. Precipitating factors

include straining and eating a large meal.

Ultrasound reveals an echogenic, ovoid, or cakelike mass in

the right midabdomen at the site of the patient’s tenderness 73,74

(Fig. 14.39). Careful assessment will reveal no underlying bowel

abnormality. he typical location of right-sided omental infarction

is anterolateral to the hepatic lexure of the colon, and it corresponds

to a circumscribed fatty mass on CT, with areas of

stranding. he mass oten adheres to the parietal peritoneum,

with bowel moving deep to it on respiration.

ENDOMETRIOSIS

Endometriosis is a common condition afecting predominantly

premenopausal women and occurs when functional endometrium

is located outside of the uterus. Patients may be asymptomatic

but frequently present with pelvic pain, dyspareunia, or infertility.

he ovaries and suspensory ligaments of the uterus are

the sites most oten afected, but endometriotic implants can

involve the bowel, urinary bladder, peritoneum, chest, or sot

tissues. 75

Sonographic evaluation is oten normal in patients with

endometriosis. If endometriomas are present, TVS is very

sensitive at detecting and characterizing the masses, oten showing

the typical “chocolate” cysts with uniform, low-level internal

echoes (Fig. 14.40, Video 14.8). here may be associated complex

free luid with stranding. Occasionally, tiny echogenic foci may

be identiied along the pelvic peritoneal surfaces. hese foci are

not speciic for endometriosis and may also be seen with serous

papillary ovarian neoplasms. Clinical correlation is essential,

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