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658 PART II Abdominal and Pelvic Sonography

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FIG. 18.41 Renal Artery Stenosis: Donor Portion. (A) Color Doppler ultrasound of donor renal artery anastomosis shows focal area of aliasing

(arrow). (B) Power Doppler shows area of narrowing in this region (arrow). (C) Spectral Doppler shows elevated angle-corrected velocities at the

site of the arrow, greater than 400 cm/sec.

Intraparenchymal arterial stenosis may be observed in

chronic rejection as a result of scarring in the tissues surrounding

the involved vessels. On spectral Doppler ultrasound, a prolonged

AT may be observed in the segmental and interlobar arteries,

with a normal main renal artery waveform. 45

Treatment options for renal artery stenosis include percutaneous

transluminal angioplasty, endovascular stent placement, and

surgery. Surgical management of these transplants involves

resection and revision of the stenosis with insertion of a patch

grat at the stenotic segment. 46

A false-positive Doppler diagnosis of renal artery stenosis

can occur if there is an abrupt turn in the main renal artery, if

the artery is severely tortuous, or if there are errors in Doppler

technique (Fig. 18.44). Inadvertent compression of the main

renal artery by the sonographer while performing spectral interrogation

may also produce transient narrowing of the artery and

elevated PSV readings. Turning the patient in a decubitus position

such that the anterior abdominal or pelvic wall tissues are displaced

from lying over the transplant can reduce external pressure

on the allograt during scanning.

Venous Thrombosis

Occlusive renal vein thrombosis is slightly more common than

arterial thrombosis, occurring in up to 4% of transplants, and

is associated with acute pain, swelling of the allograt, and an

abrupt cessation of renal function between the third and eighth

postoperative day. Risk factors include technical diiculties at

surgery, hypovolemia, propagation of femoral or iliac thrombosis,

and compression by luid collections. 58,64

On gray-scale ultrasound, the allograt may appear

enlarged, and in rare cases intraluminal thrombus may be

detected in a dilated main renal vein or within the intraparenchymal

venous system. Spectral and color Doppler ultrasound

will show a lack of venous low in the renal parenchyma,

absence of low in the main renal vein, and reversal of diastolic

low in the main renal artery, as well as sometimes in

the intraparenchymal arteries 65,66 (Fig. 18.45). he sonographer

should be aware that reversal of low in diastole in the

main renal artery or the intraparenchymal arterial branches

is highly suggestive of renal vein thrombosis only in the

absence of venous low in the renal parenchyma and main

renal vein.

Reversed diastolic arterial low, with preservation of

venous low, is a nonspeciic inding indicating extremely high

vascular resistance in the small intrarenal vessels or main hilar

vessels. he outcome for these patients is generally poor, with

allograt loss rates of 33% to 55%. Potential causes of reversed

diastolic low include acute rejection, ATN, peritransplant

hematomas (compressing renal grat or hilar vessels), and

glomerulosclerosis. 67

Renal Vein Stenosis

Renal vein stenosis most oten occurs from perivascular ibrosis

or external compression by adjacent luid collections. he renal

cortex appears either normal or hypoechoic, and on color Doppler,

aliasing is identiied at the stenotic region because of focal,

high-velocity turbulent low. On spectral Doppler sonography,

a threefold to fourfold increase in velocity across the region of

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